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(191, 'pH MIRACLE ACETYL L-CARNITINE BENEFITS: THE SUPPLEMENT THAT POWERS THE BRAIN & BUILDS MUSCLE', 'https://phoreveryoung.wordpress.com/2019/11/24/ph-miracle-acetyl-l-carnitine-benefits-the-supplement-that-powers-the-brain-builds-muscle/', 'pH MIRACLE ACETYL L-CARNITINE BENEFITS: THE SUPPLEMENT THAT POWERS THE BRAIN & BUILDS MUSCLE\nALCAR also helps lactic acid in muscles, use fat to buffer acidity, which can increase your endurance, give you an edge in the gym, and help you recover faster. For nootropic benefits, take two grams of ALCAR a day.For better endurance and faster recovery, take 2-3 grams of ALCAR a day — one capsule at breakfast and 2 capsules before your workout. You can also take one capsule after workout.\nAcetyl-L-carnitine (ALCAR) , is a powerful addition to your biohacking toolbox. This valuable little molecule has been a staple in the bodybuilding and brain-hacking communities for years now, thanks to its ability to help you lose weight and power up your brain.\nRead on to learn more about the benefits of acetyl L-carnitine, safety and proper supplement dosage.\nAcetyl-L-carnitine is an amino naturally produced in your body to protect cells from excess metabolic acids, use stored fats to build stem cells, and protect electrical energy for cellular function.. We suggest supplementing with acetyl L-carnitine, because of its many benefits.\nWHAT ARE THE BENEFITS OF ALCAR?\nYou can use ALCAR to enhance your cellular electron function to increase cognition function, and to reduce metabolic acids, like lactic acid during workouts, and more. ALCAR helps your body use fat for buffering excess acidic so that the body cells have more more energy — especially for your muscles and brain.\nAcetyl L-carnitine has two main benefits:\nBoosts brain power: ALCAR is an anti-aging nootropic, or smart amino, meaning, it helps your brain perform better. The acetyl group on it lets it pass your blood-brain barrier and reach your brain and nerves, for buffering metabolic acids thus improve your mood, learning, and memory.\nHelps the body use fat and builds muscle : ALCAR can give you an edge in the gym, too. It buffers lactic acid from cellular function of the muscle, which increases cellular endurance and recovery time of the muscle. ALCAR also improves acid-bound fat elimination while you are working out.\nHere’s a closer look at how ALCAR works.\nACETYL L-CARNITINE AND WEIGHT LOSS: HOW IT HELPS ELIMINATE EXCESS ACID BOUND FAT?\nWhen you take pH Miracle ALCAR, it turns into carnitine in your body, and if your insulin levels are low — for example, from eating a high carbohydrate/sugar diet, doing intermittent fasting, working out intensely, eating a highly acidic diet of animal protein or not eating enough alkalizing fruit and vegetables— carnitine becomes very important for reducing metabolic and dietary acid-loads.\nWhen your insulin (insulin buffers the metabolic acid glucose and lactic acid)levels are low, your body starts pushing metabolic and dietary acids into the interstitial fluid compartments and then into the fatty tissues.. [1]\nThe more metabolic and dietary acids that are not properly eliminated through the five channels of elimination – urination, defecation, respiration, perspiration and for women menstruation – are then stored away from the organs that sustain life in the fatty tissue. It is important to understand that overweight is NOT a fat problem but an acid problem! (Read The pH Miracle for Weight Loss)\nBasically, carnitine latches onto metabolic, dietary, respiratory and environmental acidic waste and carries them out of the interstitial fluids into the fatty tissues when they cannot be removed via the elimination organs. Depositing acidic waste into the fatty tissues is protective but is also the cause of excess weight that leads to obesity!\nIn other words, you need carnitine to eliminate acidic metabolic, dietary, respiratory, and environmental acidic waste out through the five channels of elimination. If you’re low on carnitine, acidic waste can get into your body cells that make up all of your organs causing enervation,(low energy), irritation, inflammation, ulceration, induration (stones and tumors) and finally degeneration (cancer).\nOnce again, the body retains fat to protect the organs and organ systems from breakdown. [2] But with abundant L-carnitine, your body can remove excess metabolic and dietary acidic waste and you will feel more life force energy or experience less fatigue or enervation. The best news is you will start losing excess acidic weight bound to fat and start building muscle. That’s why the pH Miracle ALCAR supplement is so popular with folks who want to lose weight and build muscle and prevent chronic and acute diseases.\nACETYL L-CARNITINE FOR MOOD, MEMORY AND COGNITIVE DECLINE\nAcetyl-L-carnitine is a supplement form of L-carnitine that can pass your blood-brain barrier, giving it access to the dense collection of mitochondria in your brain, as well as to the mitochondria in the rest of your body. That explains why ALCAR is becoming so popular as a nootropic supplement (something that makes your brain work better).\nMore mental & physical energy : Older men and women who took ALCAR daily saw a significant boost to their physical and mental energy levels, as well as better overall cognitive ability. [3]\nImproves cognitive function : People with low carnitine levels see a major decrease in brain function. Taking ALCAR improves that brain function. A lot. In a recent study, 8 out of 14 participants reversed a diagnosis of dementia and returned to their full cognitive abilities after taking ALCAR daily. [4]\nLowers inflammation : Acetyl-L-carnitine decreases inflammation in healthy adults. Part of it is because ALCAR increases glutathione, your body’s master antioxidant. [5] Side note: glutathione is awesome for hacking hangovers. And, as it turns out, ALCAR decreases damage to brain cells after drinking, too. [6]\nBoosts circulation : In healthy adults, ALCAR increased circulation and nutrient-rich blood flow after a high-fat meal. [7] Good blood flow is an essential part of brain and muscle function and the removal of dietary and metabolic acids from the interstitial fluid compartments of the Interstitium that make YOU sick and tired.\nImproves focus : Acetyl-L-carnitine improves attention, both in kids with ADHD [8] and adults who struggle with mental fatigue. [9]\nWHO SHOULD USE ACETYL L-CARNITINE AS A NOOTROPIC?\nAs is the case with many nootropics, ALCAR is popular in the brain-hacking world thanks to lots of individual reports about how much it helps cognitive function. That is why pH Miracle L-Carnitine is one of those supplements that’s worth trying on your own, to experience ALL of its benefits.\nWhat about pH Miracle ALCAR for improving your workouts?\nMORE FROM BULLETPROOF ACETYL L-CARNITINE FOR EXERCISE AND RECOVERY!\nIncreases strength : Resistance-trained young men who took carnitine 90 minutes before intense exercise enjoyed a significant increase in power output, as well as lower lactate levels (lactate contributes to the burning feeling in your muscles). [10]\nBurns more fat : Young men who took carnitine burned more fat during moderate exercise, and their muscles produced more energy during intense exercise. [11]\nBoosts testosterone : Men who took acetyl-L-carnitine after lifting had more dense hormone receptors in their muscles, which may have improved their testosterone uptake. https://www.ncbi.nlm.nih.gov/pubmed/16826026 In other words, ALCAR doesn’t increase your testosterone; it makes you better at using the testosterone you have (Here’s how to increase your testosterone).\nSpeeds recovery : pH Miracle ALCAR can speed up recovery and decrease muscle inflammation after exercise, in both men and women. [12] [13] [14] [15]\nBetter insulin sensitivity : pH Miracle ALCAR improves insulin sensitivity in people who don’t process glucose well. [16] [17] [18]\nBuilds muscle : ALCAR is a powerful anti-aging supplement if you’re older and want to build muscle. In older people, it decreases muscle fatigue and increases fat loss. [19] [20] [21]\nReduces muscle fatigue : One study found that acetyl-L-carnitine decreases muscle fatigue in young people, too. [22]\nThe pH Miracle ALCAR can help your workout in the gym, reduce acidic waste that causing inflammation that leads to faster recovery. And if you’re older, pH Miracle acetyl-L-carnitine is especially useful for maintaining your energy levels and shedding acidic body fat\nDOSAGE: HOW MUCH PH MIRACLE ACETYL-L-CARNITINE SHOULD YOU TAKE?\npH Miracle Acetyl-L-carnitine is a valuable addition to your daily supplement routine , especially if you’re an athlete or you’re looking for anti-aging benefits. As with most supplements, timing matters. Here’s what you need to know.\nMost of the studies you just read about used between 2 to 3 grams of L-carnitine, taken every day. Two grams a day is a good place to start. From there, you can increase or decrease the dose when you reach your goals. Here are sample plans for taking pH Miracle ALCAR:\npH Miracle ACETYL L-CARNITINE FOR NOOTROPIC BENEFITS/ANTI-AGING:\n2g/day; half first thing in the morning, half in the afternoon\npH Miracle ACETYL L-CARNITINE FOR FITNESS AND REMOVING ACID BOUND FAT:\n2-3g day; half 90 minutes before exercise (to increase your muscle endurance), half after exercise (to speed up recovery)\nWHERE DO I GET PH MIRACLE ACETYL L-CARNITINE?\nJust click on the following link: https://www.phmiracleproducts.com/collections/ph-miracle/products/l-carnitine\n[1] https://www.sciencedirect.com/topics/biochem… [2] http://europepmc.org/abstract/med/6348429 [3] Arch Gerontol Geriatr. 2008 Mar-Apr;46(2):181-90. Epub 2007 Jul 20. Acetyl L-carnitine (ALC) treatment in elderly patients with fatigue. Malaguarnera M 1, Gargante MP , Cristaldi E , Colonna V , Messano M , Koverech A , Neri S , Vacante M , Cammalleri L , Motta M . [4] J Psychiatr Pract. 2010 Jan;16(1):5-14. doi: 10.1097/01.pra.0000367773.03636.d1. Clinical outcomes and low-dose levocarnitine supplementation in psychiatric inpatients with documented hypocarnitinemia: a retrospective chart review. Cuturic M 1, Abramson RK , Moran RR , Hardin JW . [5] Tohoku J Exp Med. 2011 Jul;224(3):209-13. doi: 10.1620/tjem.224.209. Single dose administration of L-carnitine improves antioxidant activities in healthy subjects. Cao Y 1, Qu HJ , Li P , Wang CB , Wang LX , Han ZW . [6] Free Radic Biol Med. 2011 Oct 15;51(8):1601-9. doi: 10.1016/j.freeradbiomed.2011.06.020. Epub 2011 Jun 28. Stabilization of superoxide dismutase by acetyl-l-carnitine in human brain endothelium during alcohol exposure: novel protective approach. Haorah J 1, Floreani NA , Knipe B , Persidsky Y .. https://www.ncbi.nlm.nih.gov/pubmed/21782933 [7] Am J Cardiol. 2008 Nov 15;102(10):1413-7. doi: 10.1016/j.amjcard.2008.07.022. Epub 2008 Sep 11. Effects of carnitine supplementation on flow-mediated dilation and vascular inflammatory responses to a high-fat meal in healthy young adults. Volek JS 1, Judelson DA , Silvestre R , Yamamoto LM , Spiering BA , Hatfield DL , Vingren JL , Quann EE , Anderson JM , Maresh CM , Kraemer WJ . https://www.ncbi.nlm.nih.gov/pubmed/18993165 [8] Prostaglandins Leukot Essent Fatty Acids. 2002 Jul;67(1):33-8. Efficacy of carnitine in the treatment of children with attention-deficit hyperactivity disorder. Van Oudheusden LJ 1, Scholte HR . https://www.ncbi.nlm.nih.gov/pubmed/12213433 [9] Psychosom Med. 2004 Mar-Apr;66(2):276-82. Exploratory open label, randomized study of acetyl- and propionylcarnitine in chronic fatigue syndrome. Vermeulen RC 1, Scholte HR . https://www.ncbi.nlm.nih.gov/pubmed/15039515 [10] J Int Soc Sports Nutr. 2009 Apr 2;6:9. doi: 10.1186/1550-2783-6-9. Glycine propionyl-L-carnitine produces enhanced anaerobic work capacity with reduced lactate accumulation in resistance trained males. Jacobs PL 1, Goldstein ER , Blackburn W , Orem I , Hughes JJ . https://www.ncbi.nlm.nih.gov/pubmed/19341458 [11] J Physiol. 2011 Feb 15;589(Pt 4):963-73. doi: 10.1113/jphysiol.2010.201343. Epub 2011 Jan 4. Chronic oral ingestion of L-carnitine and carbohydrate increases muscle carnitine content and alters muscle fuel metabolism during exercise in humans. Wall BT 1, Stephens FB , Constantin-Teodosiu D , Marimuthu K , Macdonald IA , Greenhaff PL . https://www.ncbi.nlm.nih.gov/pubmed/21224234 [12] Metabolism. 2010 Aug;59(8):1190-9. doi: 10.1016/j.metabol.2009.11.012. Epub 2009 Dec 31. l-Carnitine l-tartrate supplementation favorably affects biochemical markers of recovery from physical exertion in middle-aged men and women. Ho JY 1, Kraemer WJ , Volek JS , Fragala MS , Thomas GA , Dunn-Lewis C , Coday M , Häkkinen K , Maresh CM . https://www.ncbi.nlm.nih.gov/pubmed/20045157 [13] J Strength Cond Res. 2008 Jul;22(4):1130-5. doi: 10.1519/JSC.0b013e31817d48d9. Effects of L-carnitine L-tartrate supplementation on muscle oxygenation responses to resistance exercise. Spiering BA 1, Kraemer WJ , Hatfield DL , Vingren JL , Fragala MS , Ho JY , Thomas GA , Häkkinen K , Volek JS . https://www.ncbi.nlm.nih.gov/pubmed/18545197 [14] J Strength Cond Res. 2003 Aug;17(3):455-62. The effects of L-carnitine L-tartrate supplementation on hormonal responses to resistance exercise and recovery. Kraemer WJ 1, Volek JS , French DN , Rubin MR , Sharman MJ , Gómez AL , Ratamess NA , Newton RU , Jemiolo B , Craig BW , Häkkinen K . https://www.ncbi.nlm.nih.gov/pubmed/12930169 [15] Am J Physiol Endocrinol Metab. 2002 Feb;282(2):E474-82. L-Carnitine L-tartrate supplementation favorably affects markers of recovery from exercise stress. Volek JS 1, Kraemer WJ , Rubin MR , Gómez AL , Ratamess NA , Gaynor P . https://www.ncbi.nlm.nih.gov/pubmed/11788381 [16] JPEN J Parenter Enteral Nutr. 2010 May-Jun;34(3):295-9. doi: 10.1177/0148607109353440. Caloric restriction and L-carnitine administration improves insulin sensitivity in patients with impaired glucose metabolism. Molfino A 1, Cascino A , Conte C , Ramaccini C , Rossi Fanelli F , Laviano A . https://www.ncbi.nlm.nih.gov/pubmed/20467011 [17] Hypertension. 2009 Sep;54(3):567-74. doi: 10.1161/HYPERTENSIONAHA.109.132522. Epub 2009 Jul 20. Ameliorating hypertension and insulin resistance in subjects at increased cardiovascular risk: effects of acetyl-L-carnitine therapy. Ruggenenti P 1, Cattaneo D , Loriga G , Ledda F , Motterlini N , Gherardi G , Orisio S , Remuzzi G . https://www.ncbi.nlm.nih.gov/pubmed/19620516 [18] Ann Nutr Metab. 2008;52(4):335-8. doi: 10.1159/000151488. Epub 2008 Aug 19. Effect of oral L-carnitine administration on insulin sensitivity and lipid profile in type 2 diabetes mellitus patients. González-Ortiz M 1, Hernández-González SO , Hernández-Salazar E , Martínez-Abundis E . https://www.ncbi.nlm.nih.gov/pubmed/18714152 [19] Am J Clin Nutr. 2007 Dec;86(6):1738-44. L-Carnitine treatment reduces severity of physical and mental fatigue and increases cognitive functions in centenarians: a randomized and controlled clinical trial. Malaguarnera M 1, Cammalleri L , Gargante MP , Vacante M , Colonna V , Motta M . https://www.ncbi.nlm.nih.gov/pubmed/18065594 [20] Arch Gerontol Geriatr. 2008 Mar-Apr;46(2):181-90. Epub 2007 Jul 20. Acetyl L-carnitine (ALC) treatment in elderly patients with fatigue. Malaguarnera M 1, Gargante MP , Cristaldi E , Colonna V , Messano M , Koverech A , Neri S , Vacante M , Cammalleri L , Motta M . https://www.ncbi.nlm.nih.gov/pubmed/17658628 [21] Drugs Aging. 2003;20(10):761-7. Levocarnitine administration in elderly subjects with rapid muscle fatigue: effect on body composition, lipid profile and fatigue. Pistone G 1, Marino A , Leotta C , Dell’Arte S , Finocchiaro G , Malaguarnera M . https://www.ncbi.nlm.nih.gov/pubmed/12875611 [22] J Clin Biochem Nutr. 2007 Nov;41(3):224-30. doi: 10.3164/jcbn.2007032. Effects of Citric Acid and l-Carnitine on Physical Fatigue. Sugino T 1, Aoyagi S , Shirai T , Kajimoto Y , Kajimoto O . https://www.ncbi.nlm.nih.gov/pubmed/18299720\n[1] https://www.sciencedirect.com/topics/biochem…\n[2] http://europepmc.org/abstract/med/6348429\n[3] Arch Gerontol Geriatr. 2008 Mar-Apr;46(2):181-90. Epub 2007 Jul 20.\nAcetyl L-carnitine (ALC) treatment in elderly patients with fatigue.\nMalaguarnera M 1, Gargante MP , Cristaldi E , Colonna V , Messano M , Koverech A , Neri S , Vacante M , Cammalleri L , Motta M .\n[4] J Psychiatr Pract. 2010 Jan;16(1):5-14. doi: 10.1097/01.pra.0000367773.03636.d1.\nClinical outcomes and low-dose levocarnitine supplementation in psychiatric inpatients with documented hypocarnitinemia: a retrospective chart review.\nCuturic M 1, Abramson RK , Moran RR , Hardin JW .\n[5] Tohoku J Exp Med. 2011 Jul;224(3):209-13. doi: 10.1620/tjem.224.209.\nSingle dose administration of L-carnitine improves antioxidant activities in healthy subjects.\nCao Y 1, Qu HJ , Li P , Wang CB , Wang LX , Han ZW .\n[6] Free Radic Biol Med. 2011 Oct 15;51(8):1601-9. doi: 10.1016/j.freeradbiomed.2011.06.020. Epub 2011 Jun 28.\nStabilization of superoxide dismutase by acetyl-l-carnitine in human brain endothelium during alcohol exposure: novel protective approach.\nHaorah J 1, Floreani NA , Knipe B , Persidsky Y ..\nhttps://www.ncbi.nlm.nih.gov/pubmed/21782933\n[7] Am J Cardiol. 2008 Nov 15;102(10):1413-7. doi: 10.1016/j.amjcard.2008.07.022. Epub 2008 Sep 11.\nEffects of carnitine supplementation on flow-mediated dilation and vascular inflammatory responses to a high-fat meal in healthy young adults.\nVolek JS 1, Judelson DA , Silvestre R , Yamamoto LM , Spiering BA , Hatfield DL , Vingren JL , Quann EE , Anderson JM , Maresh CM , Kraemer WJ .\nhttps://www.ncbi.nlm.nih.gov/pubmed/18993165\n[8] Prostaglandins Leukot Essent Fatty Acids. 2002 Jul;67(1):33-8.\nEfficacy of carnitine in the treatment of children with attention-deficit hyperactivity disorder.\nVan Oudheusden LJ 1, Scholte HR .\nhttps://www.ncbi.nlm.nih.gov/pubmed/12213433\n[9] Psychosom Med. 2004 Mar-Apr;66(2):276-82.\nExploratory open label, randomized study of acetyl- and propionylcarnitine in chronic fatigue syndrome.\nhttps://www.ncbi.nlm.nih.gov/pubmed/15039515\n[10] J Int Soc Sports Nutr. 2009 Apr 2;6:9. doi: 10.1186/1550-2783-6-9.\nGlycine propionyl-L-carnitine produces enhanced anaerobic work capacity with reduced lactate accumulation in resistance trained males.\nJacobs PL 1, Goldstein ER , Blackburn W , Orem I , Hughes JJ .\nhttps://www.ncbi.nlm.nih.gov/pubmed/19341458\n[11] J Physiol. 2011 Feb 15;589(Pt 4):963-73. doi: 10.1113/jphysiol.2010.201343. Epub 2011 Jan 4.\nChronic oral ingestion of L-carnitine and carbohydrate increases muscle carnitine content and alters muscle fuel metabolism during exercise in humans.\nWall BT 1, Stephens FB , Constantin-Teodosiu D , Marimuthu K , Macdonald IA , Greenhaff PL .\nhttps://www.ncbi.nlm.nih.gov/pubmed/21224234\n[12] Metabolism. 2010 Aug;59(8):1190-9. doi: 10.1016/j.metabol.2009.11.012. Epub 2009 Dec 31.\nl-Carnitine l-tartrate supplementation favorably affects biochemical markers of recovery from physical exertion in middle-aged men and women.\nHo JY 1, Kraemer WJ , Volek JS , Fragala MS , Thomas GA , Dunn-Lewis C , Coday M , Häkkinen K , Maresh CM .\nhttps://www.ncbi.nlm.nih.gov/pubmed/20045157\n[13] J Strength Cond Res. 2008 Jul;22(4):1130-5. doi: 10.1519/JSC.0b013e31817d48d9.\nEffects of L-carnitine L-tartrate supplementation on muscle oxygenation responses to resistance exercise.\nSpiering BA 1, Kraemer WJ , Hatfield DL , Vingren JL , Fragala MS , Ho JY , Thomas GA , Häkkinen K , Volek JS .\nhttps://www.ncbi.nlm.nih.gov/pubmed/18545197\n[14] J Strength Cond Res. 2003 Aug;17(3):455-62.\nThe effects of L-carnitine L-tartrate supplementation on hormonal responses to resistance exercise and recovery.\nKraemer WJ 1, Volek JS , French DN , Rubin MR , Sharman MJ , Gómez AL , Ratamess NA , Newton RU , Jemiolo B , Craig BW , Häkkinen K .\nhttps://www.ncbi.nlm.nih.gov/pubmed/12930169\n[15] Am J Physiol Endocrinol Metab. 2002 Feb;282(2):E474-82.\nL-Carnitine L-tartrate supplementation favorably affects markers of recovery from exercise stress.\nVolek JS 1, Kraemer WJ , Rubin MR , Gómez AL , Ratamess NA , Gaynor P .\nhttps://www.ncbi.nlm.nih.gov/pubmed/11788381\n[16] JPEN J Parenter Enteral Nutr. 2010 May-Jun;34(3):295-9. doi: 10.1177/0148607109353440.\nCaloric restriction and L-carnitine administration improves insulin sensitivity in patients with impaired glucose metabolism.\nMolfino A 1, Cascino A , Conte C , Ramaccini C , Rossi Fanelli F , Laviano A .\nhttps://www.ncbi.nlm.nih.gov/pubmed/20467011\n[17] Hypertension. 2009 Sep;54(3):567-74. doi: 10.1161/HYPERTENSIONAHA.109.132522. Epub 2009 Jul 20.\nAmeliorating hypertension and insulin resistance in subjects at increased cardiovascular risk: effects of acetyl-L-carnitine therapy.\nRuggenenti P 1, Cattaneo D , Loriga G , Ledda F , Motterlini N , Gherardi G , Orisio S , Remuzzi G .\nhttps://www.ncbi.nlm.nih.gov/pubmed/19620516\n[18] Ann Nutr Metab. 2008;52(4):335-8. doi: 10.1159/000151488. Epub 2008 Aug 19.\nEffect of oral L-carnitine administration on insulin sensitivity and lipid profile in type 2 diabetes mellitus patients.\nGonzález-Ortiz M 1, Hernández-González SO , Hernández-Salazar E , Martínez-Abundis E .\nhttps://www.ncbi.nlm.nih.gov/pubmed/18714152\n[19] Am J Clin Nutr. 2007 Dec;86(6):1738-44.\nL-Carnitine treatment reduces severity of physical and mental fatigue and increases cognitive functions in centenarians: a randomized and controlled clinical trial.\nMalaguarnera M 1, Cammalleri L , Gargante MP , Vacante M , Colonna V , Motta M .\nhttps://www.ncbi.nlm.nih.gov/pubmed/18065594\n[20] Arch Gerontol Geriatr. 2008 Mar-Apr;46(2):181-90. Epub 2007 Jul 20.\nAcetyl L-carnitine (ALC) treatment in elderly patients with fatigue.\nMalaguarnera M 1, Gargante MP , Cristaldi E , Colonna V , Messano M , Koverech A , Neri S , Vacante M , Cammalleri L , Motta M .\nhttps://www.ncbi.nlm.nih.gov/pubmed/17658628\n[21] Drugs Aging. 2003;20(10):761-7.\nLevocarnitine administration in elderly subjects with rapid muscle fatigue: effect on body composition, lipid profile and fatigue.\nPistone G 1, Marino A , Leotta C , Dell’Arte S , Finocchiaro G , Malaguarnera M .\nhttps://www.ncbi.nlm.nih.gov/pubmed/12875611\n[22] J Clin Biochem Nutr. 2007 Nov;41(3):224-30. doi: 10.3164/jcbn.2007032.\nEffects of Citric Acid and l-Carnitine on Physical Fatigue.\nSugino T 1, Aoyagi S , Shirai T , Kajimoto Y , Kajimoto O .\nhttps://www.ncbi.nlm.nih.gov/pubmed/18299720\nShare on X (Opens in new window) X\nShare on Pinterest (Opens in new window) Pinterest', '[-0.058639705181121826, -0.0037595380563288927, -0.007056924514472485, 0.04539123550057411, 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(192, 'Pathological Blood Coagulation and Systemic Acidosis', 'https://drrobertyoung.com/pathological-blood-coagulation-and-systemic-acidosis/', 'The following micrographs reveal the unhealthy blood of a Type 1 diabetic in severe metabolic acidosis (lactic acid) of the blood and the interstitial fluids (pH of the Blood 7.2 and the interstitial fluids 7.2. Ideal is a pH of 7.365 as determined by the 3D Bio-electro scan – www.universalmedicalimaging.com). The following micrograph shows a green crystal indicating the body’s attempt to solidify the increased lactic acid (the green crystal) in order to protect the remaining healthy blood and body cells. Lactic acid in a major metabolic waste responsible for diabetes, heart disease and cancer.\nThe next picture shows the breakdown of the cellular membrane of all the red blood cells giving birth to mycoplasma bacteria born-out of the red blood cell body as indicated as black discs.\nThis is the condition of the red blood cells will increase the A1c or blood sugars on the surface and is always present in medically diagnosed Type 1 Diabetics! This increase in the A1c or sugar on the surface of the red blood cells is the result of metabolic and dietary acidosis in the the fluids of the blood and interstitial fluids of the Interstitium.\nThe origin or genesis of Type 1, 2 and 3 diabetes begins in the small and large bowels as shown in the next micrographs indicated by large round polymerized protein pools in the center of the coagulated blood smears. This pattern in the Dried Coagulated Blood is a sure sign of congestion and inflammation of the bowels that leads to diabetes, cancer and heart disease.\nTo reverse this congestive and inflammatory condition of the bowels that is mis-labeled as Type 1 or 2 or 3 diabetes you must first heal the bowels, then the blood and finally the interstitial fluids. The origin and genesis of diabetes begins in the bowels and NOT the pancreas as outlined in the pH Miracle for Diabetes.\nTo learn more read the peer-reviewed scientific article, “Pathological Blood Coagulation” by Dr. Robert O. Young CPT, MSc, DSc, PhD, Naturopathic Practitioner.\nTo order Dr. Young’s article on Pathological Blood Coagulation go to: https://www.amazon.com/gp/product/B01JNPLP9C/ref=dbs_a_def_rwt_hsch_vapi_taft_p3_i10\nTo learn how you can reverse Type 1, Type 2 and Type 3 Diabetes read, The pH Miracle for Diabetes: https://www.amazon.com/gp/product/0446691003/ref=dbs_a_def_rwt_hsch_vapi_taft_p1_i5\nTo learn more about live and dried blood analysis order the following book: https://www.amazon.com/gp/product/B01JVK48XE/ref=dbs_a_def_rwt_hsch_vapi_taft_p3_i9', '[-0.02639094553887844, -0.047293614596128464, -0.07411076128482819, 0.01432217936962843, 0.013162700459361076, 0.0069973343051970005, 0.08277583122253418, 0.14921970665454865, 0.04064904898405075, -0.03803976625204086, -0.08758958429098129, 0.019953204318881035, -0.020738879218697548, -0.014223615638911724, -0.10918232053518295, -0.057215429842472076, -0.0043394784443080425, 0.004999545868486166, -0.0707089900970459, 0.028374288231134415, 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(193, 'The Truth Shall Set You Free!', 'https://phoreveryoung.wordpress.com/2019/08/04/the-truth-shall-set-you-free/', 'Ian Jacklin & Robert O Young PhD, get each others back on the pH cure. It’s true. Dr. Bernardo’s dad knew it. Dr. B taught me and Dr. Young backs it up. Get the protocols in our books. I Cure Cancer by Ian Jacklin and The pH Miracle for Cancer by Dr. Young. Learn about donating electrons and keeping your body healthy, fit and young!\nYou don’t need a gun when you have a camera and microscope to record the truth!\nTo learn more read The pH Miracle, The pH Miracle revised and updated, The pH Miracle for Weight Loss, The pH Miracle for Diabetes, The pH Miracle for the Heart, The pH Miracle for Cancer, The Cancer Solution, Sick and Tired Reclaim Your Inner Terrain, and the Back to the House of Health Books 1 and 2.\nwww.phmiracleproducts.com and www.drrobertyoung.com\nShare on X (Opens in new window) X\nShare on Pinterest (Opens in new window) Pinterest', '[-0.03719913586974144, 0.040398914366960526, 0.050339192152023315, 0.0026673090178519487, 0.010746912099421024, 0.023546066135168076, -5.6239798141177744e-05, 0.08120536059141159, -0.01624457724392414, -0.0487976111471653, 0.016452034935355186, 0.1349533200263977, -0.013301498256623745, 0.005144319497048855, -0.053636182099580765, 0.011684664525091648, -0.048719536513090134, 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(194, 'Pure Energy', 'https://phoreveryoung.wordpress.com/2019/04/21/pure-energy/', 'THE INCREDIBLE POWER OF MOLECULAR REDUCED MAGNESIUM BRINGING CUTTING EDGE NUTRITIONAL SCIENCE TO YOU NOW!’\nTHE MOST POWERFUL SELECTIVE ANTI-OXIDANT, ALKALIZER, ENERGIZER AND CELL HYDRATOR IN THE WORLD!\nWITH AN INSTANT IMPACT ON ALKALINITY, BLOOD QUALITY AND ENERGY!\nNearly every health condition and chronic dis-ease have three factors often associated with them:\nExcess metabolic and dietary acidity in the interstitial body fluids That excess interstitial acidity triggers tissue inflammation This tissue inflammation activates cell signaling to release anti-oxidants or acid buffers to protect the tissues, glands and organs. Medical Science has been looking at a way of tackling these three issues for quite some time now.\nWe have recently discovered that molecular activated and reduced magnesium (MgOH-) that is potentiated with lithium ions tackles all of these problems! It acts as a super anti-oxidant or a super anti-acid, a super anti-inflammatory, a super neutralizer of excess metabolic, dietary, respiratory and environmental acids, acting alone as its own cell signaling molecule!\nIn our body’s, activated and reduced magnesium that carries an extra electron (MgOH-) acts as a very powerful and selective anti-oxidant helping to prevent cellular damage from acid caused inflammation, protecting DNA and combating out of control metabolic and dietary acids that are the primary cause of cellular degeneration.\nThis is how I know proper alkalizing nutritional support in any cancerous condition is important in the prevention and treatment of cancer, the metastasis of cancer, and the shrinking of a cancerous cyst or mass without chemotherapy and-or radiation. The best part about these alkalizing nutritional treatments is they are helpful in most, if not in all cancerous conditions.”\nThere are now well over 1000 studies from peer reviewed medical journals discussing the countless health benefits from elemental magnesium.\nActivated and reduced magnesium (MgOH-) has been shown to reduce inflammation and joint discomfort, increase stamina and energy.\nActivated and reduced magnesium (MgOH-) has shown promise to be cardio protective, neuro-protective, offer intestinal protection, skin rejuvenation and many more conditions caused by metabolic and dietary acids that are not properly eliminated via the four channels of elimination – skin, lungs, bowels and kidneys.\nActivated and reduced magnesium (MgOH-) also acts as a powerful cell signalling molecule to maintain our cellular communications system. The interference in this through excess metabolic and dietary acidity is the cause of many health and fitness problems in the body.\nTHE BENEFITS OF ACTIVATED AND REDUCED MAGNESIUM\nThe health benefits of activated and reduced magnesium are new to the world.\nThis was because, until NOW, the delivery was only attainable through:\nElectron-enriched ionized water (inefficiently delivered from water current electrical\nElectrons release through hydroxyl Gas from a metallic cylinder under high pressure.\nThe problem with these delivery systems is that electrons disappear very quickly and therefore are not very accessible to our cells to buffer or neutralize metabolic and/or dietary acids.\nBasic chemistry has demonstrated that in the stomach, the generation of electrons carried by sodium bicarbonate is more complete and faster than any other means.\nFAST RED BLOOD CELL TRANSFORMATION: Immediate improved difference which is viewable using phase contrast microscopy in just five minutes.\nALKALIZING EFFECTs: Pure Energy(TM) is a potent alkalizer serving to effectively neutralize all types of metabolic and dietary acids in our body including lactic acid.\nNEGATIVE-CHARGED ORP EFFECT: Pure Energy(TM) has a high Negative-Charged Oxidative Reduction Potential. My electron tests show an ORP of up to -1000mV! In this case the negative-charge is good, not bad and represents a concentration of free electron energy! As we age, we oxidize or ferment, like an old car that rusts. Things that oxidize have a positive ORP or positive-charge. Therefore if we want to slow down the aging (rusting or fermenting) process, then it’s a good to ingest foods, liquids or supplements that carry a negative-charged ORP. This was only available before with an expensive water ionizers – but is now available in supplement form to all through my latest invention called Pure Energy(TM).\nBIO-AVAILABLE: Pure Energy(TM) is activated and reduced magnesium potentiated with lithium and is 100% bio-available, so it will act on ALL of the body’s fluids, tissues, glands and organs.\nANTI-INFLAMMATORY: Pure Energy(TM) acts as a powerful anti-inflammatory in the body because it neutralizes the metabolic, respiratory, dietary and environmental acids that cause inflammation.\nENHANCED CELLULAR HYDRATION: Pure Energy(TM) has been shown to increase cellular hydration by enhancing the ability to move alkalizing extracellular water into the cell.\nENERGY PRODUCTION: Pure Energy(TM) when added to distilled or purified water will activate and reduce and potentiated with lithium as it releases electron-energy which becomes available to the mitochondria in our cells. Activated and reduced magnesium potentiated with lithium also increases electron stores in the liver and may improve functioning of all organs in the body by increasing stores of available electron energy.\nANTI-AGING: Aging is created by the body being broken down by metabolic and dietary acids. By having a continual supply of electrons released from activated and reduced magnesium potentiated with lithium, the body can use the increased electrons to neutralize the acids that cause aging and slow down the aging process.\nSPORTS RECOVERY & LEGAL PERFORMANCE ENHANCER: By increasing alkalizing cellular hydration, reducing the acids that cause inflammation and most importantly reducing the lactic acid by up to 18% that causes inflammation that leads to pain, Pure Energy(TM) is a powerful sports performance and recovery enhancer.\nPOWERFUL ANTIOXIDANT: Due to its extremely small size (0.24 Trillionth of a Meter), it can spread throughout the body in seconds and penetrate all tissue, cells, and cell components providing rapid protection from metabolic and dietary acids.\nSELECTIVE ANTIOXIDANT: Activated and reduced magnesium has special SELECTIVE properties that allow the abundant release of electrons to deal with the “bad” acid but leave the “good” alkalizing buffers, such as sodium bicarbonate and hydrogen peroxide to do their tissue protective jobs.\nHOLISTIC ANTIOXIDANT: Due to its molecular size, Pure Energy(TM) can provide protection to the inside and outside of body cells; external surface of the cell membrane (lipid-bi-layer), the extra-cellular matrix, plasma, interstitial fluids and all external surfaces of cells, organs, and all tissue.\nPURE ENERGY(TM) ENABLES THE PRODUCTION OF YOUR BODY’S OWN ANTIOXIDANTS: Acts as a natural Nrf2 transcription factor activator that allows the body to make its own antioxidant compounds (e.g., superoxide dismutase (SOD), catalase, and glutathione peroxidase).\nCELL SIGNALING: Cell signaling is a way the body can send a message to different parts of the body to supply it with required red blood cells. The released hydroxyl ions or OH- that releases an extra electron has recently been found to act as a cell signalling molecule. The release of this electron from reduced and activated magnesium acts as a powerful antioxidant and anti-inflammatory.\nNATURAL & SAFE: Pure Energy(TM) contains natural mineral ingredients and tested to be 100% safe, even at high doses. There are no known negative direct or side-effects.\nHOW TO PREPARE THE PURE ENERGY(TM) MAGNESIUM FOR ACTIVATION AND REDUCTION:\n1) Take a 12 ounce surgical stainless steel bottle and fill the bottle to the top with distilled or purified water. Make sure there is no air at the top of the bottle where electrons might escape.\n2) Drop one Pure Energy(TM) magnesium tablet in the water and immediately seal the bottle with the screw on top in order to trap all the electrons inside.\n3) Let the distilled or purified water sit for 2 hours while the magnesium activates and reduces.\n4) When you are ready to drink the Pure Energy(TM) activated and reduced magnesium water you can take off the lid or you can flip up the straw to drink. Drink the whole 12 ounces once opened.\nPlease note that the Pure Energy(R) activated reduced magnesium water is stable and drinkable for up to 1 year as long as the cap or the lid has not been opened.\nDrink one 12 ounce Pure Energy(TM) activated reduced magnesium water in the morning and one 12 ounce Pure Energy(TM) activated reduced magnesium water in the afternoon.\nShare on X (Opens in new window) X\nShare on Pinterest (Opens in new window) Pinterest', '[-0.0685117095708847, -0.06350049376487732, 0.04573274403810501, 0.12029992043972015, 0.061005085706710815, -0.014849136583507061, 0.03192417323589325, 0.04763815179467201, -0.061045411974191666, 0.002753962529823184, -0.03588864952325821, 0.05089740827679634, 0.06448747962713242, -0.005181596614420414, 0.021927503868937492, 0.0053130025044083595, 0.07513146847486496, 0.03193316608667374, -0.11719667911529541, 0.0021254909224808216, 0.04546670988202095, -0.03671841323375702, 0.007236950099468231, 0.04001003876328468, 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(195, 'Fly Me To The Moon!', 'https://phoreveryoung.wordpress.com/2019/02/14/fly-me-to-the-moon/', 'LOVE! is the theory of everything. It is literally what makes the Moon rotate around the World and the World rotate around the Sun. Love is the unseen force and source of ALL Power in the Universe! It is YOUR love that makes me want to fly to the moon and play amongst the stars.\nClick here to listen: https://youtu.be/K9TKHgkZzF0\nI know you know of Albert Einstein’s theory of relativity but are you familiar with Einstein’s proposed theory of love that controls everything? His theory was magically brought to the Big Screen in two incredible movies several years ago, The Theory of Everything or the life of David Hawkins and the movie Interstellar. Both of these movies shared the ultimate and simple hypothesis of Einstein that there is a universal force that controls everything and that force is LOVE!\nI have been giving great thought concerning this theory of love in relationship to a Mother’s Love and how a Mother loves her new born child. From my own perspective, as a man, how could I ever understand this love and its power other than knowing it is real having been on the receiving end of my own Mother’s love. My Mother loved me dearly. She was always looking out for me and trying to protect me. It has been said that a child is like a Mother’s heart running around outside her body. This statement of love makes sense to me because a woman has the God given power to give of herself in love to bring into the World a manifestation of that love – a child! .\nIn the words of John Lennon’s song, “all YOU need is love, all YOU need is love, all YOU need is love, all YOU need is love.” Can you hear yourself singing this Beatles song in your head – that is the power of love.\nSeveral months ago and while I was asleep I felt this power of love in a dream where I saw and heard myself singing a song to you called, Fly Me To The Moon. In the morning when I awoke from my dream I was singing this love song again having never sung this song before wondering how this was possible. And then I remembered that from love my parents brought me into the World! From love God organized the Universe and Worlds without number! From love God gave HIS only begotten son! From love God promised each of us eternal life! From love the sun shines upon us all! And from love he brought you back into my life. For love is light and light is love and without love there would be no light just darkness! I suddenly realized that we long and live for love! Love is all we need and what makes our World go around.\nWhen Albert Einstein proposed the theory of relativity, very few understood him, and what I will reveal now to transmit to mankind will also collide with the misunderstanding and prejudice in the world. There is an extremely powerful force that, so far, science has not found a formal explanation to. It is a force that includes and governs all others, and is even behind any phenomenon operating in the universe and has not yet been identified by us. This universal force is LOVE.\nWhen scientists looked for a unified theory of the universe they forgot the most powerful unseen force. Love is Light, that enlightens those who give and receive it. Love is gravity, because it makes some people feel attracted to others. Love is power, because it multiplies the best we have, and allows humanity not to be extinguished in their blind selfishness. Love unfolds and reveals. For love we live and die. Love is God and God is Love.\nThis force explains everything and gives meaning to life. This is the variable that we have ignored for too long, maybe because we are afraid of love because it is the only energy in the universe that man has not learned to drive at will.\nTo give visibility to love, I made a simple substitution in my most famous equation. If instead of E = mc2, we accept that the energy to heal the world can be obtained through love multiplied by the speed of light squared, we arrive at the conclusion that love is the most powerful force there is!\nSo I ask YOU my Valentine! What is the ultimate answer? Need I tell you that it is LOVE! LOVING YOU and YOU LOVING ME with all our heart, mind and soul. Loving our neighbor as ourself. LOVING GOD with ALL our heart, might and strength. This is the ultimate answer to happiness and joy and the ultimate solution to any challenge that you or I might face individually or together.\nThis Valentines Day I want you to know that I desire to LOVE YOU with all my heart, mind and soul. I truly believe that the ultimate goal in life is NOT to acquire the things of this World but to ultimately embrace the power of love so that we might first love God, then ourselves, so that we might love others and especially love each other.\nMay the blessing of this unseen force fill our hearts with the power of true love this Valentines Day as we fly together to the moon and sing amongst the stars.\nhttp://www.drrobertyoung.com and http://www.phmiracleretreat.com\nPS Here is another Valentines Day Story I hope You enjoy! Just click below to read The Power of Love!\nhttps://phoreveryoung.wordpress.com/20 …/…/23/the-power-love/\nShare on X (Opens in new window) X\nShare on Pinterest (Opens in new window) Pinterest', '[-0.05959030240774155, -0.03785416856408119, 0.02804596722126007, 0.08455770462751389, 0.011023152619600296, 0.0543019063770771, 0.005872645415365696, 0.016516603529453278, 0.10963543504476547, 0.023850517347455025, -0.0042951032519340515, -0.020091041922569275, -0.06898893415927887, -0.015653545036911964, -0.011575579643249512, 0.01413501426577568, 0.022486863657832146, 0.008395383134484291, -0.1239347904920578, 0.06363481283187866, 0.009053670801222324, -0.06802010536193848, -0.018713349476456642, 0.004982681013643742, -0.11618907004594803, 0.16087250411510468, 0.021903349086642265, -0.015110713429749012, -0.08840283751487732, -0.010979214683175087, 0.045869190245866776, 0.034845732152462006, -0.017266616225242615, 0.004934112541377544, -0.04759901016950607, 0.053786102682352066, 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INSERT INTO `dr_young_all_articles` (`id`, `title`, `url`, `content`, `embedding`) VALUES
(196, 'The Life and Work of Dr. Bennie Johnson Will Live On pHorever!', 'https://phoreveryoung.wordpress.com/2019/01/31/the-life-and-work-of-dr-bennie-johnson-will-live-on-phorever/', 'The Life and Work of Dr. Bennie Johnson Will Live On pHorever!\nDr. Ben Johnson passed away on January 19th, 2019 from causes unknown. The important thing to remember is NOT how Dr. Ben died but how he LIVED!\nDr. Ben’s greatest virtues, that he will always be remembered, by his wife, children, grandchildren, friends, co-workers, associates and patients, was his kindness, generosity and charity. He was a true healer and always ready to help anyone in need, regardless of their ability to pay. He truly served others beginning from his heart and then his mind.\nWatch the following YouTube video as Dr. Ben Johnson and Dr. Robert O. Young share the latest technology for non-invasive and non-radioactive medical diagnostics, created, designed and offered by Dr. Galina Migalko, at the Universal Medical Imaging Group, in Valley Village, California. (www.universalmedicalimaging.com)\nListen carefully to Dr. Ben, as a voice from the grave, that will never be silenced, as he shares the future of non-invasive medical diagnostics and treatment, that is here today.\nThank you Dr. Ben for your fearless nature to speak ‘Truth to Power’.\nThe way you lived your life brings to my mind the beautiful words, that I believe, exemplifies your journey here on Planet Earth and now the Other World. These words are found in the King James Version of the Bible, 2 Timothy, Chapter 4, verses 7-8.\n7. You have fought a good fight, You have finished Your course, You have kept the faith:\n8. Henceforth there is laid up for YOU a crown of righteousness, which the Lord, the righteous judge, shall give YOU at that day: and not to YOU only, but unto all them also that love his appearing.\nMay God continue to bless you, Dr. Ben on your journey, without end to perfection and glory, beyond the vail of forgetfulness.\nYour brother and friend pHorever!\nShare on X (Opens in new window) X\nShare on Pinterest (Opens in new window) Pinterest', '[-0.05203244462609291, 0.021436037495732307, -0.056800369173288345, -0.013544919900596142, 0.019373683258891106, -0.05932902172207832, 0.054699283093214035, 0.036325059831142426, -0.025573132559657097, 0.0023559790570288897, -0.0622752383351326, 0.08846794068813324, 0.05672238767147064, 0.0276234932243824, -0.0720134750008583, 0.09624993056058884, -0.0003027695056516677, -0.02233804203569889, -0.03575678542256355, 0.10602908581495285, -0.08903726935386658, 0.08625387400388718, 0.03171053156256676, -0.014796189032495022, 0.016031155362725258, 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(197, 'Life is a Choice', 'https://phoreveryoung.wordpress.com/2018/12/10/life-is-a-choice/', 'The following are the top ten regrets that people make on their death beds!\n1) I wish that I’d had the courage to live a life true to myself and not the life others expected of me.\n2) I wish I hadn’t worked so hard.\n3) I wish I’d had the courage to express my true feelings.\n4) I wish I’d stayed in touch with my friends.\n5) I wish I’d let myself be happier.\n6) I wish I’d spent more time with my spouse and/or family.\n7) I wish I’d listened more and talked less.\n8) I wish I’d taken that dream trip.\n9) I wish I’d taken better care of my mental and physical health.\nIt is your life and your choice to be sad or to be happy. To do or not to do. Choose consciously, Choose wisely. Choose honestly. Choose health. Choose happiness. Choose to live your life with passion and with NO regrets! Choose to start today!\nOnly three days in Tel Aviv, Israel !!!!\nFrom December 10th to December 13th, 2018\nIndividual Scans or Full Body Medical Comprehensive, Non-Radioactive, Painless with Non-Radioactive and Non-invasive Scans which include:\n– Full Body Bio-Electro Interstitial Scan\n– Non-Invasive Quantitative Blood Analysis\nAll scans can be done at the same appointment time.\nShare on X (Opens in new window) X\nShare on Pinterest (Opens in new window) Pinterest', '[0.033966027200222015, 0.1156044751405716, 0.05327322706580162, -0.034099556505680084, 0.025355825200676918, -0.003011563792824745, 0.08325426280498505, 0.01790730655193329, 0.061399057507514954, 0.023649713024497032, -0.0373537577688694, -0.0068862708285450935, 0.02484898455440998, -0.01370167825371027, 0.024310274049639702, -0.0521712601184845, -0.11089207977056503, 0.020164938643574715, -0.05288591608405113, 0.13087517023086548, -0.0368693470954895, 0.027935996651649475, 0.051082074642181396, 0.028976671397686005, 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INSERT INTO `dr_young_all_articles` (`id`, `title`, `url`, `content`, `embedding`) VALUES
(198, 'The pH Miracle of Vitamin D3', 'https://phoreveryoung.wordpress.com/2018/07/11/the-ph-miracle-of-vitamin-d3-2/', 'Many years ago a clinical observation published in April 2000 in the Archives of Internal Medicine caught my attention. Dr. Anu Prabhala and his colleagues reported on the treatment of five patients confined to wheelchairs with severe weakness and fatigue. Blood tests revealed that all suffered from severe vitamin D deficiency. The patients received 50,000 IU vitamin D per week and all became mobile within six weeks. [1]\nAny discussion of vitamin D must begin with the discoveries of the Canadian-born dentist Weston A. Price. In his masterpiece Nutrition and Physical Degeneration, Dr. Price noted that the diet of isolated, so-called “primitive” peoples contained “at least ten times” the amount of “fat-soluble vitamins” as the standard American diet of his day. [2] Dr. Price determined that it was the presence of plentiful amounts of fat-soluble vitamins A and D in the diet, along with calcium, phosphorus and other minerals, that conferred such high immunity to tooth decay and resistance to disease in nonindustrialized population groups.\nPick up any popular book on vitamins and you will read that ten minutes of daily exposure of the arms and legs to sunlight will supply us with all the vitamin D that we need. Humans do indeed manufacture vitamin D from cholesterol by the action of sunlight on the skin but it is actually very difficult to obtain even a minimal amount of vitamin D with a brief foray into the sunlight. [4][5]\nUltraviolet (UV) light is divided into 3 bands or wavelength ranges, which are referred to as UV-C, UV-B and UV-A. [6] UV-C is the most energetic and shortest of the UV bands. It will burn human skin rapidly in extremely small doses. Fortunately, it is completely absorbed by the ozone layer. However, UV-C is present in some lights. For this reason, fluorescent and halogen and other specialty lights may contribute to skin cancer.\nUV-A, known as the “tanning ray,” is primarily responsible for darkening the pigment in our skin. Most tanning bulbs have a high UV-A output, with a small percentage of UV-B. UV-A is less energetic than UV-B, so exposure to UV-A will not result in a burn, unless the skin is photosensitive or excessive doses are used. UV-A penetrates more deeply into the skin than UV-B, due to its longer wavelength. Until recently, UV-A was not blocked by sunscreens. It is now considered to be a major contributor to the high incidence of non-melanoma skin cancers. [7] Seventy-eight percent of UV-A penetrates glass so windows do not offer protection.\nThe ultraviolet wavelength that stimulates our bodies to produce vitamin D is UV-B. It is sometimes called the “burning ray” because it is the primary cause of sunburn (erythema).\nHowever, UV-B initiates beneficial responses, stimulating the production of vitamin D that the body uses in many important processes. Although UV-B causes sunburn, it also causes special skin cells called melanocytes to produce melanin, which is protective. UV-B also stimulates the production of Melanocyte.\nVitamin D3, an important vitamin in weight loss and energy production. [8]\nThe reason it is difficult to get adequate vitamin D from sunlight is that while UV-A is present throughout the day, the amount of UV-B present has to do with the angle of the sun’s rays. Thus, UV-B is present only during midday hours at higher latitudes, and only with significant intensity in temperate or tropical latitudes. Only 5 percent of the UV-B light range goes through glass and it does not penetrate clouds, smog or fog.\nIt takes about 24 hours for UV-B-stimulated vitamin D to show up as maximum levels of vitamin D in the blood. Cholesterol-containing body oils are critical to this absorption process. [10]\nBecause the body needs 30-60 minutes to absorb these vitamin-D-containing oils, it is best to delay showering or bathing for one hour after exposure. The skin oils in which vitamin D is produced can also be removed by chlorine in swimming pools.\nThe current suggested exposure of hands, face and arms for 10-20 minutes, three times a week, provides only 200-400 IU of vitamin D each time or an average of 100-200 IU per day during the summer months. In order to achieve optimal levels of vitamin D, 85 percent of body surface needs exposure to prime midday sun. (About 100-200 IU of vitamin D is produced for each 5 percent of body surface exposed, although we actually want and need a minimum of 4,000 iu.) Light skinned people need 10-20 minutes of exposure while dark skinned people need 90-120 minutes. [11]\nLatitude and altitude determine the intensity of UV light. UV-B is stronger at higher altitudes. Latitudes higher than 30° (both north and south) have insufficient UV-B sunlight two to six months of the year, even at midday. [12] Latitudes higher than 40° have insufficient sunlight to achieve optimum levels of D during six to eight months of the year. In much of the US, which is between 30° and 45° latitude, six months or more during each year have insufficient UV-B sunlight to produce optimal D levels. In far northern or southern locations, latitudes 45° and higher, even summer sun is too weak to provide optimum levels of vitamin D. [13][15] A simple UV-B meter is available to determine UV-B levels where you live.\nVitamin D From Toxic Acidic Food that May Cause Sickness and/or Disease\nFor food sources to provide us with D the source must be sunlight exposed. With exposure to UV-B sunlight, vitamin D is produced from fat in the fur, feathers, and skin of animals, birds and reptiles. Carnivores get additional D from the tissues and organs of their prey. Lichen contains vitamin D and may provide a source of vitamin D in the UV-B sunlight-poor northern latitudes. [16] Vitamin D content will vary in the organs and tissues of animals, pigs, cows, and sheep, depending on the amount of time spent in UV-B containing sunlight and/or how much D is given as a supplement. Poultry and eggs contain varying amounts of vitamin D obtained from insects, fishmeal, and sunlight containing UV-B or supplements. Fish, unlike mammals, birds and reptiles, do not respond to sunlight and rely on vitamin D found in phytoplankton and other fish. Salmon must feed on phytoplankton and fish in order to obtain and store significant vitamin D in their fat, flesh, skin, and organs. Thus, modern farm-raised salmon, unless artificially supplemented, may be a poor source of this essential nutrient.\nModern diets usually do not provide adequate amounts of vitamin D; [17] partly because of the trend to low fat foods and partly because we no longer eat vitamin-D-rich foods like naturally reared poultry and fatty fish such as kippers, and herring.\nOften we are advised to consume the egg white while the D is in the yolk or we eat the flesh of the fish avoiding the D containing skin, organs and fat.\nSun avoidance combined with reduction in food sources contribute to escalating D deficiencies. Vegetarian and vegan diets are exceptionally poor or completely lacking in vitamin D predisposing to an absolute need for UV-B sunlight. Using food as one’s primary source of D is difficult to impossible.\nThe pH Miracle of Vitamin D3 Leads pH Miracles\nSunlight and vitamin D are critical to all life forms. Standard textbooks state that the principal function of vitamin D is to promote calcium absorption in the gut and calcium transfer across cell membranes, thus contributing to strong bones and a calm, contented nervous system. It is also well recognized that vitamin D aids in the absorption of magnesium, iron and zinc, as well as calcium. And finally, Vitamin D helps to reduce dietary, respiratory, environmental and metabolic acidic waste.\nActually, vitamin D does not in itself promote healthy bones. Vitamin D controls the levels of calcium in the blood. If there is not enough calcium in the diet, then it will be drawn from the bones to help to maintain the alkalinity of the blood plasma and the interstitial fluids of the Interstitium.\nVitamin D deficiency decreases biosynthesis and causes the pancreas to release of insulin as a buffer of excess acidity, including sugar and lactic acids. [21] Glucose intolerance has been inversely associated with the concentration of vitamin D in the blood. Thus, vitamin D may protect against both Type I and Type II diabetes. [22]\nThe risk of senile cataract is reduced in persons with optimal levels of D and carotenoids. [23]\nPCOS (Polycystic Ovarian Syndrome) has been corrected by supplementation of D and calcium. [24]\nVitamin D plays a role in regulation of both the “infectious” immune system and the “inflammatory” immune system. [25]\nLow vitamin D is associated with several autoimmune diseases including multiple sclerosis, Sjogren’s Syndrome, rheumatoid arthritis, thyroiditis and Crohn’s disease. [26][27]\nOsteoporosis is strongly associated with low vitamin D. Postmenopausal women with osteoporosis respond favorably (and rapidly) to higher levels of D plus calcium and magnesium. [28]\nD deficiency has been mistaken for fibromyalgia, chronic fatigue or peripheral neuropathy. [1][28-30]\nInfertility is associated with low vitamin D.3 [1] Vitamin D supports the male and female reproductive system and normal levels of estrogen in men and women. [32] PMS has been completely reversed by addition of calcium, magnesium and vitamin D. [33] Menstrual migraine is associated with low levels of vitamin D and calcium. [81]\nBreast, prostate, skin and colon cancer have a strong association with low levels of D and lack of sunlight. [34-38]\nActivated vitamin D in the adrenal gland regulates tyrosine hydroxylase, the rate limiting enzyme necessary for the production of dopamine, epinephrine and norepinephrine. Low D may contribute to chronic fatigue and depression. [39]\nSeasonal Affective Disorder has been treated successfully with vitamin D. In a recent study covering 30 days of treatment comparing vitamin D supplementation with two-hour daily use of light boxes, depression completely resolved in the D group but not in the light box group. [40]\nHigh stress may increase the need for vitamin D or UV-B sunlight and calcium. [41]\nPeople with Parkinsons and Alzheimers have been found to have lower levels of vitamin D. [42][43]\nLow levels of D, and perhaps calcium, in a pregnant mother and later in the child may be the contributing cause of “crooked teeth” and myopia. When these conditions are found in succeeding generations it means the genetics require higher levels of one or both nutrients to optimize health. [44-47]\nBehavior and learning disorders respond well to D and/or calcium combined with an adequate alkaline diet and trace minerals. [48][49]\nThus, high dietary levels of calcium, when D is insufficient, may contribute to calcification of the arteries, joints, kidney and perhaps even the brain. [52-54]\nWe have also postulated that vitamin D deficiency leads to the deposition of calcium in the arteries and hence atherosclerosis, noting that northern countries have higher levels of cardiovascular disease and that more heart attacks occur in winter months. [55-56]\nAdministration of dietary vitamin D or UV-B treatment has been shown to lower blood pressure, restore insulin sensitivity and lower cholesterol. [58-60]\nDid you ever wonder why some people can eat all they want and not get fat, while others are constantly battling extra pounds? The answer may have to do with vitamin D and calcium status. Sunlight, UV-B, and vitamin D normalize food intake and normalize the acid sugar in the blood. Weight normalization is associated with higher levels of vitamin D and adequate calcium. [61] Obesity is associated with vitamin-D deficiency. [62-64] In fact, obese persons have impaired production of UV-B-stimulated D and impaired absorption of food source and supplemental D. [65]\nWhen the diet lacks calcium, whether from D or calcium deficiency, there is an increase in fatty acid synthase, an enzyme that converts calories into fat. Higher levels of calcium with adequate vitamin D inhibit fatty acid synthase while diets low in calcium increase fatty acid synthase by as much as five-fold.\nIn one study, genetically obese rats lost 60 percent of their body fat in six weeks on a diet that had moderate calorie reduction but was high in calcium. All rats supplemented with calcium showed increased body temperature indicating a shift from calorie storage to calorie burning (thermogenesis). [61]\nThe assimilation and utilization of vitamin D is influenced by the kinds of fats we consume. Increasing levels of both polyunsaturated and monounsaturated fatty acids in the diet decrease the binding of vitamin D to D-binding proteins.\nSaturated fats, the kind found in butter, tallow and coconut oil, do not have this effect. Nor do the omega-3 fats. [66]\nD-binding proteins are key to local and peripheral actions of vitamin D. This is an important consideration as Americans have dramatically increased their intake of polyunsaturated oils (from commercial vegetable oils) and monounsaturated oils (from olive oil and canola oil) and decreased their intake of saturated fats over the past 100 years.\nIn traditional diets, saturated fats supplied varying amounts of vitamin D. Thus, both reduction of saturated fats and increase of polyunsaturated and monounsaturated fats may contribute to the current widespread D deficiency.\nTrans fatty acids, found in margarine and shortenings used in most commercial baked goods, should always be avoided. There is evidence that these fats can interfere with the alkaline buffering systems the body uses to convert vitamin D in the liver. [80]\nIn my clinical practice, I test for vitamin-D status in all the extra-cellular fluids first. If D is needed, I try to combine sunlight exposure with vitamin D and Vitamin D3 supplementation.\nSingle, infrequent, intense, skin exposure to Ultra-Blue Light (www.innerlightblue.com) for only 20 minutes will not cause sunburn and will not suppresses the immune system. In addition, cold-laser ultra-blue light for 20 minutes a day will normalize immune function, enhance NK-cell and T-cell production, reduce abnormal inflammatory responses typical of autoimmune disorders, and reduce occurrences of infectious disease. [26] [67][68-71]\nThus it is important to sunbathe frequently for short periods of time, when UV-B is present, rather than spend long hours in the sun at infrequent intervals. Adequate UV-B exposure or Innerlight Ultra-Blue Light will provide needed Vitamin D for the body and can be achieved in less time than it takes to cause any redness in the skin with direct sunlight. It is never necessary to burn or tan to obtain sufficient vitamin D.\nIf sunlight is not available in your area because of latitude or season, Ultra Blue Light made by Innerlight Blue (www.innerlightblue.com) can be used to provide a natural balance of Ultra Blue and Ultra Violet Light. Used according to instructions, these cold laser lights provide a safe equivalent of sunlight and will not cause burning or even heavy tanning. Tanning beds, on the other hand, are not acceptable as a means of getting your daily dose of vitamin D because they provide high levels of UV-A and very little UV-B.\nIf you have symptoms of vitamin-D insufficiency or are unable to spend time in the sun, due to season or lifestyle or prior skin cancer, consider adding a supplement of 50,000 IU daily of Vitamin pH Miracle D3 or use the Innerlight Blue Light for 20 minutes daily. [www.innerlightblue.com]\n[Kourtney Kardashian recently treated her followers to a selfie wearing a innerlight blue mask as she underwent blue light therapy.]\nHigher levels may be needed but should be recommended and monitored by your health care practitioner after testing serum 25(OH)D.\nSupplementation of Vitamin D3 is safe as long as you diet is alkalizing and contains adequate alkalizing minerals such a sodium, calcium, magnesium and potassium that you can supplement by taking the pH Miracle pHour Salts. [www.phoreveryoung.com]\nHigher amounts of calcium are important for anyone diagnosed with bone loss. Total daily calcium as a supplement may range from 1,500 mg to 2,000 mg depending on current bone status and your body size. Make the effort to split up your daily dose. Do not take all your calcium and magnesium once a day. A higher percentage of the calcium dose is absorbed if delivered in smaller, more frequent amounts. [82]\nClients on vitamin-D3 therapy report a wide range of beneficial results including increased energy and strength, resolution of hormonal problems, weight loss, an end to sugar cravings, blood sugar normalization and improvement of nervous system disorders.\nA paradoxical transient and non-complicating hyper-calciuria (more calcium in the urine) may occur when the program is first initiated. This resolves quickly when adequate calcium and other minerals are consumed. Two other temporary side effects may occur during the first several months of treatment. One is daytime sleepiness after calcium is taken. This usually resolves itself after about one week. The other condition is the reappearance of pain and discomfort at the site of old injuries, a sign of injury remodeling or proper healing, which may take some time to clear up.\nDoses used in clinical studies range from as little as 400 IU daily to 10,000-500,000 IU, given either as a single onetime dose or daily, weekly or monthly. Such large doses are given either as a prophylactic or because compliance is considered a problem. There seems to be some evidence that vitamin D works better, without toxicity, when given in lower, more physiologic doses of 2,000-4,000 IU daily rather than as 100,000 IU once a month. However, a single monthly dose of 100,000 IU did replete low levels of vitamin D in adolescents during winter. [77]\nMany consider D3 the preferred vitamin, having more biologic activity. Vitamin D3 as found in food or in human skin always comes with various metabolites or isomers that may have biological benefit.\nWhen humans take in vitamin D from food or sunlight, it is converted first in the liver to the form 25(OH)D and then in the kidney to 1,25(OH)D. These active forms of vitamin D are available by prescription and are given to patients with liver or kidney failure or those with an hereditary metabolic defect in vitamin-D conversion.\nClick here: https://www.youtube.com/edit?video_referrer=watch&video_id=xi14BufojOU\nRecent headlines is now touting vitamin D3 as the new wonder supplement, with claims ranging from its ability to reduce cancer risk to its link to cognitive function in older men. While studies show connections exist, experts debate the amount of vitamin D necessary for optimal health, however.\n“Low vitamin D status is linked to a number of different conditions,” said James C. Fleet, Ph.D. professor in the department of foods and nutrition at Purdue University. “These include certain cancers, muscle weakness and types I and II diabetes—possibly even schizophrenia and multiple sclerosis.”\nStudies also show a decrease in colon cancer with an increase in vitamin D status, and it seems protective against other acidic cancerous risks as well. “One theory is that vitamin D may indirectly inhibit pro-cancer pathways,” said Fleet. “The question is finding the protective level, which remains under some debate.”\nAlthough it remains controversial, 30 nanograms/milliliter (ng/mL) of vitamin D is associated with fewer fractures and falls, according to Karen Hansen, assistant professor of medicine within the rheumatology section at the University of Wisconsin. “Vitamin D deficiency causes osteoporosis by triggering decreased calcium absorption, secondary hyperparathyroidism, increased bone resorption and decreased bone mineral density.” Study variables and inconsistencies make further studies necessary. Currently, 700 to 800 International Units (IU) of vitamin D a day seems most effective.\nAccording to Dr. Young, “recommendations for an “adequate intake” of vitamin D3 should be at 20,000 IU’s per day for maintenance and 100,000 IU’s per day in any acute or chronic condition, including diabetes, MS, heart dis-ease and cancerous conditions.”\n* UV-B Meter: Sunsor, Inc. (800) 492-9815 Sunsor\n* pH Testing Papers: http://www.phoreveryoung.com\n* Vitamin D3: (760) 751-8321 http://www.phoreveryoung.com\n* Order Blue Light: Innerlight Blue: (760) 484-1075 http://www.innerlightblue.com\n* Order pH Miracle D3: phoreveryoung: (760) 751-8321 http://www.phoreveryoung.com\n* Order pH Miracle D3: pH Miracle Store: http://www.phmiraclestore.com\n* Order pH Miracle D3: pHm Life: http://www.phmlife.com\n* Information on Robert O Young: http://www.drrobertyoung.com\n*  Health Retreats: http://www.phmiracleretreat.com\n1. Prabhala A, Garg R, Dandona P. Severe myopathy associated with vitamin D deficiency in western New York. Arch.Intern.Med. 2000;160:1199-203.\n2. Price, Weston A. Characteristics of Primitive and Modernized Dietaries. Nutrition and Physical Degeneration. New Canaan, Connecticut: Keats Publishing, Inc 1989:256-81.\n3. Vieth R. Vitamin D supplementation, 25-hydroxyvitamin D concentrations, and safety [see comments]. Am.J.Clin.Nutr. 1999;69:842-56.\n4. Glerup H, Mikkelsen K, Poulsen L et al. Commonly recommended daily intake of vitamin D is not sufficient if sunlight exposure is limited. J.Intern.Med. 2000;247:260-8.\n5. Glerup H, Eriksen EF. [Vitamin D deficiency. Easy to diagnose, often overlooked (see comments)]. Ugeskr.Laeger 1999;161:2515-21. 6. Diffey BL. Solar ultraviolet radiation effects on biological systems. Phys.Med.Biol. 1991;36:299-328.\n7. Moan J, Dahlback A, Setlow RB. Epidemiological support for an hypothesis for melanoma induction indicating a role for UVA radiation. Photochem.Photobiol. 1999;70:243-7.\n8. Ranson M, Posen S, Mason RS. Human melanocytes as a target tissue for hormones: in vitro studies with 1 alpha-25, dihydroxyvitamin D3, alpha-melanocyte stimulating hormone, and beta-estradiol. J.Invest Dermatol.1988;91:593-8.\n9. Sayre, R. M., Dowdy, J. C., Shepherd, J., Sadig, I., Bager, A., and Kollias, N. Vitamin D Production by Natural and Artificial Sources. 1998. Orlando, Florida, Photo Medical Society Meeting. 3-1-1998. Ref Type: Conference Proceeding\n10. Holick MF. The cutaneous photosynthesis of previtamin D3: a unique photoendocrine system. J.Invest Dermatol. 1981;77:51-8.\n11. Matsuoka LY, Wortsman J, Haddad JG, Kolm P, Hollis BW. Racial pigmentation and the cutaneous synthesis of vitamin D [see comments]. Arch.Dermatol. 1991;127:536-8.\n12. Matsuoka LY, Wortsman J, Haddad JG, Hollis BW. In vivo threshold for cutaneous synthesis of vitamin D3. J.Lab Clin.Med. 1989;114:301-5.\n13. Season, latitude, and ability of sunlight to promote synthesis of vitamin D3 in skin. Nutr.Rev. 1989;47:252-3.\n14. Pettifor JM, Moodley GP, Hough FS et al. The effect of season and latitude on in vitro vitamin D formation by sunlight in South Africa. S.Afr.Med.J. 1996;86:1270-2.\n15. Webb AR, Kline L, Holick MF. Influence of season and latitude on the cutaneous synthesis of vitamin D3: exposure to winter sunlight in Boston and Edmonton will not promote vitamin D3 synthesis in human skin. J.Clin.Endocrinol.Metab 1988;67:373-8.\n16. Bjorn LO, Wang T. Vitamin D in an ecological context. Int.J.Circumpolar.Health 2000;59:26-32.\n17. Xue L, Lipkin M, Newmark H, Wang J. Influence of dietary calcium and vitamin D on diet-induced epithelial cell hyperproliferation in mice. J.Natl.Cancer Inst. 1999;91:176-81.\n18. Moon J. The role of vitamin D in toxic metal absorption: a review. J.Am.Coll.Nutr. 1994;13:559-64.\n19. Sardar S, Chakraborty A, Chatterjee M. Comparative effectiveness of vitamin D3 and dietary vitamin E on peroxidation of lipids and enzymes of the hepatic antioxidant system in Sprague-Dawley rats. Int.J.Vitam.Nutr.Res. 1996;66:39-45.\n20. Wiseman H. Vitamin D is a membrane antioxidant. Ability to inhibit iron-dependent lipid peroxidation in liposomes compared to cholesterol, ergosterol and tamoxifen and relevance to anticancer action. FEBS Lett. 1993;326:285-8.\n21. Bourlon PM, Billaudel B, Faure-Dussert A. Influence of vitamin D3 deficiency and 1,25 dihydroxyvitamin D3 on de novo insulin biosynthesis in the islets of the rat endocrine pancreas. J.Endocrinol. 1999;160:87-95.\n22. Baynes KC, Boucher BJ, Feskens EJ, Kromhout D. Vitamin D, glucose tolerance and insulinaemia in elderly men [published erratum appears in Diabetologia 1997 Jul;40(7):870]. Diabetologia 1997;40:344-7.\n23. Jacques PF, Hartz SC, Chylack LT, Jr., McGandy RB, Sadowski JA. Nutritional status in persons with and without senile cataract: blood vitamin and mineral levels. Am.J.Clin.Nutr. 1988;48:152-8.\n24. Thys-Jacobs S, Donovan D, Papadopoulos A, Sarrel P, Bilezikian JP. Vitamin D and calcium dysregulation in the polycystic ovarian syndrome. Steroids 1999;64:430-5.\n25. Abu-Amer Y, Bar-Shavit Z. Regulation of TNF-alpha release from bone marrow-derived macrophages by vitamin D [published erratum appears in J Cell Biochem 1994 Nov;56(3):426]. J.Cell Biochem. 1994;55:435-44.\n26. Cantorna MT. Vitamin D and autoimmunity: is vitamin D status an environmental factor affecting autoimmune disease prevalence? Proc.Soc.Exp.Biol.Med. 2000;223:230-3.\n27. Vogelsang H, Ferenci P, Woloszczuk W et al. Bone disease in vitamin D-deficient patients with Crohn’s disease. Dig.Dis.Sci. 1989;34:1094-9.\n28. Bettica P, Bevilacqua M, Vago T, Norbiato G. High prevalence of hypovitaminosis D among free-living postmenopausal women referred to an osteoporosis outpatient clinic in northern Italy for initial screening. Osteoporos.Int. 1999;9:226-9.\n29. Glerup H, Mikkelsen K, Poulsen L et al. Hypovitaminosis D myopathy without biochemical signs of osteomalacic bone involvement. Calcif.Tissue Int. 2000;66:419-24.\n30. Kyriakidou-Himonas M, Aloia JF, Yeh JK. Vitamin D supplementation in postmenopausal black women. J.Clin.Endocrinol.Metab 1999;84:3988-90.\n31. Uhland AM, Kwiecinski GG, DeLuca HF. Normalization of serum calcium restores fertility in vitamin D-deficient male rats. J.Nutr. 1992;122:1338-44.\n32. Kinuta K, Tanaka H, Moriwake T, Aya K, Kato S, Seino Y. Vitamin D is an important factor in estrogen biosynthesis of both female and male gonads. Endocrinology 2000;141:1317-24.\n33. Thys-Jacobs S. Micronutrients and the premenstrual syndrome: the case for calcium. J.Am.Coll.Nutr. 2000;19:220-7.\n34. Garland CF, Garland FC, Gorham ED. Calcium and vitamin D. Their potential roles in colon and breast cancer prevention. Ann.N.Y.Acad.Sci. 1999;889:107-19.\n35. John EM, Schwartz GG, Dreon DM, Koo J. Vitamin D and breast cancer risk: the NHANES I Epidemiologic follow-up study, 1971-1975 to 1992. National Health and Nutrition Examination Survey. Cancer Epidemiol.Biomarkers Prev. 1999;8:399-406.\n36. Miller GJ. Vitamin D and prostate cancer: biologic interactions and clinical potentials. Cancer Metastasis Rev. 1998;17:353-60.\n37. Gorham ED, Garland CF, Garland FC. Acid haze air pollution and breast and colon cancer mortality in 20 Canadian cities. Can.J.Public Health 1989;80:96-100.\n38. Kleibeuker JH, Van der MR, de Vries EG. Calcium and vitamin D: possible protective agents against colorectal cancer? Eur.J.Cancer 1995;31A:1081-4.\n39. Puchacz E, Stumpf WE, Stachowiak EK, Stachowiak MK. Vitamin D increases expression of the tyrosine hydroxylase gene in adrenal medullary cells. Brain Res.Mol.Brain Res. 1996;36:193-6.\n40. Gloth FM, III, Alam W, Hollis B. Vitamin D vs broad spectrum phototherapy in the treatment of seasonal affective disorder. J.Nutr.Health Aging 1999;3:5-7.\n41. Fujita T, Ohgitani S, Nomura M. Fall of blood ionized calcium on watching a provocative TV program and its prevention by active absorbable algal calcium (AAA Ca). J.Bone Miner.Metab 1999;17:131-6.\n42. Sato Y, Kikuyama M, Oizumi K. High prevalence of vitamin D deficiency and reduced bone mass in Parkinson’s disease. Neurology 1997;49:1273-8.\n43. Sato Y, Asoh T, Oizumi K. High prevalence of vitamin D deficiency and reduced bone mass in elderly women with Alzheimer’s disease. Bone 1998;23:555-7.\n44. Nikiforuk G, Fraser D. The etiology of enamel hypoplasia: a unifying concept. J.Pediatr. 1981;98:888-93.\n45. Taylor AN. Tooth formation and the 28,000-dalton vitamin D-dependent calcium- binding protein: an immunocytochemical study. J.Histochem.Cytochem. 1984;32:159-64.\n46. Price, Weston A. Primitive Control of Dental Caries. Nutrition and Physical Degeneration. New Canaan, Connecticut: Keats Publishing, Inc 1989:326-52.\n47. Price, Weston A. Prenatal Nutritional Deformities and Disease Types. Nutrition and Physical Degeneration. New Canaan, Connecticut: Keats Publishing, Inc 1989:326-52.\n48. Kozielec T, Starobrat-Hermelin B, Kotkowiak L. [Deficiency of certain trace elements in children with hyperactivity]. Psychiatr.Pol. 1994;28:345-53.\n49. Starobrat-Hermelin B. [The effect of deficiency of selected bioelements on hyperactivity in children with certain specified mental disorders]. Ann.Acad.Med.Stetin. 1998;44:297-314.\n50. Boucher BJ. Inadequate vitamin D status: does it contribute to the disorders comprising syndrome ‘X’? [published erratum appears in Br J Nutr 1998 Dec;80(6):585]. Br.J.Nutr. 1998;79:315-27.\n51. Schilli MB, Paus R, Czarnetzki BM, Reichrath J. [Vitamin D3 and its analogs as multifunctional steroid hormones. Molecular and clinical aspects from the dermatologic viewpoint]. Hautarzt 1994;45:445-52.\n52. Fujita T, Okamoto Y, Sakagami Y, Ota K, Ohata M. Bone changes and aortic calcification in aging inhabitants of mountain versus seacoast communities in the Kii Peninsula. J.Am.Geriatr.Soc. 1984;32:124-8.\n54. Sugihara N, Matsuzaki M, Kato Y. [Assessment of the relation between bone mineral metabolism and mitral annular calcification or aortic valve sclerosis-the relation between mitral annular calcification and post menopausal osteoporosis in elderly patients]. Nippon Ronen Igakkai Zasshi 1990;27:605-15.\n55. Segall JJ. Latitude and ischaemic heart disease [letter]. Lancet 1989;1:1146.\n56. Williams FL, Lloyd OL. Latitude and heart disease [letter]. Lancet 1989;1:1072-3.\n57. MacPherson A, Balint J, Bacso J. Beard calcium concentration as a marker for coronary heart disease as affected by supplementation with micronutrients including selenium. Analyst 1995;120:871-5.\n58. Krause R, Buhring M, Hopfenmuller W, Holick MF, Sharma AM. Ultraviolet B and blood pressure [letter]. Lancet 1998;352:709-10.\n59. Jorde R, Bonaa KH. Calcium from dairy products, vitamin D intake, and blood pressure: the Tromso Study. Am.J.Clin.Nutr. 2000;71:1530-5.\n60. Rostand SG. Ultraviolet light may contribute to geographic and racial blood pressure differences [see comments]. Hypertension 1997;30:150-6.\n61. Zemel MB, Shi H, Greer B, Dirienzo D, Zemel PC. Regulation of adiposity by dietary calcium. FASEB J. 2000;14:1132-8.\n62. Bell NH, Epstein S, Greene A, Shary J, Oexmann MJ, Shaw S. Evidence for alteration of the vitamin D-endocrine system in obese subjects. J.Clin.Invest 1985;76:370-3.\n63. Buffington C, Walker B, Cowan GS, Jr., Scruggs D. Vitamin D Deficiency in the Morbidly Obese. Obes.Surg. 1993;3:421-4.\n64. Liel Y, Ulmer E, Shary J, Hollis BW, Bell NH. Low circulating vitamin D in obesity. Calcif.Tissue Int. 1988;43:199-201.\n65. Wortsman J, Matsuoka LY, Chen TC, Lu Z, Holick MF. Decreased bioavailability of vitamin D in obesity. Am.J.Clin.Nutr. 2000;72:690-3.\n66. Bouillon R, Xiang DZ, Convents R, Van Baelen H. Polyunsaturated fatty acids decrease the apparent affinity of vitamin D metabolites for human vitamin D-binding protein. J.Steroid Biochem.Mol.Biol. 1992;42:855-61.\n68. Amento EP, Bhalla AK, Kurnick JT et al. 1 alpha,25-dihydroxyvitamin D3 induces maturation of the human monocyte cell line U937, and, in association with a factor from human T lymphocytes, augments production of the monokine, mononuclear cell factor. J.Clin.Invest 1984;73:731-9.\n69. Aslam SM, Garlich JD, Qureshi MA. Vitamin D deficiency alters the immune responses of broiler chicks. Poult.Sci. 1998;77:842-9.\n70. Corman LC. Effects of specific nutrients on the immune response. Selected clinical applications. Med.Clin.North Am. 1985;69:759-91.\n71. Muller K, Bendtzen K. 1,25-Dihydroxyvitamin D3 as a natural regulator of human immune functions. J.Investig.Dermatol.Symp.Proc. 1996;1:68-71.\n72. Barger-Lux MJ, Heaney RP, Dowell S, Chen TC, Holick MF. Vitamin D and its major metabolites: serum levels after graded oral dosing in healthy men. Osteoporos.Int. 1998;8:222-30.\n73. Rehak NN, Cecco SA, Csako G. Biochemical composition and electrolyte balance of “unstimulated” whole human saliva [In Process Citation]. Clin.Chem.Lab Med. 2000;38:335-43.\n74. Talbot JR, Guardo P, Seccia S et al. Calcium bioavailability and parathyroid hormone acute changes after oral intake of dairy and nondairy products in healthy volunteers. Osteoporos.Int. 1999;10:137-42.\n75. Heaney RP, Dowell MS, Barger-Lux MJ. Absorption of calcium as the carbonate and citrate salts, with some observations on method. Osteoporos.Int. 1999;9:19-23.\n76. Chesney RW. Vitamin D: can an upper limit be defined? J.Nutr. 1989;119:1825-8.\n77. Duhamel JF, Zeghoud F, Sempe M et al. [Prevention of vitamin D deficiency in adolescents and pre-adolescents. An interventional multicenter study on the biological effect of repeated doses of 100,000 IU of vitamin D3 (see comments)]. Arch.Pediatr. 2000;7:148-53.\n78. Davies PS, Bates CJ, Cole TJ, Prentice A, Clarke PC. Vitamin D: seasonal and regional differences in preschool children in Great Britain [published erratum appears in Eur J Clin Nutr 1999 Jul;53(7):584]. Eur.J.Clin.Nutr. 1999;53:195-8.\n79. Mariani E, Ravaglia G, Forti P et al. Vitamin D, thyroid hormones and muscle mass influence natural killer (NK) innate immunity in healthy nonagenarians and centenarians [published erratum appears in Clin Exp Immunol 1999 Jul;117(1):206]. Clin.Exp.Immunol.\n80. Enig, Mary G. Modification of Membrane Lipid Composition and Mixed-Function Oxidases in Mouse Liver Microsomes by Dietary Trans Fatty Acids. 1984. University Microfilms International. Ann Arbor, Michigan.\n81. Thys-Jacobs S. Vitamin D and calcium in menstrual migraine. Headache 1994;34:544-6.\nShare on X (Opens in new window) X\nShare on Pinterest (Opens in new window) Pinterest', '[-0.061342574656009674, -0.0059982589446008205, -0.006283093709498644, 0.04619608446955681, -0.04147832840681076, -0.05066239461302757, -0.019002284854650497, 0.09863487631082535, -0.036112964153289795, 0.1216076985001564, 0.0015151965199038386, 0.03784007951617241, 0.009435311891138554, 0.01712154410779476, -0.02501215599477291, -0.05285508930683136, 0.045444998890161514, 0.06579526513814926, -0.09032613039016724, 0.10323827713727951, -0.08686003088951111, 0.06562190502882004, 0.025804411619901657, -0.0012077400460839272, 0.008710822090506554, 0.02614251710474491, 0.02851293422281742, -0.06761855632066727, 0.04030117765069008, 0.11982639878988266, -0.04062564671039581, 0.011150222271680832, 0.06748074293136597, -0.0948815569281578, -0.0013628897722810507, 0.013203759677708149, 0.04591751843690872, 0.017757060006260872, -0.12391682714223862, -0.009810400195419788, 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INSERT INTO `dr_young_all_articles` (`id`, `title`, `url`, `content`, `embedding`) VALUES
(199, 'The pH Miracle Performance Center', 'https://phoreveryoung.wordpress.com/2018/05/19/the-ph-miracle-performance-center/', 'The pH Miracle Performance Center\nTotal personalization, maximum results\nThe pH Miracle Performance Center powered by Acquaforte is a centre of excellence for professional and amateur sports players, who want a personalized training strategy to achieve the best results in terms of muscle balance, personalized improvement in their individual sport, weight loss and general health, fitness and well-being.\nUnder the guidance of an extraordinary and close-knit team of athletics trainers, sports doctors, massage therapists, osteopaths, chiropractors and nutritionists, the Performance Center provides the most accurate analysis of every pathological condition and then an effective course of treatment , even when time is short, such as on holiday.\nEach type of training is designed to the needs of the guests and takes place in the state-of-the-art Fitness Room at Acquaforte Thalasso & Spa. Depending on the different cases in question, activity in the gym is combined with the thalassotherapy course , which, in a weightless environment, allows you to effortlessly perform a series of exercises that are totally effective for getting your body back in shape.\n#ph, #phmiracle, #alkalinelifestyleandiet, #alkaline, #alkalinebydesign, #acidicbyfunction, #drrobertoyoung, #dryoung, #phmiracleretreat, #alkalineretreat, #drgalina,\nShare on X (Opens in new window) X\nShare on Pinterest (Opens in new window) Pinterest', '[-0.04820273071527481, 0.0008424489060416818, 0.020413555204868317, 0.0346534326672554, -0.042948175221681595, 0.043551672250032425, 0.00793133582919836, 0.049553971737623215, -0.014482622966170311, -0.055114712566137314, 0.0017592001240700483, 0.03847920522093773, 0.030833564698696136, 0.040184225887060165, 0.050661601126194, -0.02906309813261032, 0.05945518612861633, 0.044568583369255066, -0.05410953238606453, -0.013196922838687897, 0.04483265429735184, 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(200, 'Using Alkalizing Herbs in the Prevention, Treatment and Reversal of Any Cancerous Condition', 'https://phoreveryoung.wordpress.com/2016/01/16/using-herbs-in-the-prevention-and-treatment-of-any-cancerous-condition/', 'Using Alkalizing Herbs in the Prevention, Treatment and Reversal of Any Cancerous Condition\nDr. Robert O. Young MSc., DSc., Ph.D, ND\n“The cure for cancer is found in its prevention NOT in its treatment”  Dr. Robert O. Young\n[Key words; cancer, chemotherapy, herbs, spices, natural cancer treatments, garlic, turmeric, ginger, cayenne, alkalizing, liver disease, oral cancer, prostate cancer, blood cancer, breast cancer, thyroid cancer, stomach cancer, skin cancer, pancreatic cancer, lung cancer, bowel cancer]\nMake room in your diet for the following foods and drinks to prevent cancer.  Why?  Because an ounce of prevention is worth more than a pound of cure.  The following are eleven herbs or spices that have been shown to be effective in the prevention, treatment and reversal of many cancerous conditions.\nSeveral large studies have found that those who eat more garlic are less likely to develop various kinds of cancer, especially in digestive organs such as the esophagus, stomach, and colon. Ingredients in the pungent bulbs may keep cancer-causing substances in your body from working, or they may keep cancer cells from multiplying. I recommend a clove a day may be helpful.[1=22 ]\nMost people know cayenne pepper for its spice. But it is actually an extraordinarily strong antioxidant and vaso-dialator. Studies have shown that by consuming cayenne pepper is highly alkaline and a powerful buffer of dietary and metabolic acids that cause cells to become cancerous. If you consume it regularly you can neutralize the acids that cause body cells to become cancerous.[23-29 ]\nMilk thistle is a crucial plant when it comes to liver health and cancer prevention. Milk thistle and the seeds from the plant can be used to eliminate acidic toxins that can bind to the liver, causing damage to the liver setting the stage for a cancerous condition. It protects the alkaline interstitial fluids that surround every body cell protecting them and indirectly preventing the formation of tumors, calcifications and/or cysts which make milk thistle a powerful antioxidant in the chelation of dietary and/or metabolic acids that cause cancer.[30-64 ]\n[Figure 4: Turmeric root and spice]\nThis orange-colored spice, a staple in Indian curries, contains an ingredient called curcumin (not the same as cumin) that might be useful in reducing cancer risk. According to the American Cancer Society, curcumin can inhibit some kinds of cancer cells in laboratory studies and slow the spread of cancer or shrink tumors in some animals. Turmeric is easy to find in grocery stores, and you can use it in a variety of recipes.[65-130 ]\nBloodroot is actually used in a medicine for treating cancer named Black Salve. You can use bloodroot on its own, because it has been shown in tests to be effective in shrinking of tumors.[131-159]\n[Figure 5: Bloodroot plant and flower]\nFeverfew was used in a study at a university in New York. The study found that it was great at killing off leukemia cells, even better than the actual cancer medication.[160-191]\n[Figure 6: Feverfew plant and flower]\nConsuming one tiny glass of wheatgrass a day either orally or even-better rectally has shown to dramatically increase the health of cancer patients and non-cancer patients alike. It is particularly useful for people who are suffering from the side effects of chemotherapy. It will help purify the blood from dietary and/or metabolic acids, improve blood and lymph circulation, increase the oxygen levels in the microenvironments, and help the body repair and continue to reduce acids loads in the extracellular fluids, interstitial fluids and intracellular fluids to prevent and/or reverse and spoiling of the body cells.[192-204]\nThis herb is always known as Butchers Broom and it is great at fighting cancer due to its active ingredient, ruscogenins. The active ingredient has been proven to shrink tumors and increase the cancer fighting cells in the body.[205-221 ]\n[Figure 8: Ruscus Aculeatus or Butchers Broom]\nSheep’s Sorrell can be used in people who are suffering the harmful effects of cancer medications. It helps the tissues rebuild and get back to the condition that they were in before the cancer and medication to use it was introduced. Some have suggested that it can be used to ward off cancer cells and keep them from growing.[222-224]\nThis herb is grown in China and has been proven to help with cancer on a couple of different levels. First it boosts your body’s immune system, which in turn helps it identify cancer cells. A study showed that cancer patients who took this herb survived twice as long.[225-250 ]\nA new study reveals ginger contains a pungent compound that could be up to 10,000 times more effective than conventional chemotherapy in targeting the cancer stem cells at the root of cancer malignancy. [251]\nIt reduces the expression of CD44/CD24 cancer stem cell surface markers in breast cancer spheroids (3-dimensional cultures of cells modeling stem cell like cancer)\nIt significantly affects the cell cycle, resulting in increased cancer cell death\nIt induces programmed cell death primarily through the induction of autophagy, with apoptosis a secondary inducer\nIt inhibits breast cancer spheroid formation by altering Notch signaling pathway through γ-secretase inhibition.\nIt exhibits cytotoxicity (cell killing properties) against monolayer (1-dimensional cancer model) and spheroid cells (3-dimensional cancer model)\n“In contrast [to 6-shagoal], taxol, even though was highly active in monolayer cells, did not show activity against the spheroids even at 10000 fold higher concentration compared to 6-shogoal.”\nThe monolayer model, which does not account for the complex immunity of actual cancer stem-cell based tumors against chemoagents like taxol, represents the old preclinical model of testing cancer treatments. The spheroid model, on the other hand, clearly shows that even 10,000 times higher concentrations of taxol are not capable of beating this ginger component at selectively targeting the root cause of the tumor malignancy .\nUnlike modern synthetically produced and patented chemicals , ginger, curcumin, garlic, and hundreds of other compounds naturally found in the human diet, have been “time-tested” as acceptable to the human body in the largest and longest running “clinical trials” known: the tens of thousands of years of direct human experience, spanning thousands of different cultures from around the world, that constitute human prehistory. These experientially-based “trials” are validated not by RCTs, or a peer-reviewed publication process, but by the fact that we all made it through this incalculably vast span of time to be alive here today. Consider also that if our ancestors made the wrong dietary choice by simply mistaking an edible berry for a poisonous one, the consequences could be deadly. This places even greater emphasis on how the “time testing” of dietary compounds was not an academic but a life-death affair, and by implication, how the information contained within various cultural traditions as “recipes” passed down from generation to generation are “epigenetic inheritance systems” no less important to our health and optimal gene expression as the DNA in our own bodies.\n[Figure 11: Cannabis plant with buds]\nA study published in the British Journal of Cancer , conducted by the Department of Biochemistry and Molecular Biology at Complutense University in Madrid, this study determined that Tetrahydrocannabinol (THC) and other cannabinoids inhibit tumour growth. They were responsible for the first clinical study aimed at assessing cannabinoid antitumoral action. Cannabinoid delivery was safe and was achieved with zero psychoactive effects. THC was found to decrease tumour cells in two out of the nine patients.[289] A study published in The Journal of Neuroscience examined the biochemical events in both acute neuronal damage and in slowly progressive, neurodegenerative diseases. They conducted a magnetic resonance imaging study that looked at THC (the main active compound in marijuana) and found that it reduced neuronal injury in rats. The results of this study provide evidence that the cannabinoid system can serve to protect the brain against neurodegeneration.[290] A study published in The Journal of Pharmacology And Experimental Therapeutics already acknowledged the fact that cannabinoids have been shown to possess antitumor properties. This study examined the effect of cannabidiol (CBD, non psychoactive cannabinoid compound) on human glioma cell lines. The addition of cannabidiol led to a dramatic drop in the viability of glioma cells. Glioma is the word used to describe brain tumour.  The study concluded that cannabidiol was able to produce a significant antitumor activity.[291] A study published in the journal Molecular Cancer Therapeutics outlines how brain tumours are highly resistant to current anticancer treatments, which makes it crucial to find new therapeutic strategies aimed at improving the poor prognosis of patients suffering from this disease. This study also demonstrated the reversal of tumour activity in Glioblastoma multiforme.[292]\nA study published in the US National Library of Medicine , conducted by the California Pacific Medical Centre determined that cannabidiol (CBD) inhibits human breast cancer cell proliferation and invasion. They also demonstrated that CBD significantly reduces tumour mass.[293] A study published in The Journal of Pharmacology and Experimental Therapeutics determined that THC as well as cannabidiol dramatically reduced breast cancer cell growth. They confirmed the potency and effectiveness of these compounds.[294] A study published in the Journal Molecular Cancer showed that THC reduced tumour growth and tumour numbers. They determined that cannabinoids inhibit cancer cell proliferation, induce cancer cell apoptosis and impair tumour angiogenesis (all good things). This study provides strong evidence for the use of cannabinoid based therapies for the management of breast cancer.[295] A study published in the Proceedings of the National Academy of Sciences of the United States of America (PNAS) determined that cannabinoids inhibit human breast cancer cell proliferation.[296]\nA study published in the journal Oncogene , by Harvard Medical Schools Experimental Medicine Department determined that THC inhibits epithelial growth factor induced lung cancer cell migration and more. They go on to state that THC should be explored as novel therapeutic molecules in controlling the growth and metastasis of certain lung cancers.[297 ] A study published by the US National Library of Medicine by the Institute of Toxicology and Pharmacology, from the Department of General Surgery in Germany determined that cannabinoids inhibit cancer cell invasion. Effects were confirmed in primary tumour cells from a lung cancer patient.  Overall, data indicated that cannabinoids decrease cancer cell invasiveness.[298 ] A study published by the US National Library of Medicine , conducted by Harvard Medical School investigated the role of cannabinoid receptors in lung cancer cells. They determined its effectiveness and suggested that it should be used for treatment against lung cancer cells.[299 ]\nA study published in the US National Library of Medicine illustrates a decrease in prostatic cancer cells by acting through cannabinoid receptors.[300] A study published in the US National Library of Medicine outlined multiple studies proving the effectiveness of cannabis on prostate cancer.[301] Another study published by the US National Library of Medicine determined that clinical testing of CBD against prostate carcinoma is a must. That cannabinoid receptor activation induces prostate carcinoma cell apoptosis. They determined that cannabidiol significantly inhibited cell viability.[302]\nA study published by the US National Library of Medicine results show cannabinoids are potent inhibitors of cellular respiration and are toxic to highly malignant oral Tumours. [306]\nA study published by the US National Library of Medicine determined that that THC reduces the viability of human HCC cell lines (Human hepatocellular liver carcinoma cell line) and reduced the growth.[307]\nA study published in The American Journal of Cancer determined that cannabinoid receptors are expressed in human pancreatic tumor cell lines and tumour biopsies at much higher levels than in normal pancreatic tissue. Results showed that cannabinoid administration induced apoptosis. They also reduced the growth of tumour cells, and inhibited the spreading of pancreatic tumour cells.[308]\nAccording to a 2004 report by Morgan, Ward, and Barton: “The contribution of cytotoxic chemotherapy to 5-year survival in adult malignancies. … survival in adults was estimated to be 2.3% in Australia and 2.1% in the USA.”Jun 16, 2014[309]\nMilner JA. Garlic: Its anticarcinogenic and antitumorigenic properties. Nutrition Reviews 1996; 54:S82–S86.\nRoss SA, Finley JW, Milner JA. 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Cannabis use in HIV for pain and other medical symptoms. J Pain Symptom Manage . 2005;29:358-367.\nM Guzmán, M J Duarte, C Blázquez, J Ravina, M C Rosa, I Galve-Roperh, C Sánchez, G Velasco and L González-Feria, A pilot clinical study of Δ9-tetrahydrocannabinol in patients with recurrent glioblastoma multiforme, British Journal of Cancer (2006) 95 , 197–203. doi:10.1038/sj.bjc.6603236 www.bjcancer.com Published online 27 June 2006.\nM. van der Stelt , W. B. Veldhuis , P. R. Bär , G. A. Veldink , J. F. G. Vliegenthart , and K. Nicolay, Neuroprotection by Δ9-Tetrahydrocannabinol, the Main Active Compound in Marijuana, against Ouabain-Induced In Vivo Excitotoxicity. The Journal of Neuroscience, 1 September 2001, 21 (17): 6475-6479.\nPaola Massi , Angelo Vaccani , Stefania Ceruti , Arianna Colombo , Maria P. Abbracchio and Daniela Parolaro , Antitumor Effects of Cannabidiol, a Nonpsychoactive Cannabinoid, on Human Glioma Cell Lines. Published online before print November 14, 2003 , doi: 10.1124/jpet.103.061002 JPET March 2004 vol. 308 no. 3 838-845.\nSofía Torres , Mar Lorente 1 , Fátima Rodríguez-Fornés 1 , Sonia Hernández-Tiedra 1 , María Salazar 1 , 2 , A Combined Preclinical Therapy of Cannabinoids and Temozolomide against Glioma.  doi: 10.1158/1535-7163.MCT-10-0688 Mol Cancer Therapy, January 2011 10; 90.\nMcAllister SD , Murase R , Christian RT , Lau D , Zielinski AJ , Allison J , Almanza C , Pakdel A , Lee J , Limbad C , Liu Y , Debs RJ , Moore DH , Desprez PY ,  Pathways mediating the effects of cannabidiol on the reduction of breast cancer cell proliferation, invasion, and metastasis. Breast Cancer Res Treat. 2011 Aug;129(1):37-47. doi: 10.1007/s10549-010-1177-4. Epub 2010 Sep 22.\nMaría M Caffarel , Clara Andradas , Emilia Mira , Eduardo Pérez-Gómez , Camilla Cerutti , Gema Moreno-Bueno , Juana M Flores , Isabel García-Real , José Palacios , Santos Mañes , Manuel Guzmán and Cristina Sánchez, Cannabinoids reduce ErbB2-driven breast cancer progression through Akt inhibition, Molecular Cancer 2010 9:196, 22 July 2010.\nLUCIANO DE PETROCELLIS, DOMINIQUE MELCK, ANTONELLA PALMISANO§, TIZIANA BISOGNO, CHIARA LAEZZA, MAURIZIO BIFULCO, AND VINCENZO DI MARZO, The endogenous cannabinoid anandamide inhibits human breast cancer cell proliferation.  Proc. Natl. Acad. Sci. USA, Vol. 95, pp. 8375–8380, July 1998 Pharmacology.\nA Preet, R K Ganju, J E Groopman,  Δ9-Tetrahydrocannabinol inhibits epithelial growth factor-induced lung cancer cell migration in vitro as well as its growth and metastasis in vivo. 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Ramos and Fernando J. Bianco , The role of cannabinoids in prostate cancer: Basic science perspective and potential clinical applications.  Indian J Urol. 2012 Jan-Mar; 28(1): 9–14.\nDe Petrocellis L , Ligresti A , Schiano Moriello A , Iappelli M , Verde R , Stott CG , Cristino L , Orlando P , Di Marzo V , Non-THC cannabinoids inhibit prostate carcinoma growth in vitro and in vivo: pro-apoptotic effects and underlying mechanisms. Br J Pharmacol. 2013 Jan;168(1):79-102. doi: 10.1111/j.1476-5381.2012.02027.\nKristin Gustafsson , Birger Christensson , Birgitta Sander and Jenny Flygare , Cannabinoid Receptor-Mediated Apoptosis Induced by R (+)-Methanandamide and Win55,212-2 Is Associated with Ceramide Accumulation and p38 Activation in Mantle Cell Lymphoma. Published online before print August 25, 2006 , doi: 10.1124/mol.106.025981 Molecular Pharmacology November 2006 vol. 70 no. 5 1612-1620.\nKristin Gustafsson, Xiao Wang1, Denise Severa1, Maeve Eriksson1, Eva Kimby2, Mats Merup2, Birger Christensson1, Expression of cannabinoid receptors type 1 and type 2 in non-Hodgkin lymphoma: Growth inhibition by receptor activation. International Journal of Cancer, Volume 123 , Issue 5 , pages 1025–1033 , 1 September 2008.\nJia W , Hegde VL , Singh NP , Sisco D , Grant S , Nagarkatti M , Nagarkatti PS , Delta9-tetrahydrocannabinol-induced apoptosis in Jurkat leukemia T cells is regulated by translocation of Bad to mitochondria. Mol Cancer Res. 2006 Aug;4(8):549-62.\nWhyte DA , Al-Hammadi S , Balhaj G , Brown OM , Penefsky HS , Souid AK .  Cannabinoids inhibit cellular respiration of human oral cancer cells. Pharmacology. 2010;85(6):328-35. doi: 10.1159/000312686. Epub 2010 Jun 2.\nVara D , Salazar M , Olea-Herrero N , Guzmán M , Velasco G , Díaz-Laviada , Anti-tumoral action of cannabinoids on hepatocellular carcinoma: role of AMPK-dependent activation of autophagy. Cell Death Differ. 2011 Jul;18(7):1099-111. doi: 10.1038/cdd.2011.32. Epub 2011, April 8.\nMorgan G 1, Ward R , Barton M .The contribution of cytotoxic chemotherapy to 5-year survival in adult malignancies. Clin Oncol (R Coll Radiol). 2004 Dec;16(8):549-60.\n– See more at: www.phoreveryoung.com\nThe Cure for Cancer? That’s an easy question to answer! The Cure for Cancer is Found in its Prevention NOT in its Treatment! – Dr. Robert O. Young\nDo you know what rotten apples, grapefruit or bananas look like? If you do then you know what cancer cells look like. Cancer cells are nothing more that healthy cells that are spoiling because of a compromised environment! Look at the picture below and you will see colorized cancerous body cells rotting in their toxic acidic environment.\nWhat compromises the internal environment of a human body that causes body cells to begin spoiling and rotting? The answer is simple! The body’s build-up of acidic metabolic and dietary waste that has not been properly eliminated through the four channels of elimination – urination, defecation, respiration and perspiration!\nShare on X (Opens in new window) X\nShare on Pinterest (Opens in new window) Pinterest', '[-0.02636130154132843, 0.022641576826572418, -0.01331249251961708, 0.0017871004529297352, 0.030029311776161194, 0.02528334967792034, -0.015121500939130783, 0.03834683820605278, 0.04807116463780403, -0.06461722403764725, 0.03302237018942833, -0.006239810958504677, 0.04848060756921768, -0.09401729702949524, -0.004318030085414648, 0.044953521341085434, 0.022615084424614906, 0.05172031745314598, -0.09305376559495926, -0.09138435125350952, -0.02055533602833748, 0.01325944997370243, 0.07994464784860611, 0.03824281692504883, 0.04161936417222023, -0.03593927249312401, 0.0001709893113002181, -0.02368916943669319, -0.07045020163059235, -0.015534883365035057, 0.1502951979637146, -0.016774829477071762, 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(201, 'Using Sodium and Potassium Bicarbonates in the Prevention and Treatment of ALL Sickness and Disease', 'https://phoreveryoung.wordpress.com/2016/01/09/7407/', 'Using Sodium and Potassium Bicarbonates in the Prevention and Treatment of ALL Sickness and Disease\nNaturopathic Practitioner – The pH Miracle Ti Sana Detox Medical Spa\nUsing Sodium and Potassium Bicarbonates in the Prevention and Treatment of ALL Sickness and Disease\nThis article suggests that the use sodium and potassium bicarbonates are non-toxic primary alkalizing agents in the prevention and  treatment of all cancers, kidney disease, liver disease, Type I & Type II diabetes, Lupus, heart disease, Pharmacological toxicosis, vascular surgery operation, tonsillar herniation due to cerebral edema, lactic acid toxicosis, and hyponatremia or low salt or loss of salts due to excessive or over-exercise!\n[Key words: cancer, diabetes, lupus, heart disease, vascular surgery, herniation, cerebral edema, lactic acid toxicosis, liver disease, kidney disease, hyponatremia, Pharmacological toxicosis]\n1) Severe diabetic ketoacidosis (1)\n2) Cardiopulmonary resuscitation (2)\n5) Peritoneal dialysis (5)6) Pharmacological toxicosis (6)\n8) Vascular surgery operations (8)\nMedics and emergency room medical doctors are accustomed to participating in a flurry of activity when trying to save a persons live after a cardiac arrest–inserting IVs and breathing tubes, performing defibrillation to restart the heart, etc. Sodium bicarbonate is a constant performer under such conditions and is more commonly used than magnesium injections, which is traditionally at the top of every doctor’s protocol for cardiac arrest.\nWhat is Latent Tissue Acidosis? Medical doctors are not taught in medical school and therefore do not understand or recognize latent tissue acidosis. They understand and recognize compensated acidosis and decompensated acidosis. In compensated acidosis, breathing increases in order to blow off more carbonic acid which decreases PCO2 because of the lowered carbonate or HCO3. When the breathing rate can no longer get any faster and when the kidneys can no longer increase its’ function to keep up with the acid load, then the blood pH starts to change from a pH of 7.365 to 7.3 then to 7.2. At a blood pH of 6.95 the heart relaxes and the client goes into a coma or dies.\nLatent “acidosis” is a condition that exists when there are not enough bases in the alkalophile glands because they have been used up in the process of neutralizing the acids adsorbed to the collagen fibers. This leads to compensated “acidosis.” This means the blood pH has not changed but other body systems have changed. This can then lead to decompensated “acidosis” where the alkaline reserves of the blood are used up and the pH of the blood is altered. Decompensated “acidosis” can be determined by testing the blood pH, urine pH and the saliva pH. The decrease in the alkaline reserves in the body  can occur because of hyper-proteinization, (eating meat and cheese!) or too much protein, and hyper-carbonization, or too much sugar or from excessive or over-excercise. This is why young athletes fall over dead or why 80 to 90 year old folks are all shrunk up and look like prunes. They have very little or no alkaline reserves in their alkalophile glands. When all the alkaline minerals are gone, so are you and your battery runs out of charge. The charge of your cellular battery can be measured by testing the ORP or the oxidative reduction potential of the blood, urine or saliva using an ORP meter. As you become more acidic this energy potential or ORP increases.\nHow Is Sodium Bicarbonate Created In The Body?\nThe parietal or cover cells of the stomach split the sodium chloride of the blood. The sodium ion is used to bind with water and carbon dioxide to form the alkaline salt, sodium bicarbonate or NaHCO3. The biochemistry is: H20 + CO2 + NaCl = NaHCO3 + HCL. This is why I call the stomach an alkalizing organ NOT an organ of digestion. The stomach DOES NOT digest the food or liquids we ingest but it alkalizes the foods and liquids we ingest.  We have one instrument in the human body to digest food and it is NOT the stomach it is your teeth.  Once we swallow our food or drink the stomach begins to prepare the food by alkalizing it in a bath of sodium bicarbonate.\nFor each molecule of sodium bicarbonate (NaHCO3) made, a molecule of hydrochloric acid (HCL) is made and secreted into the so-called digestive system – specifically, the stomach (the gastric pits in the stomach) – to be eliminated via the blood. Therefore HCL is an acidic waste product of sodium bicarbonate created by the stomach to alkalize the food and liquids ingested.\nExercise Creates Metabolic Acidic Waste Products Which Are Harmful To The Blood and Tissues\nWhen one exercises or over-exercises the body needs additional alkaline bicarbonate salts to buffer lactic acids.  The additional bicarbonate is created in the stomach lining to buffer the increased amounts of lactic acids produced as a waste product of metabolism.  The production of sodium bicarbonate will always leave an acidic waste product of hydrochloric acid in the gastric pits of the stomach leading to nausea, light headedness, dizziness, muddle thingking, and poor circulation.  If the excessive exercise continues this can then lead to a dificiency of mineral and bicarbonate salts (electrolytes lost through perspiration or urination) which may lead to latent tissue acidosis, pain, edema, hyponatrenia and death.\nBut how does something like sodium and/or potassium bicarbonate, so seemingly innocuous have such a dramatic effect? During prolonged or intense exercise muscles produce large amounts of acidic waste products, such as lactic acid, that lead to soreness, stiffness, fatigue and possible edema if these acids are not buffered and eliminated through urination or perspiration. Because sodium and potassium bicarbonate naturally reduces metabolic acids, it acts as a buffer against these performance-limiting by-products.\nIn Dr Folland’s study, swimmers who took the sodium bicarbonate knocked 1.5 seconds off their time for 200m, a difference that may seem insignificant to recreational swimmers but which is substantial at elite level.\n“At the last Olympics, the top four swimmers in the men’s 200m freestyle were separated by just 1.4 seconds,” Dr Folland says. “So, in theory, it could be the difference between winning a medal and not.”\nAnyone can try it, he says, but only those who are serious enough to monitor their times and progress in sports such as running, swimming or cycling may notice the few seconds advantage it might provide. “The increments of improvement are relatively small to the average person, although significant to someone who competes,” Dr Folland says.\nAthletes for years have sworn that taking a spoonful of bicarbonate of soda (baking soda) helps them to keep going for longer. For years, experts doubted that there was anything other than a placebo effect to these claims until they subjected the substance to rigorous examination. Most exercise scientists investigating the trend for “soda-doping” among athletes and gym-goers have shown that it offers significant benefits for endurance and speed.”\nAt Loughborough University, for instance, physiologists reporting in the June issue of the International Journal of Sports Medicine showed that swimmers who took baking soda about one hour before a 200m event were able to shave a significant time off their usual performances. Dr Jonathan Folland, who led the study, says that it is not uncommon for top swimmers to take sodium bicarbonate (another name for the substance) before a competition to give them an edge. Indeed, he showed that of nine swimmers tested, eight recorded their fastest times after ingesting a supplement of the common baking ingredient – sodium bicarbonate.\nWhere are Bicarbonates Created In The Human Body and Why?\nThe chloride ion from the sodium chloride (salt) binds to an acid or proton forming HCL as a waste product of sodium bicarbonate production. HCL has a pH of 1 and is highly toxic to the blood and tissues and the cause of indigestion, acid reflux, ulcers, diabetes, cancer, hyponatremia, edema, tonsilar herniation and death.  When large amounts of acids, including HCL, enter the stomach from a rich animal protein or dairy product meal, such as meat and cheese, or from starchy foods from root vegetables like potatoes or during extreme exercise, acid is withdrawn from the acid-base household. The organism would die if the resulting alkalosis – or NaHCO3 (base flood) or base surplus – created by the stomach was not taken up by the alkalophile glands (salivary glands, pancreas, kidney, pylorus glands, Brunner’s glands, Lieberkuhn glands and liver) that need these quick bases in order to build up their strong sodium bicarbonate secretions. These alkalizing glands and organs are the stomach, pancreas, Brunner’s glands (between the pylorus and the junctions of the bile and pancreatic ducts), Lieberkuhn’s glands in the liver and its bile with its strong acid binding capabilities which it has to release on the highly acidic meat, cheese, potato, acid water or metabolic and/or respiratory acids from over-exercise to buffer its strong acids of nitric, sulphuric, phosphoric, uric and lactic acids in daily metabolism, respiration and excessive or over-exercise.\nBicarbonate acts to stimulate the ATPase by acting directly on it.(12)\nThe simple household product used for baking, cleaning, bee stings, treating asthma, cancer and acid indigestion is so effective in treating disease that it prevents patients from having to be put on kidney dialysis. The findings have been published in the Journal of the American Society of Nephrology. Bicarbonate is a truly strong universal concentrated nutritional medicine that works effectively in many clinical situations that we would not normally think of. Bicarbonates of sodium and potassium are a prime emergency room and intensive care medicine that can save a person’s life in a heartbeat and it is also a supermarket item that you can take right off the shelf and use for more things than one can imagine – including diaper rash.\nA dialysis technician at the same center as Dr. Hariachar, who used to be on dialysis himself for 2 years as a result of kidney failure, had his kidneys miraculously start functioning to the point where dialysis was no longer needed. He states that he was prescribed oral doses of sodium bicarbonate throughout his treatment, and still takes it daily to prevent recurrences of kidney failure. Dr. Hariachar maintains though, that not everyone will be helped by taking bicarbonate. He says that those patients who have difficulty excreting acids, even with dialysis using a bicarbonate dialysate bath, that, “oral bicarbonate makes all the difference.”\nThe Stomach, Pancreas and Kidneys Naturally Produce Sodium Bicarbonate Every Day\nThe exocrine section of sodium bicarbonate from the stomach and the pancreas have been greatly ignored in the treatment of diabetes and cancer even though its impairment is a well documented condition. The stomach and the pancreas is primarily responsible for the production of sodium bicarbonate necessary for normal alkalization of food and liquids ingested. Sodium bicarbonate is so important for protecting the kidney’s that even the kidneys get into the act of producing sodium bicarbonate.  We now know the common denominator between hyponatremia, inflammation, edema, diabetes, kidney disease, and cancer is the lack of sodium and potassium bicarbonate or the body’s inability to produce sodium and potassium bicarbonate because of a lack of mineral salts in the diet. When the body is hit with reductions in sodium bicarbonate output by these three organs,’ acid conditions build up and then the entire body physiology begins to change from a state of oxygenation to fermentation. Likewise when acid build-up outstrips these organs normal sodium bicarbonate capacity, cellular, tissue, glandular and organ deterioration begins.\nThe stomach, pancreas and the kidneys alone produce about five hundred grams (about one pound) of sodium and/or potassium bicarbonate per day in an attempt to neutralize dietary and/or metabolic acid in the blood and interstitial fluids that surround the body cells.\nThe stomach, pancreas and the kidneys monitor and control the acidity or “acid-base” (pH) balance of the blood and tissues. If the blood and tissues are too acidic, the stomach and/or the kidney’s make sodium bicarbonate to restore the blood and tissue pH back to a delicate pH balance of 7.365. If the blood or tissues are too alkaline, then the kidney excretes sodium bicarbonate into the urine to restore the 7.365 alkaline balance. Acid-base balance is the net result of two processes, first, the removal of sodium bicarbonate subsequent to hydrogen ion production from the metabolism or dietary constituents; second, the synthesis of “new” sodium bicarbonate by the stomach and/or the  kidney’s.(13)  The stomach and kidneys pull salt, water and carbon dioxide from the blood to make sodium bicarbonate to maintain the alkaline design of the body during all functions of the body from the ingestion of food or drink to exercise.  The chemical formula is as follows:  NaCl + H2O + CO2 = NaHCO3 + HCL.  The waste product of sodium bicarbonate is hydrochloric acid which is eliminated by kidneys as an acidic excretion of the urine. It is considered that normal adults eating ordinary Western diets have chronic, low-grade acidosis which increases with age. This excess acid, or acidosis, is considered to contribute to many diseases and to contribute to the aging or rotting process. Acidosis occurs often when the body cannot produce enough sodium bicarbonate ions (or other alkaline compounds) to neutralize the acids in the body formed from metabolism and eating and drinking highly acid foods and drinks like chicken, pork, beef, dairy products, coffee, tea, alcohol, chocolate, soft drinks, just to name a few.  We are also testing bottled mineral water and finding that these waters are acidic and may contribute to overall tissue acidosis. Acid-buffering by means of base supplementation (The pH Miracle pHour Salts) of sodium bicarbonate is one of the major roles of dialysis. Sodium bicarbonate concentration in the dialysate (solution containing water and chemicals (electrolytes) that passes through the artificial kidney to remove excess fluids and wastes from the blood, also called “bath.”) should be personalized in order to reach a midweek pre-dialysis serum sodium bicarbonate concentration of 22 mmol/l.(14)  Use of sodium bicarbonate in dialysate has been shown in studies to better control some metabolic aspects and to improve both treatment tolerance and patients’ life quality. Sodium bicarbonate dialysis, unlike acetate-free biofiltration, triggers mediators of inflammation and apoptosis.(15)\nUnless a treatment actually removes acidic toxins  from the body and increases oxygen, water, and nutrients most medical interventions come to naught.\nThese metabolic and dietary acids must be excreted by the kidney’s because they contain sulfur, phosphorus, and/or nitrogen which cannot break down into water and carbon dioxide to be eliminated as weak acids. In their passage through the kidney’s these strong acids of ntric, sulphuric, phosphoric and uric acid must take a basic mineral with them because in this way they are converted into their neutral salts and don’t burn or destroy the kidney’s on their way out. This would happen if these strong acids were excreted in their free acidic form.\nSubstituting a sodium bicarbonate solution for saline infusion prior to administration of radiocontrast material seems to reduce the incidence of nephropathy .(18) Dr. Thomas P. Kennedy American Medical Association\nSodium and potassoum b icarbonate ions neutralize the acids that cause chronic inflammatory reactions . Hence, sodium and potassium bicarbonate are of benefit in the treatment of a range of chronic inflammatory and autoimmune diseases. Sodium and potassium bicarbonate are well-studied and used salts with known effects. Sodium and potassium bicarbonate are effective in treating poisonings or overdoses from many chemicals and pharmaceutical drugs by negating their cardiotoxic and neurotoxic effects.(19)  It is the main reason it is used by orthodox oncology – to mitigate the highly toxic effects of chemotherapy.\nSodium and potassium bicarbonates possess the property of absorbing heavy metals, dioxins and furans. Comparison of cancer tissue with healthy tissue from the same person shows that the cancer tissue has a much higher concentration of toxic chemicals, pesticides, etc. Sodium and potassium bicarbonate intravenous infusions are indicated in the treatment of metabolic acidosis, which may occur in severe renal disease, uncontrolled diabetes, and circulatory insufficiency due to shock or severe dehydration, extracorporeal circulation of blood, cardiac arrest, tonsillar herniation due to cerebral edema, severe primary lactic acidosis and hyponatremia due to excessive or over-exercise.  During heavy exercise, if the the resulting lactic acid is not adsorbed by the collagen fibers, the specific acid catchers of the body, the blood pH will drop and the body will go into a coma and the person will die.\nThe total collection of these fibers is the largest organ of the body called SCHADE, the colloidal connective tissue organ. NO liquid exchange occurs between the blood and the parenchyma cells, or in reverse, unless it passes through this connective tissue organ. This organ connects and holds everything in our bodies in place. This organ is composed of ligaments, tendons, sinew, and the finer fibers that become the scaffolding that holds every single cell in our bodies in place. When acids are stored in this organ, which includes the muscles, inflammation or edema and pain develop. The production of lactic acid is increased with excessive exercise and the ingestion of milk, cheese, yogurt, butter, ice cream, high sugar fruit and starchy root vegetables like potatoes.\nThat is why I have stated, “acid is pain and pain is acid or acid is edema and edema is pain”.  You cannot have one without the other. This is the beginning of latent tissue acidosis leading to irritation, inflammation, edema and degeneration of the cells, tissues and organs and eventual or sudden death.  It is why we are seeing so many amateur and professional atheletes pass out and die on the playing fields.  Metabolic, respiratory and gastrointestinal acids can and do kill and death can be overted by simply maintaining the alkaline design of the body fluids with protective hydration of alkaine sodium bicarbonate fluids.\nThe acid/alkaline balance is one of the most overlooked aspects of diagnostic medicine. In general, the world population is heavily acidic, excepting alkalarian vegans (those who ingest raw, organic green fruit, vegetables, mineral salts, alkaline water and unsaturated seed and nut oils), and even their bodies have to face increasing levels of environmental toxic exposure, which may contribute to an acidic pH condition of the blood and then tissues.\nAll enervation, under-performance, sensitivity, irritation, inflammation, edema, catarrh, induration, ulcerations, degeneration, aging and cancerous conditions are caused by a four letter word – ACID, which is an acronym which stands for:\nA = acidic food and drink, attitudes and activities, C = compromised internal acidic environment, I = illness and dis-ease, and, D = desire for more acidic foods, drinks, attitudes and activities, and the cycle repeats itself.[20]\nWe ingest acidic medicines to lessen the symptoms of our illness. We stimulate the body with unhealthy forms of energy providing quick, often temporary relief from our symptoms which begins the cycle all over again creating a very powerful pattern of poor health and dis-ease.\nThe pH Alkalizing Lifestyle and Diet is a low acid producing diet and lifestyle that focuses on the foundational principal that the body is alkaline by design and yet acidic by function. This makes this program the ultimate program for preventing and reversing aging and the onset of sickness and disease. I would say that the pH Alkalizing Lifestyle and Diet is the perfect diet and lifestyle for a longer healthier life.(20)\n1. Gamba, G., “Bicarbonate therapy in severe diabetic ketoacidosis. A double blind, randomized, placebo controlled trial.” (Rev Invest Clin 1991 Jul-Sep;43(3):234-8). Miyares Gom ez A. in “Diabetic ketoacidosis in childhood: the first day of treatment.” (An Esp Pediatr 1989 Apr;30(4):279-83)\n2. Levy, M.M., “An evidence-based evaluation of the use of sodium bicarbonate during cardiopulmonary resuscitation.” (Crit Care Clin 1998 Jul;14(3):457-83). Vukmir, R.B., Sodium bicarbonate in cardiac arrest: a reappraisal (Am J Emerg Med 1996 Mar;14(2):192-206). Bar-Joseph, G., “Clinical use of sodium bicarbonate during cardiopulmonary resuscitation–is it used sensibly?” (Resuscitation 2002 Jul;54(1):47-55).\n3. Zhang. L., “Perhydrit and bicarbonate improve maternal gases and acid-base status during the second stage of labor.” Department of Obstetrics and Gynecology, Xiangya Hospital, Hunan Medical University, Changsha 410008. Maeda, Y., “Perioperative administration of bicarbonated solution to a patient with mitochondrial encephalomyopathy.” (Masui 2001 Mar;50(3):299-303).\n4. Avdic. E., “Bicarbonate versus acetate hemodialysis: effects on the acid-base status.” (Med Arh 2001;55(4):231-3).\n5. Feriani, M., “Randomized long-term evaluation of bicarbonate-buffered CAPD solution.” (Kidney Int 1998 Nov;54(5):1731-8).\n6. Vrijlandt, P.J., odium bicarbonate infusion for intoxication with tricyclic antidepressives: recommended inspite of lack of scientific evidence. Ned Tijdschr Geneeskd 2001 Sep 1;145(35):1686-9). Knudsen, K., â€œEpinephrine and sodium bicarbonate independently and additively increase survival in experimental amitriptyline poisoning.” (Crit Car e Med 1997 Apr;25(4):669-74).\n7. Silomon, M., “Effect of sodium bicarbonate infusion on hepatocyte Ca2+ overload during resuscitation from hemorrhagic shock.” (Resuscitation 1998 Apr;37(1):27-32). Mariano, F., “Insufficient correction of blood bicarbonate levels in biguanide lactic acidosis treated with CVVH and bicarbonate replacement fluids.” (Minerva Urol Nefrol 1997 Sep;49(3):133-6).\n8. Dement’eva, I.I., “Calculation of the dose of sodium bicarbonate in the treatment of metabolic acidosis in surgery with and deep hypothermic circulatory arresta.” (Anesteziol Reanimatol 1997 Sep-Oct;(5):42-4).\n10. A sexually transmitted virus that causes cervical cancer is also to blame for half of all cases of cancer of the penis.\n1 1. www.nelm.nhs.uk/en/NeLM-Area/News/2009—July/20/ Bicarbonate-supplementation-may-slow-renal-decline-in-chronic-kidney-disease/\n1 2 . Origin of the Bicarbonate Stimulation of Torpedo Electric Organ Synaptic Vesicle ATPase. Joan E. Rothlein  1 Stanley M. Parsons. Department of Chemistry and the Marine Science Institute, University of California, Santa Barbara, Santa Barbara, California, U.S.A.\n1 3 . Levine DZ, Jacobson HR: The regulation of renal acid secretion: New observations from studies of distal nephron segments. Kidney Int 29:1099–1109, 1986\n1 4 . www.uptodate.com/patients/content/abstract.do?topicKey=~G/p55S8w8sQDwqG&refNum=28\n1 5 . www.ncbi.nlm.nih.gov/pubmed/16523427\n1 6 . news.bbc.co.uk/2/hi/health/7655405.stm\n17. Cancer Res. 2009 Mar 15;69(6):2260-8. Epub 2009 Mar 10. Bicarbonate increases tumor pH and inhibits spontaneous metastases. Robey IF , Baggett BK , Kirkpatrick ND , Roe DJ , Dosescu J , Sloane BF , Hashim AI , Morse DL , Raghunand N , Gatenby RA , Gillies RJ . Source: Arizona Cancer Center, University of Arizona, Tucson, Arizona, USA\n18.  JAMA 2004;291:2328-2334,2376-2377. www.urotoday.com/56/browse_categories/renal_transplantation_vascular_disease/ sodium_bicarbonate_may_prevent_radiocontrastinduced_renal_injury.html\n19. These include, Benzotropines (valium) cyclic antidepressants (amytriptayine), organophosphates, methanol (Methyl alcohol is a cheap and potent adulterant of illicit liquors) Diphenhydramine (Benedryl), Beta blockers (propanalol) Barbiturates, and Salicylates (Aspirin).   Poisoning by drugs that block voltage-gated sodium channels produces intraventricular conduction defects, myocardial depression, bradycardia, and ventricular arrhythmias. Human and animal reports suggest that hypertonic sodium bicarbonate may be effective therapy for numerous agents possessing sodium channel blocking properties, including cocaine, quinidine, procainamide, flecainide, mexiletine, bupivacaine, and others.\n20. www.phmiracle.com. Young.R.O., Young, S.R., The pH Miracle Revised and Updated, Hachett, 2010.\nShare on X (Opens in new window) X\nShare on Pinterest (Opens in new window) Pinterest', '[0.056171759963035583, 0.0036766701377928257, 0.04336028918623924, -0.026543142274022102, -0.05332338064908981, 0.04880448058247566, 0.00891886930912733, 0.11525461077690125, -0.019398685544729233, -0.03858012333512306, -0.03684752807021141, -0.0021623563952744007, 0.05130768194794655, 0.0008883061236701906, -0.04692139849066734, 0.008627860806882381, 0.07221294939517975, 0.049533065408468246, -0.04987414553761482, 0.05833166465163231, 0.09035561233758926, -0.010194966569542885, -0.016240453347563744, 0.054867301136255264, -0.026646273210644722, 0.05363733321428299, 0.029483402147889137, -0.034828994423151016, -0.005084451753646135, -0.00993918627500534, -0.04180452972650528, 0.02516378089785576, -0.023524083197116852, -0.07368064671754837, -0.032112572342157364, 0.0480053573846817, -0.08955152332782745, 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INSERT INTO `dr_young_all_articles` (`id`, `title`, `url`, `content`, `embedding`) VALUES
(202, 'Harvard Trained Immunologist Demolishes California Legislation That Terminates Vaccine Exemptions', 'https://phoreveryoung.wordpress.com/2016/01/04/harvard-trained-immunologist-demolishes-california-legislation-that-terminates-vaccine-exemptions/', 'Harvard Trained Immunologist Demolishes California Legislation That Terminates Vaccine Exemptions\nThe following open letter by a PhD Immunologist completely demolishes the current California legislative initiative to remove all vaccine exemptions. That such a draconian and cynical state statute is under consideration in the ‘Golden State’ is as shocking as it is predictable.  After all, it was mysteriously written and submitted shortly after the manufactured-in-Disneyland measles ‘outbreak’.\nThe indisputable science that is employed by Tetyana Obukhanych, PhD ought to be read by every CA legislator who is entertaining an affirmative vote for SB277.  Dr. Obukhanych skillfully deconstructs the many false and fabricated arguments that are advanced by Big Pharma and the U.S Federal Government as they attempt to implement a nationwide Super-Vaccination agenda.\nAn Open Letter to Legislators Currently Considering Vaccine Legislation from Tetyana Obukhanych, PhD in Immunology\nMy name is Tetyana Obukhanych. I hold a PhD in Immunology.  I am writing this letter in the hope that it will correct several common misperceptions about vaccines in order to help you formulate a fair and balanced understanding that is supported by accepted vaccine theory and new scientific findings.\nDo unvaccinated children pose a higher threat to the public than the vaccinated?\nIt is often stated that those who choose not to vaccinate their children for reasons of conscience endanger the rest of the public, and this is the rationale behind most of the legislation to end vaccine exemptions currently being considered by federal and state legislators country-wide. You should be aware that the nature of protection afforded by many modern vaccines – and that includes most of the vaccines recommended by the CDC for children – is not consistent with such a statement. I have outlined below the recommended vaccines that cannot prevent transmission of disease either because they are not designed to prevent the transmission of infection (rather, they are intended to prevent disease symptoms), or because they are for non-communicable diseases. People who have not received the vaccines mentioned below pose no higher threat to the general public than those who have, implying that discrimination against non-immunized children in a public school setting may not be warranted.\nIPV (inactivated poliovirus vaccine) cannot prevent transmission of poliovirus (see appendix for the scientific study, Item #1). Wild poliovirus has been non-existent in the USA for at least two decades. Even if wild poliovirus were to be re-imported by travel, vaccinating for polio with IPV cannot affect the safety of public spaces.  Please note that wild poliovirus eradication is attributed to the use of a different vaccine, OPV or oral poliovirus vaccine. Despite being capable of preventing wild poliovirus transmission, use of OPV was phased out long ago in the USA and replaced with IPV due to safety concerns.\nTetanus is not a contagious disease , but rather acquired from deep-puncture wounds contaminated with C. tetani spores. Vaccinating for tetanus (via the DTaP combination vaccine) cannot alter the safety of public spaces; it is intended to render personal protection only.\nWhile intended to prevent the disease-causing effects of the diphtheria toxin, the diphtheria toxoid vaccine (also contained in the DTaP vaccine) is not designed to prevent colonization and transmission of C. diphtheriae . Vaccinating for diphtheria cannot alter the safety of public spaces; it is likewise intended for personal protection only.\nThe acellular pertussis (aP) vaccine (the final element of the DTaP combined vaccine), now in use in the USA, replaced the whole cell pertussis vaccine in the late 1990s, which was followed by an unprecedented resurgence of whooping cough. An experiment with deliberate pertussis infection in primates revealed that the aP vaccine is not capable of preventing colonization and transmission of B. pertussis (see appendix for the scientific study, Item #2). The FDA has issued a warning regarding this crucial finding.[1]\nFurthermore, the 2013 meeting of the Board of Scientific Counselors at the CDC revealed additional alarming data that pertussis variants (PRN-negative strains) currently circulating in the USA acquired a selective advantage to infect those who are up-to-date for their DTaP boosters (see appendix for the CDC document, Item #3), meaning that people who are up-to-date are more likely to be infected, and thus contagious, than people who are not vaccinated.\nAmong numerous types of H. influenzae , the Hib vaccine covers only type b. Despite its sole intention to reduce symptomatic and asymptomatic (disease-less) Hib carriage, the introduction of the Hib vaccine has inadvertently shifted strain dominance towards other types of H. influenzae (types a through f). These types have been causing invasive disease of high severity and increasing incidence in adults in the era of Hib vaccination of children (see appendix for the scientific study, Item #4).  The general population is more vulnerable to the invasive disease now than it was prior to the start of the Hib vaccination campaign.  Discriminating against children who are not vaccinated for Hib does not make any scientific sense in the era of non-type b H. influenzae disease.\nHepatitis B is a blood-borne virus . It does not spread in a community setting, especially among children who are unlikely to engage in high-risk behaviors, such as needle sharing or sex. Vaccinating children for hepatitis B cannot significantly alter the safety of public spaces. Further, school admission is not prohibited for children who are chronic hepatitis B carriers. To prohibit school admission for those who are simply unvaccinated – and do not even carry hepatitis B – would constitute unreasonable and illogical discrimination.\nIn summary, a person who is not vaccinated with IPV, DTaP, HepB, and Hib vaccines due to reasons of conscience poses no extra danger to the public than a person who is.  No discrimination is warranted.\nHow often do serious vaccine adverse events happen?\nIt is often stated that vaccination rarely leads to serious adverse events. Unfortunately, this statement is not supported by science. A recent study done in Ontario, Canada, established that vaccination actually leads to an emergency room visit for 1 in 168 children following their 12-month vaccination appointment and for 1 in 730 children following their 18-month vaccination appointment (see appendix for a scientific study, Item #5).\nWhen the risk of an adverse event requiring an ER visit after well-baby vaccinations is demonstrably so high, vaccination must remain a choice for parents, who may understandably be unwilling to assume this immediate risk in order to protect their children from diseases that are generally considered mild or that their children may never be exposed to.\nCan discrimination against families who oppose vaccines for reasons of conscience prevent future disease outbreaks of communicable viral diseases, such as measles?\n“The apparent paradox is that as measles immunization rates rise to high levels in a population, measles becomes a disease of immunized persons.”[2]\nRe-vaccination cannot correct low-responsiveness: it appears to be an immuno-genetic trait.[4]  The proportion of low-responders among children was estimated to be 4.7% in the USA.[5]\nStudies of measles outbreaks in Quebec, Canada, and China attest that outbreaks of measles still happen, even when vaccination compliance is in the highest bracket (95-97% or even 99%, see appendix for scientific studies, Items #6&7). This is because even in high vaccine responders, vaccine-induced antibodies wane over time.  Vaccine immunity does not equal life-long immunity acquired after natural exposure.\nIt has been documented that vaccinated persons who develop breakthrough measles are contagious. In fact, two major measles outbreaks in 2011 (in Quebec, Canada, and in New York, NY) were re-imported by previously vaccinated individuals.[6] – [7]\nTaken together, these data make it apparent that elimination of vaccine exemptions, currently only utilized by a small percentage of families anyway, will neither solve the problem of disease resurgence nor prevent re-importation and outbreaks of previously eliminated diseases.\nIs discrimination against conscientious vaccine objectors the only practical solution?\nThe majority of measles cases in recent US outbreaks (including the recent Disneyland outbreak) are adults and very young babies, whereas in the pre-vaccination era, measles occurred mainly between the ages 1 and 15. Natural exposure to measles was followed by lifelong immunity from re-infection, whereas vaccine immunity wanes over time, leaving adults unprotected by their childhood shots. Measles is more dangerous for infants and for adults than for school-aged children.\nDespite high chances of exposure in the pre-vaccination era, measles practically never happened in babies much younger than one year of age due to the robust maternal immunity transfer mechanism. The vulnerability of very young babies to measles today is the direct outcome of the prolonged mass vaccination campaign of the past, during which their mothers, themselves vaccinated in their childhood, were not able to experience measles naturally at a safe school age and establish the lifelong immunity that would also be transferred to their babies and protect them from measles for the first year of life.\nLuckily, a therapeutic backup exists to mimic now-eroded maternal immunity. Infants as well as other vulnerable or immunocompromised individuals, are eligible to receive immunoglobulin, a potentially life-saving measure that supplies antibodies directed against the virus to prevent or ameliorate disease upon exposure (see appendix, Item #8).\nIn summary: 1) due to the properties of modern vaccines, non-vaccinated individuals pose no greater risk of transmission of polio, diphtheria, pertussis, and numerous non-type b H. influenzae strains than vaccinated individuals do, non-vaccinated individuals pose virtually no danger of transmission of hepatitis B in a school setting, and tetanus is not transmissible at all; 2) there is a significantly elevated risk of emergency room visits after childhood vaccination appointments attesting that vaccination is  not risk-free; 3) outbreaks of measles cannot be entirely prevented even if we had nearly perfect vaccination compliance; and 4) an effective method of preventing measles and other viral diseases in vaccine-ineligible infants and the immunocompromised, immunoglobulin, is available for those who may be exposed to these diseases.\nTaken together, these four facts make it clear that discrimination in a public school setting against children who are not vaccinated for reasons of conscience is completely unwarranted as the vaccine status of conscientious objectors poses no undue public health risk.\nDr. Obukhanych offers online classes for those who want to gain deeper understanding of how the immune system works and whether the immunologic benefits of vaccines are worth the risks: Natural Immunity Fundamentals.\nItem #1. The Cuba IPV Study collaborative group. (2007) Randomized controlled trial of inactivated poliovirus vaccine in Cuba . N Engl J Med 356:1536-44\nhttp://www.ncbi.nlm.nih.gov/pubmed/17429085\nThe table below from the Cuban IPV study documents that 91% of children receiving no IPV (control group B) were colonized with live attenuated poliovirus upon deliberate experimental inoculation.  Children who were vaccinated with IPV (groups A and C) were similarly colonized at the rate of 94-97%.  High counts of live virus were recovered from the stool of children in all groups.  These results make it clear that IPV cannot be relied upon for the control of polioviruses.\nItem #2. Warfel et al. (2014) Acellular pertussis vaccines protect against disease but fail to prevent infection and transmission in a nonhuman primate model. Proc Natl Acad Sci USA 111:787-92\nhttp://www.ncbi.nlm.nih.gov/pubmed/24277828\n“Baboons vaccinated with aP were protected from severe pertussis-associated symptoms but not from colonization, did not clear the infection faster than naïve [unvaccinated] animals, and readily transmitted B. pertussis to unvaccinated contacts. By comparison, previously infected [naturally-immune] animals were not colonized upon secondary infection.”\nItem #3. Meeting of the Board of Scientific Counselors, Office of Infectious Diseases, Centers for Disease Control and Prevention, Tom Harkins Global Communication Center, Atlanta, Georgia, December 11-12, 2013\nhttp://www.cdc.gov/maso/facm/pdfs/BSCOID/2013121112_BSCOID_Minutes.pdf\n“Findings indicated that 85% of the isolates [from six Enhanced Pertussis Surveillance Sites and from epidemics in Washington and Vermont in 2012] were PRN-deficient and vaccinated patients had significantly higher odds than unvaccinated patients of being infected with PRN-deficient strains.  Moreover, when patients with up-to-date DTaP vaccinations were compared to unvaccinated patients, the odds of being infected with PRN-deficient strains increased, suggesting that PRN-bacteria may have a selective advantage in infecting DTaP-vaccinated persons.”\nItem #4. Rubach et al. (2011) Increasing incidence of invasive Haemophilus influenzae disease in adults, Utah, USA. Emerg Infect Dis 17:1645-50\nhttp://www.ncbi.nlm.nih.gov/pubmed/21888789\nThe chart below from Rubach et al . shows the number of invasive cases of H. influenzae (all types) in Utah in the decade of childhood vaccination for Hib.\nItem #5. Wilson et al. (2011) Adverse events following 12 and 18 month vaccinations: a population-based, self-controlled case series analysis. PLoS One 6:e27897\nhttp://www.ncbi.nlm.nih.gov/pubmed/22174753\n“Four to 12 days post 12 month vaccination, children had a 1.33 (1.29-1.38) increased relative incidence of the combined endpoint compared to the control period, or at least one event during the risk interval for every 168 children vaccinated.  Ten to 12 days post 18 month vaccination, the relative incidence was 1.25 (95%, 1.17-1.33) which represented at least one excess event for every 730 children vaccinated.  The primary reason for increased events was statistically significant elevations in emergency room visits following all vaccinations.”\nItem #6. De Serres et al . (2013) Largest measles epidemic in North America in a decade–Quebec, Canada, 2011: contribution of susceptibility, serendipity, and superspreading events. J Infect Dis 207:990-98\nhttp://www.ncbi.nlm.nih.gov/pubmed/23264672\n“The largest measles epidemic in North America in the last decade occurred in 2011 in Quebec, Canada.”\n“A super-spreading event triggered by 1 importation resulted in sustained transmission and 678 cases.”\n“The index case patient was a 30-39-year old adult, after returning to Canada from the Caribbean.  The index case patient received measles vaccine in childhood.”\n“Provincial [Quebec] vaccine coverage surveys conducted in 2006, 2008, and 2010 consistently showed that by 24 months of age, approximately 96% of children had received 1 dose and approximately 85% had received 2 doses of measles vaccine, increasing to 97% and 90%, respectively, by 28 months of age.  With additional first and second doses administered between 28 and 59 months of age, population measles vaccine coverage is even higher by school entry.”\n“Among adolescents, 22% [of measles cases] had received 2 vaccine doses.  Outbreak investigation showed this proportion to have been an underestimate; active case finding identified 130% more cases among 2-dose recipients.”\nItem #7. Wang et al . (2014) Difficulties in eliminating measles and controlling rubella and mumps: a cross-sectional study of a first measles and rubella vaccination and a second measles, mumps, and rubella vaccination. PLoS One 9:e89361\nhttp://www.ncbi.nlm.nih.gov/pubmed/24586717\n“The reported coverage of the measles-mumps-rubella (MMR) vaccine is greater than 99.0% in Zhejiang province.  However, the incidence of measles, mumps, and rubella remains high.”\nItem #8. Immunoglobulin Handbook, Health Protection Agency\nhttp://webarchive.nationalarchives.gov.uk/20140714084352/http://www.hpa.org.uk/webc/HPAwebFile/HPAweb_C/1242198450982\nHUMAN NORMAL IMMUNOGLOBULIN (HNIG):\nTo prevent or attenuate an attack in immuno-compromised contacts\nTo prevent or attenuate an attack in pregnant women\nTo prevent or attenuate an attack in infants under the age of 9 months\n[1] http://www.fda.gov/NewsEvents/Newsroom/PressAnnouncements/ucm376937.htm\n[2] http://archinte.jamanetwork.com/article.aspx?articleid=619215\n[3] Poland (1998) Am J Hum Genet 62:215-220\nhttp://www.ncbi.nlm.nih.gov/pubmed/9463343\n“ ‘poor responders,’ who were re-immunized and developed poor or low-level antibody responses only to lose detectable antibody and develop measles on exposure 2–5 years later.”\n[5] LeBaron et al. (2007) Arch Pediatr Adolesc Med 161:294-301\nhttp://www.ncbi.nlm.nih.gov/pubmed/17339511\n“Titers fell significantly over time [after second MMR] for the study population overall and, by the final collection, 4.7% of children were potentially susceptible.”\n[6] De Serres et al . (2013) J Infect Dis 207:990-998\nhttp://www.ncbi.nlm.nih.gov/pubmed/23264672\n“The index case patient received measles vaccine in childhood.”\n[7] Rosen et al. (2014) Clin Infect Dis 58:1205-1210\nhttp://www.ncbi.nlm.nih.gov/pubmed/24585562\n“The index patient had 2 doses of measles-containing vaccine.”\nShare on X (Opens in new window) X\nShare on Pinterest (Opens in new window) Pinterest', '[-0.018875474110245705, 0.0025907871313393116, 0.04271964356303215, -0.05436002090573311, -0.03852633014321327, -0.002180224983021617, 0.0950741320848465, 0.002328017493709922, -0.034761201590299606, 0.06799411028623581, 0.016776299104094505, 0.014023875817656517, 0.03034598007798195, 0.051121342927217484, -0.060278698801994324, 0.05204995349049568, 0.038974273949861526, -0.04455650970339775, -0.024835538119077682, 0.026949837803840637, -0.02807191200554371, 0.0746598169207573, 0.03370954096317291, 0.055461470037698746, -0.0796990767121315, 0.009109437465667725, -0.031395476311445236, -0.03233713656663895, -0.060673996806144714, 0.0027437377721071243, 0.006665890570729971, -0.04228668659925461, -0.0373675711452961, -0.0021458491683006287, 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INSERT INTO `dr_young_all_articles` (`id`, `title`, `url`, `content`, `embedding`) VALUES
(203, 'Second Thoughts About Viruses, Vaccines, and the HIV/AIDS Hypothesis', 'https://phoreveryoung.wordpress.com/2015/12/28/second-thoughts-about-viruses-vaccines-and-the-hivaids-hypothesis/', 'Second Thoughts About Viruses, Vaccines, and the HIV/AIDS Hypothesis\nIn the sciences, people quickly come to regard as their own personal property that which they have learned and had passed on to them at the universities and academies. If however, someone else now comes along with new ideas that contradict the Credo (that has been recited for years and passed on in turn to others) and in fact even threaten to overturn it, then all passions are raised against this threat and no method is left untried to suppress it. People resist it in every way possible: pretending not to have heard about it; speaking disparagingly of it, as if it were not even worth the effort of looking into the matter. And so a new truth can have a long wait before finally being accepted.\nThe first isolation of a virus was achieved in 1892 by Russian bacteria hunter Dimitri Iwanowski, who gathered fluid from diseased tobacco plants. He passed this liquid through a filter fine enough to retain bacteria; yet to Iwanowski’s surprise, the bacteria-free filtrate easily made healthy plants sick. In 1898 a Dutch botanist, Martinus Willem Beijerinck, repeating the experiment, also recognized that there was an invisible cause and named the infectious agent “tobacco mosaic virus.” In the same year as Beijerinck’s report, two German scientists purified a liquid containing filterable viruses that caused foot-and-mouth disease in cattle (viruses were at one time called “filterable viruses,” but eventually the term “filterable” came to apply only to viruses, and was dropped). Walter Reed followed in 1901 with a filtrate responsible for yellow fever, and soon dozens of other disease-causing viruses were found.\nIn 1935 another American, Wendell M. Stanley, went back to the beginning and created pure crystals of tobacco mosaic virus from a filtered liquid solution. He affirmed that these crystals could easily infect plants, and concluded that a virus was not a living organism, since it could be crystallized like salt and yet remain infectious. Subsequently, bacteriologists all over the world began filtering for viruses, and a new area of biology was born-virology.\nHistorically, medical science has vacillated on the question of whether a virus is alive. Originally it was described as nonliving, but is currently said to be an extremely complex molecule or an extremely simple microorganism, and is usually referred to as a parasite having a cycle of life. (The term “killed” is applied to certain viral vaccines, thus implying an official conviction that viruses live.) Commonly composed of either DNA or RNA cores with protein coverings, and having no inherent reproductive ability, viruses depend upon the host for replication. They must utilize the nucleic acids of living cells they infect to reproduce their proteins (i.e., trick the host into producing them), which are then assembled into new viruses like cars on an assembly line. Theoretically, this is their only means of surviving and infecting new cells or hosts.\n{Key words: Vaccines, Vaccination, Virus, HIV, AIDS, Ebola, Hunta virus, Hepatitis C, Pleomorphism, Yeast, Candida albicans, Koch’s postulate, Farr’s law, Virology, Louis Pasteur, Antione BeChamp, Microzyma]\nBirth of Virology-a Miscarriage?\nIn the early third of the 20th century, the heated debate took place over filterable bacteria versus non-filterable. This was a major battle concerning micromorphology (discussed briefly below). The orthodox view prevailed: bacterial forms were not small enough to pass, or did not have a smaller, earlier stage. What passed through “bacteria-proof filters was something else, i.e., viruses. Standard medical textbooks long made this fdtering distinction between bacteria and viruses. Subsequently, however, the cellular nature of many filterable forms originally thought to be viruses, such as some mycoplasmas, rickettsias, and various other groups, has been established. In this writer’s opinion, with the victory of the monomorphic view, deeper understanding of infectious “disease” was lost, setting the stage for cancer, degenerative symptoms and AIDS.\nA typical bacterium is about 1 micron in size. Most filterable forms now called viruses range in size from .3 microns (300 millimicrons) to .01 micron (10 millimicrons)-partially in the colloidal range (.1 to .001 micron). Most of the larger viruses are a third to a quarter the size of the average bacterium. Size is critical because .3 microns is the resolution limit of modern-day light microscopes (except for the claimed resolution of Canadian microscopist Gaston Naessens’ Somatoscope, at .015 microns). Thus, as viruses were discovered (except for the very large ones, such as mumps), they required an electron microscope to be seen, especially given the fact that Royal Rife’s microscope technology and career were destroyed by vested interests (see Section 1). Unfortunately, electron microscopes and the process of chemical staining disorganize all specimens, whereas Rife’s technology allowed life to proceed and thus evolve under its lens. As viruses became visible to advancing technology, the ramification was that the technology revealed, to minds infected with monomorphism, protein structures deemed foreign to the body.\nFormulated by Bechamp in the 19th century, microzymian principle is the basis of a new theory about “viruses.” Briefly, this principle holds that in all living organisms are biologically indestructible anatomical elements, which he called microzymas. They are independently living organized ferments, capable of producing enzymes and capable of evolution into more complex microforms, such as bacteria. Bechamp’s thesis is that disease is a condition of one’s internal environment (terrain); that disease (and its symptoms) are “born of us and in us”; and that disease is not produced by an attack of microentities but calls forth their endogenous evolution. (The common biological basis for this is discussed below.)\nIt is known that normal cell activity includes genetic repair. Both enzymes and proteins must be involved. What is the mechanism? Viruses are organized around DNA or RNA, not both. Thus, they are quite probably intended to repair genetic molecules or other structures, and show up with disease symptoms because the body needs them. Since viruses require a living cell/host for reproduction, how do we know that the scenario is not set in motion for a purpose by the cell (i.e., its microzymas), rather than being the result of invasion? Because disease (disturbance of balance in the organism) is so prevalent, especially that which has not yet become indicated by common symptoms, repair proteins may be frequently or constantly present. A toxified cell may easily suffer localized damage to the genome. Since most observers are not even aware of microzymian principle, much less understand or even consider it, and since monomorphism stresses invasion, these protein complexes are regarded as foreign and disease is attributed to them.\nAnother note of interest is the size of viruses compared to the microzyma. Viruses are considered to be some of the smallest biological particles and are frequently of colloidal size: e.g., hepatitis A, 27 nanometers (.027 microns); hepatitis B (.042 microns); poliovirus (.03 microns); EBV (.042 microns); fflV (.080 to .12 microns), influenza (.08 to .12 microns); mumps (.15 to .30 microns); smallpox (.30 x .24 microns); and, according to Bechamp, the microzyma (.0005 microns). This coincides with what Gaston Naessens says about the size of his somatid, which ranges from “a few Angstroms to a tenth of a micron.”[1]\nIn his book, T he Blood and Its Third Anatomical Element , Bechamp states: “The microzyma is at the beginning and at the end of all organization. It is the fundamental anatomical element whereby the cellules, the tissues, the organs, the whole of an organism are constituted living. … In a state of health the microzymas act harmoniously and our life is, in every meaning of the word, a regular fermentation. In the condition of disease, the microzymas do not act harmoniously, the fermentation is disturbed, the microzymas have either changed their function or are placed in an abnormal situation by some modification of the medium.”[2]  The virus is either a self-ordered microzymian polymerization, or (less likely) a structure made by microzymas. It is enveloped in protein which is also composed of microzymas, and could well be thought of as an autonomous molecular tool box.\nAlong with Drs. Glen Dettman and Archie Kalokerinos, I wonder, “whether Bechamp’s writing anticipated, in some respects, the discovery of RNA and DNA?” Could the genetic structure be the construct, thus a tool, of the microzyma? They quote a personal communication (1974) from a Professor Bayev of the USSR Academy of Sciences, who discusses his work showing that molecular self-restoration from its parts of pure transfer RNA from brewer’s yeast is possible (see Section 1 of this Appendix for full quotation).[3]\nI suggest a developmental cycle in vivo consisting of three macrostages: (1) a primitive stage comprising the repair protein complexes; (2) an intermediate, or bacterial, stage including filterable forms such as the cell-wall deficient forms described by Lida Mattman, Ph.D. (in Cell Wall Deficient Forms, Stealth Pathogens); and (3) a culmination stage consisting of yeast and fungal phases, and then mold, the end phase. The usual course of development would be from microzyma to repair protein and then to bacterium, etc. However, under certain conditions, such as trauma for example, it is highly likely that the microzymas can skip the primitive stage and become bacteria directly. Although these transformations are as astounding as that of a larva to a butterfly, what is equally impressive under observation is the rapidity with which they can take place-in minutes, even seconds, sometimes. By the same token, when provoked by conditions and the cycle proceeds to yeast, fungus and then mold, it may occur so rapidly that the bacterial stage, if it happens, has no time to be of any significance.\nThus, symptogenic microforms can originate within higher organisms without invasion, via a permutation of the endogenous microzymas when the situation calls for such change. The situation is an imbalance referred to by Bechamp as a “modification of the medium.” Endogenous evolution is evident under the microscope when bacterial, yeast, and fungal forms are seen coming out of red blood cells which initially appear normal.\nBiological Basis for the Pleomorphic Cycle\nThere is a common biological basis for the pleomorphic cycle and its increasing complexity of organization: More complex forms evolve inherently upon the death of an organism for the purpose of recycling its anatomical and chemical structures in the carbon cycle. The process of rapid evolution (which is reversible) is an essential life process which, beyond the repair stage, is necessary to return a dead organism to the earth. The second and third-stage microforms degenerate the body’s vital substances and tissues via putrefaction (bacteria) and fermentation (yeast and fungus). Fermentation results in acid waste products, which further break down tissue. Disease symptoms, then, especially the degenerative type, are not produced by viruses, but manifest as chemical decomposition, or attempted recycling via fermentation and acid toxins, but with “host” survival processes still operable (see Section 3 and discussion of pH in the main text). Obviously, certain other factors may play important roles in producing symptoms, such as heavy metal toxicity, or state of mind, for example. Some of the body’s survival methods also produce symptoms commonly called diseases. An example is eczema, an emergency expulsion of acid toxins via the skin.\nThe aforementioned causal (alarming) situation, or modification of the medium, is chronic acidification (pH imbalance) and oxygen deprivation in the blood and tissues due to acid-forming foods, adverse lifestyle, emotional stress, and environmental stress. This is not oversimplification. Acidification/hypoxia biochemically signals a dead host to the microzymas, while creating collapsed areas (dead zones) of the colloidal system in the intercellular fluid (see Section 2), and it is the primary physiological disease condition out of which the symptoms commonly called specific diseases arise.\nThus, we distinguish between this disease condition and its consequent symptoms, which include both the morbidly evolved microzymas and the physiological signs commonly thought of as specific diseases. As they develop, microforms (bacteria, yeast, fungus and mold) are actually scavenging forms of the microzyma, developed when disease in the cell life requires tissue to be broken up. These upper development forms are the ones easily visible in the blood before physical symptoms arise. They disappear (devolve) when the recycling task is complete, once again becoming microzymas of the earth and/or air.\nRegarding the early period of virus isolation, a question is whether the unseen entities isolated in filtered fluids were accompanied by the waste products (mycotoxins) of fermentation by yeast and fungus of cellular elements, such as DNA. If virus filtrates are injected into a host to prove virulence, it is almost certain that easily filterable molecular toxins will be introduced as well. Could Dr. Stanley’s “pure crystals of tobacco mosaic virus” have been crystallized toxins? If so, they would certainly be highly symptogenic, as are exotoxins at the intermediate stage of the cycle, for example. However, it is not proof of anything that you can create illness by poison injection, except proof of that tautological fact.\nThe term “virus” is the Latin word for poison, and gives us insight into the immediate cause of disease symptoms-poisons: mycotoxins, endotoxins, exotoxins, and toxins from environmental sources (many of which are primary or secondary mycotoxins). Orthodox medicine is well aware that it is bacterial toxins more than the bacteria themselves (they feed in us), that cause the symptoms referred to as infectious disease. Little if any emphasis is placed on this fine but important distinction. Always, the germ is emphasized. There is little to no awareness (or acknowledgment), either, of the same role played by toxins of the culminate microforms of the pleomorphic cycle. Their action and the body’s response to them are frequently ascribed to viruses, which do not produce toxins but are said to wreak havoc by a number of other means. However, if they participate in symptogenesis in a host it is because they are stimulated to evolve into more complex, toxigenic forms. Somewhat less likely is the possibility that they cause damage as a result of erroneous construction or function, for one reason or another-missing mineral nutrients leading to enzyme deficiencies, for example.\nThe Encyclopaedia Britannica has this to say about bacteria, which relates also to viruses:\nThe common idea of bacteria in the minds of most people is that of a hidden and sinister scourge lying in wait for mankind. This popular conception is born of the fact that attention was first focused upon bacteria through the discovery, some 70 years ago, of the relationship of bacteria to disease in man, and that in its infancy the study of bacteriology was a branch of medical science. Relatively few people assign to bacteria the important position in the world of living things that they rightly occupy, for it is only a few of the bacteria known today that have developed in such a way that they can live in the human body, and for every one of this kind, there are scores of others which are perfectly harmless and far from being regarded as the enemies of mankind, must be numbered among his best friends.\nIt is in fact no exaggeration to say that upon the activities of bacteria the very existence of man depends; indeed, without bacteria there could be no other living thing in the world; for every animal and plant owes its existence to the fertility of the soil, and this in turn depends upon the activity of the micro-organisms which inhabit the soil in almost inconceivable numbers. It is one of the main objects of this article to show how true is this statement; there will be found in it only passing reference to the organisms which produce disease in man and animals- for information on these see Pathology and Immunity. {Encyclopaedia Britannica, 14th ed., Vol. 2, p. 899)\nThe general message of the foregoing article applies even more aptly to viruses in the sense that much fear has been bred and cultivated around them, although they never produce disease symptoms, whereas some bacteria do. The writer of the above understands bacteria, with the exceptions that symptogenic bacteria found in man and animals do not produce disease (only secondary symptoms), that their precursors are endogenous to higher organisms, and they have not “developed in such a way that they can live in the human body.” If anything, the reverse is true. According to one theory of microbiology, microforms have colonized over eons to become higher organisms. In one sense, then, the human body has developed as a specialized environment for them.\nAn important dimension of the bacterial dependence of higher life forms is the floral population in the human digestive tract. Literally, these “foreign species” keep us alive.\nMost bacteria have the same underlying function, whether found in soil, sewage, in the human digestive tract, or elsewhere in nature: they are an essential part of the life processes of higher organisms. They will not or cannot attack healthy cells or tissues, but certain ones will recycle sick or dead tissue in much the same way insect pests are drawn to weaker plants. As Bechamp said, “Nothing is the prey of death; all things are the prey of life.”\nFollowing in the wake of misconceptions arising from the fundamental biological error known as the germ theory of disease, defining the filtrates of diseased tissue as a newly discovered infectious microform was the birth of a major corollary error in bioscience.\nListed below are ways viruses are said to disrupt or destroy host cells according to orthodox medical science and the germ theory. Following each in italics is a different interpretation following from microzymian principle:\n1. Viral proteins insert into the host cell’s plasma membrane and directly damage its integrity to promote cell fusion (HIV, measles, and herpes viruses).\nProteins are attempting to repair membrane damage, or enter cells to make other repairs. There is the question as to whether viruses on cell walls are coming or going. In both cases it would be a matter of whether or not a cell has been disturbed by excess fermentation and acidity. But in the former case the cell would be dysfunctional before attachment occurs, thus requiring the repair complex. Another possibility, perhaps remote, is that dysfunctional receptors on cells are in need of repair, or they are covered by these complexes to inactivate malfunctioning cells. Positive electrical charges in a compromised terrain, primarily on acid molecules from fermentations, discharge cell membranes and act as mortar to stick cells together.\n2. Viruses inhibit host cell DNA, RNA, or protein synthesis. For example, poliovirus inactivates cap-binding protein, which is essential for protein synthesis directed by capped host cell mRNAs, while allowing protein synthesis from uncapped poliovirus mRNAs.\nProtein inactivation is probably being done by fermentation or by acidic toxins from fermentation, while “poliovirus” is produced in the cell to reverse the damage.\n3. Viruses replicate efficiently and lyse host cells, e.g., liver cells by yellow fever, and neurons by poliovirus.\nHighly unlikely. The lysing is more likely caused by acid mycotoxicosis, or by free radicals (ROTS) released in response to mycotoxic stress, or from other sources (ionizing radiation, for example). Repair particles are residual after cell wall disruption.\n4. Slow-virus infections (e.g., subacute sclerosing panencephalitis, caused by the measles virus) culminate in severe progressive diseases after a long latency period.\nHow is this demonstrated? Perhaps “latency” is a period of successful or attempted repair that eventually falters. Symptomology naturally appears in the weakest parts of the body. Excess acidity is always a systemic problem that localizes, just as cancer is a systemic condition that localizes, even though its symptogenic influence may later spread.\n5. Viral antigen proteins on the surface of the host cells are recognized by the immune system, and the host lymphocytes attack the virus-infected cells (e.g., liver cells infected with hepatitis B).\nLiver cells are damaged beyond repair by mycotoxicosis, and the immune system, our elaborate janitorial service, is cleaning up the garbage. Perhaps the repair protein antigen is expressed to signal immune response (because the cell is beyond repair), which is one explanation for why there are antibodies to these proteins.\n6. Viruses damage cells involved in host antimicrobial defense, leading to secondary infections.\nThe function of immune cells is damaged by fungal infestation and/or overwork by toxic overload, preventing proper cleanup and elimination of disharmonious, symptogenic elements.\n7. Viral killing of one cell type causes the death of other cells that depend on them, e.g., degeneration of muscle cells enervated by the attack of poliovirus on motor neurons.\nOnce again, a misinterpretation and lack of understanding that it is not viral microforms that damage neurons. Toxins from bacteria, yeast, fungus and mold-as well as the fermentation of glucose, proteins, hormones and fats-produce, or influence the body to produce, disease symptoms. Not recognizing the “virus,” for what it is, observers attribute disease to it.\n8. Host cell responses to viruses include metabolic derangements and transformations resulting in neoplastic changes.\nMetabolic derangement has occurred prior to the appearance of repair proteins, due to toxic overload in the cell. It is more likely that the proteins attempt to prevent cell transformation, and that cancerous development is cell conversion from primarily oxidative to wholly fermentative metabolism, mediated by fungus and mold.\nListed below are further orthodox views regarding virus replication, etc., with alternative interpretations in italics.\n9. According to orthodox theory, viruses enter a host cell and replicate at the host’s expense. Replication is accomplished using enzymes which are distinct for each virus family. For example, RNA polymerase is used by negative-stranded RNA viruses to generate positive-stranded mRNA, whereas reverse transcriptase is used byretroviruses to generate DNA from their RNA template and to integrate that DNA into the host genome.\nIt is normal for repair proteins to generate enzymes to do their work.\n10. One reason suggested for viral tropism (the tendency to infect some cells but not others) is the presence or absence of host cell receptors that allow the virus to attach. It is said, for example, that HIV binds to the protein (CD4) involved with antigen presentation on helper T-lymphocytes, that Epstein-Barr virus binds to the complement receptor (CD2) on macrophages, that rabies virus binds to the acetylcholine receptor on neurons, and that rhinoviruses bind to the adhesion protein (ICAM-1) on mucosal cells.\nTheoretically, once attached, the entire virion, or a portion containing the genome and essential polymerases, penetrates into the cell cytoplasm in one of three ways: (1) Translocation of the entire virus across the plasma membrane; (2) receptor-mediated endocytosis of the virus and fusion with endosomal membranes; or( 3) fusion of the viral envelope with the cell membrane. Theory suggests that within the cell the virus uncoats, separating its genome from its structural components and losing its infectivity before replication. In either the nucleus or cytoplasm, newly synthesized viral genomes and capsid proteins are assembled into progeny virions, which may then bud through the plasma membrane. Unencapsulated viruses may be released also, directly through the membrane.\nIt is interesting, however, that viruses can somehow choose the “infection” to be abortive, latent or persistent, meaning respectively: (1) viral infections with incomplete replication cycles; (2) persisting in a cryptic state, like herpes zoster within a dorsal root ganglion, which suddenly becomes active to produce shingles;( 3) continuously synthesized virions, with or without altered cell function (e.g., hepatitis B). These three ideas, especially latency, have arisen as feeble excuses for the untenable virus theory.\n11. In order for viruses to reproduce, they must complete the following four steps:\na). Adsorption and penetration of a cell. The viral particle binds to the host cell membrane. This is usually a specific interaction in which a viral encoded protein on the capsid or a glycoprotein embedded in the virion envelope binds to a host cell membrane receptor and is then internalized. This internalization occurs by endocytosis or by fusion of the virion envelope with the host cell membrane.\nThis is the mechanism whereby the viral particle enters the cell for the purposes of carrying out repairs to the damaged DNA orRNA.\nb). Uncoating of the virus, so that the nucleic acid can be released from the capsid into the nucleus or cytoplasm.\nRepair work may require uncoating. An uncoated “virus” in the cytoplasm may have come from the nucleus and not yet have a coat, as in the case of hepatitis B according to medscience. A coat is then created to protect the nucleic acid, to make a communicative or responsive protein complex, or to allow exiting the cell for remote function or for neutralization and recycling by the immune system.\nc) Synthesis and assembly of viral products as well as inhibition of the host cell’s own DNA, RNA and protein synthesis.\nProtein complexes produced in response to an alarming situation-fermentative and mycotoxic stress-are capable of self-ordered replication. As suggested by Bechamp, the microzyma is specific for each organ, therefore specific repair proteins will be needed for specific cells that make up specific organs that are being disturbed. There is the question of why the great numbers in some cases. One possibility is simply overreaction; for example, fever can be extreme.\nd) And finally, release of virions from the host cell either by budding or lysis.\n(1) Complexes leave the cell for remote function or to be neutralized; (2) Repairs have failed, and complexes are released prior to or during the breakdown of the cell by acid toxins or the immune system.\nKalokerinos and Dettman quote Dr. Fred Klenner regarding the changeability of viruses: “I am of the opinion that virus units have the potential of going from one type to another by altering their protein coat. We see chicken pox at Thanksgiving, mumps at Christmas, red measles in the spring, and polio and Coxsackie in the summer.”[6]  Seasonal appearance of different forms may be mediated by variations of imbalance in the biological terrain or nutritive medium due to the fermentation of dietary excesses such as sugar and animal proteins that accompany holidays and seasons, calling for different repair proteins.\nFor example, outbreaks of polio have been associated with sugar consumption in summer. Various psycho-emotional stresses correspond to these seasons as well.\nSupporting the general idea of dietary culpability is a statement published by the great English physician, Sir Robert McCarrison in 1936: “Obsessed with the invisible microbe, virus, protozoa as all important excitants of disease, subservient to laboratory methods of diagnosis, hidebound by our system of nomenclature, we often forget the most fundamental of all rules for the physician, that the right kind of food (nutrition) is the most important single factor in the promotion of health and the wrong kind of food the most important single factor in the promotion of disease.”7\nDiseases are not individuals arranged in classes, like cats and dogs, but conditions growing out of one another.\nIs it not living in a continual mistake to look upon diseases, as we do now, as separate entities, which must exist, like cats and dogs, instead of looking upon them as conditions, like a dirty and a clean condition, and just as much under our own control; or rather, as the reactions of kindly Nature against the conditions in which we have placed ourselves?\nI was brought up … distinctly to believe that smallpox, for instance, was a thing of which there was once a first specimen in the world, which went on propagating itself in a perpetual chain of descent, just as much as that there was a first dog, (or a first pair of dogs), and that smallpox would not begin itself any more than a new dog would begin without there having been a parent dog.\nSince then I have seen with my eyes and smelt with my nose smallpox growing up in first specimens, either in close rooms or in overcrowded wards, where it could not by any possibility have been “caught,” but must have begun. Nay, more, I have seen diseases begin, grow up, and pass into one another. … I have seen, for instance, with a little overcrowding, continued fever grow up; and with a little more, typhoid fever; and with a little more, typhus, and all in the same ward or hut.\nWould it not be far better, truer, and more practical, if we looked upon disease in this light? For diseases, as all experience shows, are adjectives, not noun-substantives.\nThat is, symptoms (called diseases) are describers of a situation.\nI find legitimate Bechamp’s conclusion that what are called germs of the air are fundamentally microzymas of beings which are being consumed by the recycling process, i.e., some kind of vegetative digestion-putrefaction or fermentation. In short, there are no pre-existing disease-germ species. The principles of microbian medicine constitute a fundamental biological error. As Bechamp said, “The microbian doctrine is the greatest scientific silliness of this age.” This is not to say that there is no transmission, only that invasion is not necessary for symptogenesis, nor is it the primary mechanism for illness. It is to say that for transmission to take place, susceptibility in the form of a compromised terrain must pre-exist in the receiver, who is then likely to be ill anyway. With the exception of the immune component in the mucosal barrier, primary host “resistance” is a function of terrain condition rather than immunity per se.\nHepatitis can be a painful symptom that has yielded profitable virus-hunting opportunities in recent years. Although there are several categories of this disorder, three main varieties of what is called “acute viral hepatitis” exist: Type A (formerly “infectious hepatitis”), Type B (formerly “serum hepatitis”), and hepatitis C (formerly “non-A, non-B”). The corresponding viruses are HAV, HBV, and the non-A, non-B “group,” now called C. Type A is said to be caused by an RNA virus, spread primarily by fecal contamination of water and food, with blood and secretions also possibly being infectious (but it is due to the toxins associated with unsanitary conditions). Hepatitis B, discovered in the ’60s, is said to be caused by a DNA virus which replicates in the hepatocyte nucleus and receives its surface coat in the cytoplasm. It is said to be transmitted by transfused blood or blood products, or via common use of needles by intravenous drug users (but it is due primarily to over-acidification from the drugs, especially heroin. The exchange of body fluids into the blood, whether by unsterilized needles, abusive sexual activity, etc., can also play a role over time because of repeated immune stress caused by foreign proteins. See Section 1 for Bechamp’s view of the invasion of blood by injection of proteins). Third World babies with poor nutrition and unsanitary conditions around the time of birth are also susceptible.\nThe third type of hepatitis, discovered in the ’70s, is found among drug users and alcoholics, and accounts for 80 to 90% of hepatitis caused by blood transfusion. It is thus akin to B type and was at first thought by scientists to be hepatitis B until thorough testing of subjects revealed no virus B-nor A, for that matter. It was thus called “non-A, non-B” hepatitis and thought to be at least two viruses and perhaps more.\nIn 1987 scientists believed they found a single virus causing the third type, what is known today as the hepatitis C virus. However, what they identified was an antibody they associated with a virus. Now, just as with HIV, they could test patients for antibodies against an elusive or invisible virus. With this new observation, however, new paradoxes confronted the viral hypothesis. Huge numbers of people testing positive for the phantom C virus never developed any symptoms. Hepatitis is truly the result of over-acidification or toxification of the largest filter in the human body by such substances as lactic acid, acetic aldehyde and ethanol-not the disease of a pathological virus. It is interesting to note also that all these hepatitis viruses have incubation periods of 2 to 25 weeks, violating Farr’s Law (see below), yet are not classified as slow viruses. Also, the point at which a “natural invasion” takes place, as opposed to a highly artificial injective one, and thus, how true incubation periods are determined, is another interesting question.\nA recent example of unwarranted panic in American biomedicine was the eminent hantavirus of 1994. Presumably it had jumped species, from mouse to man (the American Navaho Indians). However, after supposedly killing a number of people, this phantom virus apparently made peace with the Indians and retired to its mouse reservoir. The virus failed to materialize.[8] A front-page article in the San Francisco Chronicle reported that CDC “epidemiologists across the nation are carefully monitoring the deer mouse population and the level of virus within it.” But all that was left to discover of the former “Navaho flu” by the CDC epidemiologists (shown in their space suits) were healthy mice in the mountains.[9]  The Navaho flu is nothing new to the native Americans and is most likely tied to sanitation, nutrition and lifestyle.\nIn May 1995 the CDC announced the new, threatening Ebola virus. The deadly killer virus was expected to leave its hidden reservoir in the rain forests of Africa to claim Europe and the United States. An article in Time magazine was peppered with men in space suits and colored electron micrographs of the virus (even though electron microscopes cannot take color pictures). A CDC virologist suggested the virus could leave the rain forest if “we get a virus that is both deadly to man and transmitted in the air.” We are thus asked to fear the image of viruses somehow being launched into the air, perhaps by ejection from a host, and then floating on killer breezes to other lands. A more imaginable scenario was suggested by a European epidemiologist who heads the United Nations AIDS program. Echoing the CDC’s alarm, he stated, “It’s theoretically feasible that an infected person from Kuwait could go to Kinshasa, get on a plane to New York, fall ill, and present transmission risk there.” But within a month the virus had disappeared in Africa, and not a single Ebola case was reported in the United States or Europe. [10]\nThe World Health Organization announced on December 19, 1995 that the Ebola virus epidemic that killed 245 people in West Africa was over. All tests on any remaining suspected cases were negative. A somewhat unsettling revelation was that every Ebola outbreak in Africa “is associated to have spread through public hospitals.”[11]  As it turned out, it was associated with re-used hypodermic needles in these hospitals. Just like hantavirus, Ebola vanished, never to be heard from again. Most interesting is that this epidemic, as epidemics will, stopped without vaccines or other drugs. But consider the impact such stories have made upon our minds and on the way we view and understand germs. What’s next in virodrama, the Andromeda Strain?\nThere is one insidious possibility that must be mentioned in passing. Some mysterious outbreaks of the past have been shown years later to have been man-made. In some cases, government agency has used the public to test releases of organisms and weak biochemical toxins in order to verify, through medical reports, expectations of biowarfare activity. These incidents and the whole story of such behavior is well documented in the book, A Higher Form ofKillingby Robert Harris and Jeremy Paxman (Hill & Wang, 1982). In this scenario, the cause of such an incident would be constructed officially, or left as a mystery, in order to draw attention away from the truth.\nThe greatest danger of the germ theory half-truth is its promulgation and acceptance as the whole truth, thus diverting attention from endogenous factors, primarily host ecology-resistance and susceptibility. Such factors are highly significant if Bechamp and his many followers, including me, are correct. Distraction from host factors has been quite thorough, with the exception of the false notion that the immune system is the “first line of defense” against infectious symptoms.\nLouis Pasteur is credited with improving and successfully using the technique of vaccination, a practice blindly begun in 1796 by British physician Edward Jenner. Jenner happened to notice that dairy maids who had contracted the relatively mild disease cowpox did not later contract smallpox. On a hunch, he took pus from the running sores of sick cows and injected it into the blood of an eight-year-old boy. As the story goes, the boy developed cowpox. Several weeks later Jenner inoculated the boy with smallpox, but the disease failed to develop. Upon this single anecdotal event was based the supposition that this practice was safe and effective. The process has changed little to this day except perhaps to have been worsened with additives. Its understanding is still clouded by Pasteur’s theory, and it is as recklessly pursued as it was begun.\nTheoretically, vaccination works by introducing a diluted and weakened (attenuated) or “killed” version of the pathogen into the body, causing the immune system’s memory function to prepare for any subsequent contact, which is met with much greater response. It is commonly thought that infectiosity, or germ-virulence, tests are performed on laboratory animals and then vaccines are made which boost the immune system against germs. However, like Jenner’s, the tests are primarily toxicity tests, and vaccines, especially viral ones, activate the immune system primarily in response to injected toxins. Whether the response is to toxins, microforms, or both, it is a misguided approach at best. Bypassing the mucosal barrier and thus the segment of the immune system which is the organism’s interface with the environment, makes such experimentation, and vaccination itself, flawed, unscientific practice ipso facto.\nBacteria secrete a variety of enzymes (leukocidins, hemolysins, coagulases, hyaluronidases, fibrinolysins), any of which are disruptive in the body. For example, diphtheria toxin is composed of the enzymatic fragment A, which is at the amino end of the molecule, and fragment B at the carboxyl end, which allows entry into host cells. The two fragments are linked by a disulfide bond. Once bound diphtheria accesses the cell cytoplasm, the disulfide bond is broken, releasing fragment A. This enzyme catalyzes the covalent transfer of adenosine diphosphate ribose (ADPR) from nicotinamide adenine dinucleotide (NAD) to EF-2. The latter, a ribosomal elongation factor involved in protein synthesis, is thus inactivated. One molecule of diphtheria toxin can kill a cell by ADP-ribosylating more than a million EF-2 molecules. In diluted form this toxin, along with other toxic chemicals and fragments of bacteria, is what is introduced directly into the blood of infants under the guise of a health measure.\nDiphtheria toxin creates a layer of dead cells in the throat, on which Corynebacterium diphtheriae outgrows competing bacteria (the diphtheria microform is an intermediate stage of a morbidly evolved microzyma, and competing bacteria also evolve out of sick cells). Subsequent wide dissemination of diphtheria toxin causes the characteristic neural and myocardial dysfunctions. Diphtheria toxin also causes disseminated intravascular coagulation, which activates the various alarm responses of the body. Thus, we know that toxins produce symptoms, but what is it that produces the condition which creates or supports the toxin producer?\n-Charlotte Parker Department of Microbiology, U. of Texas at Austin\nTo make a vaccine you need to acquire the disease germ-a toxic bacterium or a live virus. The mumps virus is a sterile, lyophilized preparation of the Jeryl Lynn (B level) strain of mumps virus. It is adapted to, and propagated in, cell cultures of chick embryo, free and stabilized with sorbitol and hydrolyzed gelatin. The rubella virus (Wistar RA 27/3 strain) is grown in human diploid cell cultures. Measles (from Eners’ attenuated Edmonston strain) is grown in cell cultures of chick embryo.[12]  The various so-called virus strains are stored by pharmaceutical companies for later culture. Where these stockpiles come from and the specific methods used seem to be guarded secrets, but as Bechamp emphasized, they must originally be obtained from diseased higher organisms, for they are found nowhere else in nature. If protein complexes exist in the viral stores, their replication in culture is simply the behavior pattern of the repair proteins they are. It is highly likely that toxins accompany these strains as a means of stressing the culture cells.\nTo make a live vaccine, the microform must be attenuated, or weakened. This is accomplished by serial passage-passing the microform/toxin many times through animal tissues, e.g., monkey kidneys, human diploid cells (the dissected organs of an aborted fetus), chick embryos and calfs.[13]  Killed vaccines are prepared with heat or radiation, or else chemically, usually by using the mycotoxin formaldehyde.[14]\nThe weakened microform must be mixed with antibody-boosting and immune-activating adjuncts such as the antibiotics neomycin and streptomycin, as well as stabilizers such as sodium chloride, sodium hydroxide, aluminum hydroxide, aluminum hydrochloride, sorbitol, hydrolyzed gelatin, formaldehyde, and thimerosal (a mercury-based antiseptic).\nFor example, diphtheria, pertussis and tetanus (DPT) vaccine consists of a combination of tetanus and diphtheria exotoxins with pertussis microforms. Diphtheria toxin is produced by growing Corynebacterium diphtheriae in a medium composed of pig pancreatic hydrolysate of casein. Tetanus toxin is produced by growing Clostridium tetani in a medium composed of pig tryptic digest of casein. Both toxins are combined with formaldehyde, ammonium sulfate (a mycotoxin), and diluted with saline containing thimerosal. They are then adsorbed on aluminum phosphate and combined with a suspension of Bordetella pertussis organisms.[15]\nThe first pertussis (whooping cough) vaccine was created in 1912 by two French bacteriologists, Jules Bordet and Octave Gengou, who wanted to use it on the children of Tunisia. After growing the pertussis bacteria in large pots, they killed them with heat, preserved the mixture with formaldehyde, and injected it into the children.\nThe vaccine is made in essentially the same way today as in the time of Bordet and Gengou, although each manufacturer prepares it differently, and the exact processes and formulas are considered trade secrets. Pertussis bacteria are usually grown on a casein hydrolysate medium with yeast dialysate, supplemented with agar and charcoal. The mixture is prepared in vats, then washed, and the bacteria killed with heat and formaldehyde The resulting toxoid is preserved with thimerosal. Other possible ingredients are hydrochloric acid, the adjuvant (usually an aluminum compound), sodium hydroxide, and salt.\nIn the past, human blood was often added. This is now prohibited by federal law, but manufacturers are still permitted to add blood from “lower animals other than the horse.” The microzymas of horse blood destroy human blood.\nThe vaccine is stored for a while at near-freezing temperatures, then combined with the diphtheria and tetanus exotoxins and poured into vials for distribution. Ultimately it is shipped to pharmacies, private physicians, and public health clinics, whence it is injected into the blood of infants.\nThe precedent of cruelty to animals, promoted, if not set, by Louis Pasteur, is apparently a hallmark of germ theory. It is not better demonstrated than by the following description of the preparation of so-called calf serum dreamt up in the early days of vaccine manufacture, and continuing, as far as I can tell, into the late 1980s, if not to this day:\nA calf is strapped down to an operating table. A space on the abdomen of about 12-15 inches is shaved with a razor, then about 100 slashes are cut into the flesh. The seed virus, consisting of a culture of smallpox passed through a solution of glycerine, is rubbed into the wounds.\nMade to stand in a headstock so it cannot lick its belly, the calf grows very sick and the wounds become swollen and inflamed. In a few days, as the body reacts to the poison, small blisters appear, scabs form over the wounds and fill with pus. In five to seven days, the wounds are ulcerated, issuing pus and morbid cells. The calf is again strapped to the operating table, and the infested area is washed with warm water. Each scab is scraped off and its contents are pressed out of the sores into a container. An equal amount of glycerine is added to the pus, and the whole is stirred. Once thoroughly mixed, the concoction is passed through a sieve to remove solids such as pieces of flesh, scabs and hair. After being stirred once again, the mixture is put into vials, sealed, and distributed as “pure calf lymph,” commonly known as smallpox vaccine.[16]\nThese aforementioned concoctions are obviously poisonous products of disease. By injecting these products into the blood of school children, physicians, via legal manipulation of health boards and school boards, potentiate illness and ensure that medical products and services will continue to be in high demand.\nIt is interesting to note that the vaccine given to those considered to be at high risk for hepatitis A (such as highly overactive homosexual males, users of illicit injectable drugs, residents of a community experiencing hepatitis A, hemophiliacs and other recipients of therapeutic blood products), or those testing positive for hepatitis A, is made of immune serum globulin obtained by ethanol fractionation of plasma pooled from hundreds of donors. Considering that microzymas and morbidly evolved microzymas are being transferred from one individual to another, one might conclude that this could have disastrous consequences. (The fact that animal blood and fluids are transferred to humans by vaccination bears no further comment, except to say that Frankenstein would be proud.)\nIt is also very interesting that the vaccine given to those testing positive for hepatitis B is created by cloning the antigen HBsAg in a bed of yeast {Saccharomyces cerevi’s Jae, the culminate stage of the morbidly evolved microzyma) and formulated as a suspension of the antigen adsorbed on aluminum hydroxide. [17] Such morbid, poisonous vaccines are given to infants at 2, 4, and 15 months of age. The vaccine is enough to disturb the central balance of the biological terrain and cause an array of symptomologies in anyone, especially an infant. That more people are not quickly poisoned to death by this practice is testimony to the astounding resilience of human physiology.\nDoes the vaccinal approach produce wellness or any health benefit? Kalokerinos and Dettman point out that statistics in England and Wales, presented at the Presidential Address of the British Association for the Advancement of Sciences (Porter, 1971), show that deaths of children under 15 years of age attributed to scarlet fever, diphtheria, whooping cough and measles saw a 90% decline from 1850 to 1940. Yet, antibiotics and compulsory (i.e., widespread) vaccination against diphtheria were not introduced until 1940. The death rate due to these illnesses dropped from over 6,000 per million children in 1850 to under 1,000 per million children in 1940, a period marked by vastly improved public health, sanitation and nutrition.[18]\nOnce I looked into this subject and its history, microzymian principle brought it into focus for me. Since germs evolve out of, or take advantage of, the susceptible state, and are symptoms themselves, drugging or vaccinating susceptible individuals cannot render them immune, and may have the reverse effect. When and if a vaccine works as intended, the result is only to suppress the appearance of a specific set of symptoms, not to prevent disease. Therefore, it is not conferring wellness, nor reducing susceptibility, but simply creating an effect in a highly artificial and dangerous manner, while allowing the disease condition to worsen. Is there a price to pay for this invasive and unscientific approach? In this writer’s view, it is what we’ve got-pandemic degenerative disease, cancer and AIDS, because we are not dealing with the foundational disease, which may then get worse and re-expresses itself in more intense ways.\nThe November/December 1995 issue of The Vaccine Reaction, Volume 1, No. 5, issued by the National Vaccine Information Center, reveals that Swiss scientists have reported finding the enzyme reverse transcriptase (RT) in the live measles/mumps/rubella (MMR) vaccine. This has been traced to the chicken embryos whose cells are used to create the vaccines. It has reportedly been detected in yellow fever and some influenza vaccines, also prepared in chicken embryo cells. No disease has been attributed to RT in the MMR vaccine, but it is a factor in retroviral disease theory and its presence in this case is a mystery. RT, which is officially said to be produced by many “tumor-producing” viruses, supposedly the retroviruses, catalyzes the transformation of RNA into DNA. However, there is no proof of viral production of tumors-only theory.\nI suggest the following process to explain how it gets into the vaccine, based on microzymian principle: Disruption of the embryo cells, by toxins or other means, probably damages their DNA. The response is endogenous microzymian production of repair protein complexes (“retroviruses”), which in turn produce RT in order to effect repairs. As the toxification process continues, central balance in the embryo cells is disturbed sufficiently and the ensuing endogenous pleomorphic development of upper development forms results in excess fermentations, with corresponding increase in the level of toxins. In order not to “spoil the broth,” however, preservatives are added at a certain point to arrest development.\nExperiments with fertile eggs, which I later discovered were described by Bechamp, provide evidence of endogenous microzymian development. I have observed that the hypodermically extracted serum of a fresh egg looks normal under a high powered light microscope. However, when the central balance is disturbed by shaking the egg, which is then allowed to sit for a period of time, extracted serum shows the presence of bacteria, yeasts, and their associated toxins, i.e., acetic, sulfuric and butyric acids. An equally elegant, but even simpler, demonstration is bruising an apple without breaking the skin. Soon the area begins to turn brown and rot from the inside. This is a life process mediated by endogenously developed microforms.\nAnother example of unwanted or unpredictable vaccine contaminants: polio vaccines grown on monkey kidney have been identified as a source of simian viral (SV40) and spherical retroviral structures.[20]  Such stray protein structures and fragments in vaccines can be regarded as a large, uncontrolled, cross-species genetic experiment in which a gene from one species might be spliced as a repair protein into another.\nI can find no accurate statistical estimate for how many deaths and serious injuries are caused by vaccinations each year in the United States. It appears as though the government would rather not release such information, although a federal fund has been set up to cover the millions of dollars in lawsuits that are always pending. Thus, the law has been constructed so that perpetrators of this damaging practice cannot be sued, but continue to profit, while the government shields them with the people’s money.\nPerhaps the government feels that with no way to enforce accurate reporting from doctors, it is futile to indulge in a guessing game. From the doctors’ perspective, there is little to gain from reporting, except an inexorable and embarrassing statistical slide toward collision with the truth. Consider these words from Kalokerinos and Dettman 20 years ago: “Moreover, it is disappointing to observe the futility and ineffectiveness of many ‘flu’ vaccines that have been accepted by an unwary public.”[21] In this writer’s opinion, the statement applies to all vaccines.\nMontague R. Leverson, M.D., Ph.D., M.A., an American physician, happening to come across some of Professor Bechamp’s writings in New York, became fascinated with his views. Realizing that the dated works anticipated Pasteurian “revelations” in certain important points, he decided to go to France to meet Professor Bechamp, where he heard the story of Pasteur’s plagiarism of the professor’s work directly. In a lecture entitled “Pasteur, the Plagiarist,” delivered at Claridge’s Hotel, London, on May 25, 1911, he outlined briefly Bechamp’s claim to be the first to produce a ferment in a medium containing no albuminoid matter, something thought impossible up to that time. (Ethel Douglas Hume’s book about Bechamp was based on work begun by Leverson, who is also the translator of Bechamp’s masterwork, The Blood.)\nUnderstanding microzymian principle, he had this to say about inoculation:\nWhen a drug is administered by the mouth, as was beautifully pointed out by Dr. J. Garth Wilkinson, in proceeding along the alimentary canal it encounters along its whole line a series of chemical laboratories, wherein it is analyzed, synthesized, and deleterious matter is prepared for excretion, and finally excreted, or it may be ejected from the stomach, or overcome by an antidote.\nBut when nature’s coat of mail, the skin, is violated, and the drug inserted beneath the skin, nature’s line of defense is taken in the rear, and rarely can anything be done to hinder or prevent the action of the drug, no matter how injurious, even fatal it may be. All the physicians of the world are incompetent either to foresee its action or to hinder it. Even pure water has been known to act as a violent. . . poison when injected into the bloodstream. How much more dangerous is it, then, to inject poisons known to be such, whether modified in the fanciful manner at present fashionable among vivisectionists or in any other manner. . . . Inoculation should be regarded as malpractice to be tolerated only in case of extreme danger where the educated physician sees no other chance of saving life.\nNow the forcing of these inoculations upon individuals by law is one of the worst of tyrannies imaginable, and should be resisted, even to the death of the official who is enforcing it….\n. . . The entire fabric of the germ theory of disease rests upon assumptions which not only have not been proved, but which are incapable of proof, and many of them can be proved to be the reverse of truth. The basic one of these unproven assumptions, the credit for which in its present form is wholly due to Pasteur, is the hypothesis that all the so-called infectious and contagious disorders are caused by germs, each disease having its own specific germ, which germs have existed in the air from the beginning of things, and that though the body is closed to these pathogenic germs when in good health, when the vitality is lowered the body becomes susceptible to their inroads.\nDr. Leverson goes on to describe disease as nature’s attempt to eliminate waste, and diseased tissues as being due to improper living. He suggests plenty of fresh air, the best sanitation, scanty clothes, and a scientific study of diet. He saw overeating as the precursor to “an enormous number of diseased conditions.”[22]\nThe new policy is “expected” not to eliminate risk but to cut it in half. In the official oddsmanship game of risk versus benefit, this is tendered as comfort to those yet to be afflicted. It consists of two preliminary killed-virus injections given to infants in the first four months “… to build up their immunity to polio. Then they are given two oral doses of ‘weakened-virus’ vaccine between ages 1 and 6.” One can only hope that these microbists desist from this folly because, in addition to their misplaced belief in germ theory, they do not yet understand that the extent of vaccine risk goes beyond reaction.\nAs Leverson emphasized, people are forced to this abomination by law in many cases, especially schoolchildren. Overcoming this assault on human rights usually requires extreme persistence, courage and a knowledgeable approach. (I don’t recommend his approach, but it is self-defense!) The argument is literally that those at risk for damage must be sacrificed to save millions of others (i.e., “the benefits outweigh the risks”). But there is no science or even logic to this. If one is vaccinated, theoretically one is safe. If one chooses not to be vaccinated, then s/he does not threaten vaccinated people, but only those who have chosen that risk. Yet, the responsibility for the decision has been stolen from families under the guise of government responsibility to protect children from parents.\nTo summarize, if we consider Bechamp’s thesis that bacteria are evolved forms of anatomical elements called microzymas, that there are specific disease conditions rather than specific diseases, and that the microform is not the antecedent of disease, but arises in it; and if we add to this my thesis that the primitive stage of evolution, viruses, are apathological and created as response to structural breakdown, and that yeast, fungus, mold and their symptogenic poisons produce the symptoms attributed to viruses, is it possible that medical science is misdirected, if not malfeasant, in its intense pursuit of vaccinal answers? Was Bechamp on the right track? Are his many followers, including myself, correct as well? Is this why we cannot make a successful vaccine, and have, in fact, made dangerous and deadly ones?\nOn a final note of sanity, Edgar Cayce, the renowned psychic who could diagnose illnesses and treatments while in trance, was asked and answered the following question during a diagnostic session:\nQ. Can immunization against contagious diseases be set up in any other manner than by inoculations?\nA. If an alkalinity is maintained in the system-especially with lettuce, carrots and celery, these in the blood supply will maintain such a condition as to immunize a person. In an alkaline system there is less effect of cold and congestion.[23]\nHIV/AIDS and the Monomorphic Disease Model\nNone have any interest in the possibility HIV doesn’t cause AIDS, because if it doesn’t, their expertise is useless.”[25] Their embarrassment would also be considerable.\nAIDS exists on paper. It is just a new label applied to a defined combination of immune-deficiency symptoms, which are not new, and a group of existing “diseases.” Intense public attention has been focused on the combination using statistical manipulation and fear that is bred in a general lack of understanding about health and disease. The question is whether all the destruction of AIDS can be laid at the feet of a nearly undetectable virus that defies every rule of medical microbiology. For example, HIV is said to cause AIDS after the appearance of antiviral immunity. Furthermore, the establishment has shown irresponsibility in referring to this syndrome as a disease. And the fact that it has been given the handy four-letter word encourages others to do likewise. This reinforces programmed notions, especially the idea of a single evil entity causing the whole thing. To emphasize these important points, AIDS will be here designated as “AIDSyndrome” in many instances.\nA Medical Establishment on the Elastic Band Wagon\nThe HIV/AIDS theory is so elastic it stretches to embrace all reasonable criticism. Typical of this elasticity is the so-called latent period of the virus, which has gone from about one year to twelve, and shows potential of going to twenty. The elasticity is equaled only by the degree of credulousness required to accept HIV dogma. For example, it is said that in spite of the extremely low incidence of HIV in the body, it (mysteriously) tricks the immune system into attacking itself! I use the term HIV/Elastic Theory, or HIV/ET.\nAnother major factor is oppressive socio-economic and political conditions. Such conditions exist in the Third World particularly, but in their own way in sections of the United States. This aspect will not be detailed here, but includes such phenomena as corporate dumping of banned drugs on unregulated Third World markets, pesticide manufacture and use with frightening disregard for safety, squalid living conditions, and rainforest destruction. These, not HIV, are among the primary causes of what is labeled AIDSyndrome in the Third World. Pharmaceutical companies are heavily involved in the pesticide market. The corporate-interest connection with these abominations goes: pharmaceuticals, pesticides, agriculture, petroleum, international banking. Therefore, since the HIV/ET hoax has to cover a lot of financial territory, it must have considerable stretchability .\n1. The first recorded AIDSyndrome case in history, one of five reported by the CDC in June 1981, was a 33-year-old Los Angeles male. He was engaged in a lifestyle which we now consider high risk; but there are reasons for risk other than those defined by AIDSyndrome “viromania” (a word coined by microbiologist Peter Duesberg). For one thing, he admitted using “poppers,” the aphrodisiac amyl nitrite (a poisonous secondary mycotoxin), then popular in homosexual bathhouses and discos. We are familiar with nitrites, used in tiny amounts as a preservative in meat. Sodium nitrite, a relatively weak member of the family, has been regulated for years as a potential carcinogen. It is well known that once in the body it is converted into carcinogenic nitrosamines (via its reaction with mycotoxins-not so well known).\nFew mycotoxins, however, are more toxic than the organic nitrites (poppers), which react violently with almost anything. In water, they form the unstable nitrous acid, which destroys any biological molecule within reach. Nitrites and their breakdown products have long been known to scientists for their ability to mutate DNA, a point recently verified by direct experiment.[26]\nThus, in the first recorded AIDSyndrome patient, a yeast infestation of the lung instigated pneumonia (symptom of over-acidification from fermentation processes), and oral thrush, a thick overgrowth of Candida albicans, choked him to death. He died, not from the ravages of a scapegoat retrovirus, but from an overdose of mycotoxins-nitrites-and the mycotoxins of yeast and fungal infestation-acetyl aldehyde, alcohol, and uric acid.\nIn Kenya, Africa, a 39-year-old woman from Zaire entered the hospital for treatment of her lung condition, which had begun with a relatively innocent cough and an unexpected drop in weight. Soon her coughs began to bring up blood, and tuberculosis was the diagnosis. But the patient had a strong allergic reaction to prescribed drugs, and her condition progressed from bad to worse, producing diarrhea, uncontrollable fever, swollen lymph nodes, and anemic blood disorders (all symptoms of a compromised biological terrain, as described in the main text). The woman was then diagnosed with AIDSyndrome (but not I-AIDS-Iatrogenic-AIDS).\nThe woman’s husband, whom doctors assumed must have transmitted AIDSyndrome to his wife, was suffering entirely different symptoms. He had pneumonia, a Candida infestation in his mouth, and lesions of Kaposi’s sarcoma on his now irregularly pigmented skin. He lost weight to a relentless diarrhea and was constantly fighting off episodes of gonorrhea. Their children had no symptoms.[30]\nWe are asked by national public health officials to believe that the Los Angeles case and the two Zaireans all suffered the same affliction from the same cause. The irony is that in terms of germ theory this is highly questionable, but when considered in the light of microzymian principle, it is highly plausible. With one instance of overlap, each person was affected with radically different symptoms-a Pneumocystis pneumonia (as noted, yeast in the lungs); a tuberculosis (symptom of exotoxin from an intermediate pleomorphic stage); and a Kaposi’s sarcoma, or papular tumors of the skin and mucous membranes (caused by mycotoxins). Before AIDSyndrome, these conditions never would have been connected by clinical doctors. Now they are struggling to believe that the common factor is the presence of nearly undetectable antibodies against HIV, and they could not be at a much worse disadvantage.\nThe World Health Organization’s definition for African AIDSyndrome includes some opportunistic infections, like tuberculosis; also, the African version of wasting called “slim disease,” a composite of weight loss, diarrhea, and fever; plus such conditions as persistent cough, skin problems and swollen lymph nodes. These signs comprise old, indigenous African health problems. But here is another example of HIV/ET. Compromised immunity makes “diseases” worse, so whatever “diseases” are already common become the indicators. All we have to do is plug HIV into the equation and we have AIDS. This makes sense to most people.\nOn the other side of the coin, malaria, for example, the leading killer in the Third World, produces fever and other symptoms frequently misdiagnosed as AIDS.[31]  Tuberculosis, also a common killer and part of the defined African syndrome, presents a challenging situation there, as described by a Nigerian medical professor: “The serologic demonstration of HIV infection in patients with tuberculosis in Africa is very important because it aids the separation of seropositive from the seronegative patients, since such a separation may be impossible in all cases on clinical grounds.”[32]\nAccording to a Ugandan doctor treating AIDS cases, “A patient who has TB and is HIV-positive would appear exactly the same as a patient who has TB and is HIV-negative. Clinically, both patients would present with prolonged fever; both patients would present with loss of weight-massive loss of weight, actually; both patients would present with prolonged cough, and in both cases the cough would equally be productive. Now, therefore, clinically I cannot differentiate the two.”[33]  What can be the difference? Of course, a major one is that the AIDS case may be given expensive poison drugs which are nearly certain to end the patient rather than the illness, while filling pharmaceutical coffers.\nDoctor Konotey-Ahulu has illustrated the confusion created by the HIV/ET: “Immunosuppressive diseases, of course, there always have been in Africa and elsewhere before antiquity was born. … I have clinical photographs from 1965 of a Ghanaian man who looked like some of the AIDS patients I saw in Africa recently. The man, who was like a skeleton, had severe nonbloody diarrhea (more than twenty bowel actions a day); he had what looked like fungus in the mouth [candidiasis], skin changes, periodic fever and cough-all the classical features of African AIDS. . . . The patient (according to relatives) had literally consumed on average one and a half bottles of whisky [a mycotoxin] every single day for the previous eighteen months before admission. We found it difficult to believe the story, but there are photographs today showing a complete reversal in 1966 of physical signs and symptoms, including the diabetes, when hospitalization cut short his alcohol supply and active treatment was administered, with gradual protein calorie buildup and pancreatin supplements.”[34]\nOngoing HIV testing since 1985 has revealed that eight times more Africans than Americans are infected (6 to 8 million)[35], yet the continent has produced fewer AIDS cases: 129,000 by 1992 and 345,639 as of December 1994.[36]  By contrast, several large studies recently published findings that among thousands of randomly selected Africans with standard AIDS diseases, fewer than half were HIV-positive.[37]  What does this say about a supposedly raging epidemic?\nA completely separate epidemic seems to affect rural Africans, this one having no identified risk group. Some reports suggest a correlation between AIDS there and the symptoms of malnutrition. Doctors observe that AIDS patients who eat least often, or whose diets are skewed by food availability, suffer the most rapid decline in health. This should surprise no one. In rural Africa, the most important aspects to be considered, as in the entire history of epidemics, are: sanitation, which rarely exists; clean water supplies, also rare or nonexistent; and decent nutrition. It would seem that HIV/AIDS has created no new epidemic in Africa. But since HIV/ET is such a well-received hoax, it jumps in and “takes credit,” while obfuscating relevant issues.[38]\nOne might be challenged, as the Ugandan doctor was, to distinguish between an AIDS/tubercuIosis and a traditional one. Since the clinical symptoms are identical, the CDC has stipulated in its current definition that tuberculosis must be renamed AIDS if HIV antibodies are also found. In the absence of HIV antibodies, the disease is classified under its old name, tuberculosis, and treated accordingly. Therefore, simply by definition/elasticity, HIV antibodies can never be found apart from AIDS, and vice versa; and any symptomology has the potential to become an AIDS indicator with HIV around. In general, if doctors can tell the difference between AIDS on the one hand, and non-AIDS presence of its indicator diseases on the other, only by testing for antibodies to HIV, which sometimes don’t even have to be present (discussed below), it would seem we have a syndrome of contrived or arbitrary origin, circularly defined.\nHIV/AIDS and Koch’s Postulates\n1. The germ must be found in all cases of the disease. Tissues said to be affected by HIV include primarily the white blood cells of the immune system, particularly the T-cells, the brain neurons in dementia, skin cells in lesions of Kaposi’s sarcoma, as well as, theoretically, any cell in the body expressing the CD4 surface receptor said to be the key to HIV cell entry. But no trace of the virus can be found in either the Kaposi’s sarcoma or the neurons of the central nervous system. HIV/ET has now moved from involving only immune cells to other types of cells in order to explain certain AIDS-defining symptoms which are not immune deficiencies anyway, including the cancers, dementia and wasting diseases, and which have not been, or cannot be, explained in terms of a germ-theory virus model that involves destruction of the immune system.\nAnd if HIV were actively infecting T-cells or other members of the body’s immune system, extracellular virions should easily be found circulating in the blood. But in most individuals suffering from AIDSyndrome, no particles can be found anywhere in the body.\nThe abundance of uninfected T-cells (about one in 500) in all AIDSyndrome patients is the definitive argument against the false claims for high cell-wall particle “loads,” or “burdens,” in AIDS patients. The absence of active, infectious virus automatically disqualifies HIV as a player in the AIDSyndrome.\n2. The germ must be isolated from the host and grown in pure culture. Even for the most experienced virus hunters, a virus that is so extremely scarce is difficult to find. Only with rare luck and extreme persistence has HIV been extracted from an antibody-positive person. This amounts to finding the proverbial needle of HIV in a haystack of human DNA. This difficulty speaks to HIV’s lack of potential in disease.\nHIV should cause AIDS within two weeks of infection at most, but it does not, and with the complete lack of a demonstrated process by which HIV diminishes immune function, belief in a decade or more of unexplained latency requires a level of “faith” beyond this writer’s capacity. Another major stumbling block is that even once the latent period is apparently over, there is still precious little development of the virus.\n4. The germ must then be isolable from the newly infected host. We are now back to the problem of meeting requirement number 2.\nOne explanation put forth for the deadliness of such a scarce pathogen is that it somehow induces a self-destructive immune response (the system attacks itself). Evidence for this is said to be low white cell counts in people with AIDSyndrome; however, there is nothing to support the hypothesis, i.e., no plausible process by which this occurs has been suggested (see “What’s Overlooked” below).\nFor the sake of discussion, let us allow germ-theory interpretation of immune function and autoimmunity. With only one in 500 immune cells said to be infected in HIV positives, it would seem to require a virus of extraordinary cunning to get uninfected cells to attack each other and not infected ones, which would be self-defeating for the virus. Or in the latter event, such cunning could be matched only by the adroitness required to move quickly from one host cell to another just before destruction. Or, if macrophages are involved, the process should lead either to increasing titers of virions in the blood, lymph, etc., as infected cells are lysed, or to increasing concentrations in macrophages if they are ingesting T-cells. This supports the reservoir notion (if there were any viruses to be found in them). It is thus easy to expand HIV/ET.\nEstablished in the early 1900s, Farr’s Law, which is fundamental to virology, states that viral disease develops exponentially, and dictates that illness will strike soon after infection. The rate-determining factor of the exponential growth of viruses is viral generation time, which is between 8 and 48 hours. Since laws are made to be broken or excepted, viruses with incubation periods longer than allowed by Farr’s Law are called “slow viruses.” And since HIV joins an exonerated class of viruses by not multiplying according to this law of virology, virologists stretch HIV/ET to accommodate it. The question arises, though, of how anyone can determine or demonstrate when a “natural” HIV infection takes place, and thus determine latency, since no one is being tested daily or weekly, etc., and there is no animal model. Within the slow-virus concept, adopted as an exception to Farr’s Law, retrovirologists can find refuge, hold on to their theory, hibernate in their labs, and hope the long winter of HIV latency is over before they expire.\nAccording to expert retrovirologist Dr. Peter Duesberg, “The slow virus concept has never been reconciled with the short generation time of viruses and the immune system. Once the virus lies totally dormant, an intact immune system will never allow any virus to be reactivated to multiply into numbers that would threaten the host. For a virus to be reactivated, the immune system first must be destroyed by something else-the real cause of a disease. A reactivated virus would just contribute an opportunistic infection. Thus, there are no slow viruses, only slow virologists.”42 Also, says Duesberg, “Retroviruses are all very similar. I mean, there are differences, but as far as pathology is concerned, you don’t see a marker in one which is going to explain why it supposedly wakes up from sleep and becomes active.”[43]\nThe Chemotherapy Drug Azidothymidine (AZT)\nHIV-antibody-positive individuals suffer major health risks from AIDS medications routinely administered by physicians uncritical of drug-company propaganda. AZT, an isolate from herring sperm, was first synthesized in 1964 by Jerome Horwitz, heading a lab at Detroit Cancer Foundation and financed by an NIH grant. Designed to kill cancer cells, Horwitz’s creation is a chemically modified form of a DNA building block. When a cell divides, it must copy its complete genetic code, which is stored in long chromosome chains. The DNA components (nucleotides) are linked to one another in a sequence. But Horwitz’s altered DNA building block enters the growing DNA chain while a cell is preparing to divide and acts as a premature terminator, blocking addition of DNA components. Being unable to copy its DNA sequence, the cell dies.\nAZT was the perfect killer of dividing cancer cells. When the compound was tested on cancer-ridden mice, however, it failed to perform as expected and instead revealed its extraordinarily deadly nature. The experimental drug was withdrawn from testing and never approved for human use-until AIDSyndrome. Side effects of AZT include ulcerations and hemorrhaging; damage to hair follicles and skin; destruction of mitochondria, the energy dynamos of cells; wasting of muscles; and the destruction of the immune system and other blood cells. Children are affected more severely, because many more of their cells are dividing than in adults.\nAmid scandal-(l) the single, human trial that was ruined, yet was claimed to have proven effectiveness; (2) free corporate (Burroughs Wellcome) acquisition of large amounts of National Cancer Institute (taxpayer) raw material and technology; and (3) government stonewalling of other, potentially less expensive antivirals-AZT was first approved for treatment of AIDS in 1987.44 The cost was $250 a shot, or about $18,000 per year, per case. In 1990 it was approved for AIDS prevention, and has currently reached an average cost of $6,000 per year.\nI have worked with many HIV-antibody-positive individuals who have for years remained completely free of any AIDS-indicator symptoms or any other significant ones. When treated with medications like AZT, however, people are observed to sicken and die from “wasting disease” in a short period of time. I, as well as other molecular cell biologists, know of no one who has been treated with AZT and lived for more than around one year. Fortunately, it has begun to fall out of favor as the drug of choice.\nUse of AZT is a good example of two other medical phenomena: (1) the odds game called the therapeutic index, or the relationship between a drug’s effectiveness and its toxicity; and (2) the dependence upon destruction that informs “scientific medicine.” The acceptable toxicity of a drug is directly proportional to, and established by, the deemed deadliness of the disease. However, to this date the Physicians’ Desk Reference quotes the low toxicity of AZT reported by Broder, Barry, Bolognesi, and colleagues in 1986. According to at least four independent studies published since, however, the toxicity of the drug is a thousand times higher.[45]\nBroder, Barry, Bolognesi, and colleagues overlooked or disregarded two basic factors in their lab experiments: (1) In the test tube in which they tested AZT, there was a high concentration of “infected” cells. But, as noted earlier, in a person with HIV, titers are very low, and the ratio of infected to healthy cells is very low (only 1 in about 500 T-cells in HIV antibody-positive persons is ever “infected”); (2) Like all other chemotherapy drugs, AZT is unable to distinguish between target cells and healthy cells. The disastrous consequence is that AZT must poison 499 good T-cells in order to poison one inhabited by the AIDS “virus.”\nShades of doubt concerning HIV/ET validity in terms of germ theory have arisen since three-quarters of the 20,000 hemophiliacs in the United States were infected by HIV through the blood supply a little more than a decade ago. During that period, clotting factor VIII doubled life expectancies, while relatively few developed AIDSyndrome. HIV has made no measurable impact on the well-being of hemophiliacs, except for devastation of those who are treated with AZT.[46] No evidence has shown that death rates from blood transfusions ever increased from HIV transmission, nor has anyone demonstrated that death rates declined once the virus was screened out of the blood supply.\nWith the primary U.S. AIDS groups, or with any group for that matter, if you understand microzymian principle and consider the blood as a flowing tissue, it will be seen in general that body fluids which find their way from one individual directly into the blood of another are a stress factor on the body. This is by virtue of the introduction of foreign tissue and possibly morbidly evolved microzymas. Total impact depends on the degree to which the terrain is already compromised. In fact, a major danger is blood transfusion itself, essentially a “tissue transplant,” which is a threat or irritant to immune function. There is no reason to believe that such repeated stress will not, by itself, overwork and weaken immune function and drain overall energy reserves.\nCurrent medical science gives credence to the so-called autoimmune response, where white cells said to be deranged indiscriminately destroy and/or clear out healthy and unhealthy cells. This misconception arises as a consequence of germ theory mentality, which misunderstands the central function of the immune system. It is essentially a sophisticated janitorial service. It operates to keep the place clean and to recycle usable material. Should “self cells or tissue become useless or even dangerous to the body, the immune system will clean them out. Thus, it is not deranged, but is doing its job correctly. The host is somehow not doing its job, however, to maintain a balanced internal environment, which is the first line of defense, not immunity, against tissue destruction and infection. This is because infection can come from within. And it bears repeating that the fundamental misconception of the germ theory is that infection must be invasion, rather than an endogenous morbid change in chemistry or micromorphology.\nCompromised or weakened by fungal infestation (evidence for which is obvious and strong) or by drugs and chemicals such as mycotoxins, the immune system may weaken and fail to be efficient, but it will not attack healthy cells. There is a situation where this may appear to be so-when free radicals produced by the immune system in response to mycotoxins and morbidly evolved microforms damage local cells and tissue by the “shotgun” effect – but it is not a direct attack on “self,” and is frequently an overreaction to the alarming situation.\nWhat Constitutes AIDS in 1998?\nIn the CDC guideline, the caveat was given that clinicians would not rely on this definition alone to diagnose serious disease caused by HIV. Good medical practice, which was apparently expected to be employed later, could be expected to catch cases that somehow slip through the vast surveillance net because they have either a negative H1V-antibody test or, in the presence of HIV antibody, an opportunistic disease not listed in the definition. With the new rules, in the case of diagnosis of any one of several indicator diseases by a “definitive method,” AIDS had to be diagnosed even if the patient were HIV negative.\nOne question would seem to be: Why not employ good medical practice at the outset? Also, with the vast range of conditions listed, one is hard pressed to imagine what might not be included, except perhaps the common cold. But the overall effect of this change was to boost statistics and bring more people into the web of fear surrounding the syndrome. In 1992 another statistic-bumping revision was handed down.\nToday the AIDS-indicator list includes, but is not limited to, Pneumocystis pneumonia, Kaposi’s sarcoma, non-Hodgkin’s lymphoma, candidiasis, cryptococcosis, tuberculosis, herpes simplex, cryptosporidiosis, coccidioidomycosis, toxoplasmosis, wasting disease and dementia. And symptomologies such as syphilis, chronic fatigue, anemia, arthritis, nephritis, pneumonitis, diarrhea, cervical cancer, and a T-cell count of less than 200 cells per microliter, or less than 14% of the expected level, have been added to the diagnostic list. It appears that when a higher rate of new AIDS cases is needed “for public health data,” the CDC expands the definition. With the stroke of a pen an illusion of the spread of AIDS is created. To include the major symptoms of malnutrition (wasting) as an AIDSyndrome indicator, especially in Africa and the Third World, is to ensure a burgeoning statistical picture.\nNor is this the first time such statistical manipulation has occurred in medical history, polio being an excellent example. According to Dr. Herbert Ratner, former public health officer for Oak Park, Illinois, prior to vaccine introduction, doctors were being paid $25 apiece by the National Foundation for Infantile Paralysis for polio case reports. Also, Ratner indicated, it was known that paralytic polio went away in 50 percent of cases within 60 days. After the arrival of the Salk vaccine, the case definition for polio was changed to require symptoms for 60 days before a diagnosis could be reported. Thus, if someone had it and it went away within that time, it was never counted, making the vaccine look better.48 After vaccine introduction, cases previously reported as poliomyelitis were differentiated as aseptic meningitis. Despite this subterfuge, case incidence increased dramatically after vaccine introduction (80 percent from 1958 to 1959) but the Public Health Service manipulated statistics and made statements to give the opposite impression.[49]\nThere are no “diseases” created by “microbes” invading from without. Viruses are not even symptogens. HIV has no causative connection with disease, and no new epidemic exists.\n[1]  L’Orthobiologie Somatidienne. Video by Gaston Naessens, 1991.\n[2]  Bechamp, Pierre Jacques Antoine. The Blood and Its Third Anatomical Element, (Montague R. Leverson, translator). London: John Ouseley Ltd., 1912, pp. 205 and 229.\n[4]  Hume, E. Douglas. Bechamp or Pasteur? Ashingdon, Rochford, Essex, England: The C.W. Daniel Co. Ltd., 1923, p. 109.\n[5]  Farber, C. Out of Africa, Part I. Spin Magazine, March 1993: 61-63, 86-87.\n[6]  Kalokerinos and Dettman, op. cit., p. 12.\n[8]  Denetclaw, T.H. and Denetclaw, W.F.J. Is “Southwest U.S. mystery disease” caused by hantavirus? Lancet, 1994; 343: 53-54.\n[9]  Russel, S. On the Trail of Hantavirus. San Francisco Chronicle, July 4,1995: AI, AI2.\n[10]  Russel, S. Signs that Ebola Virus Is Fading Away. San Francisco Chronicle, May 24,1995: A6.\n[11]  Kaiser, R. Africa State Hospitals Make Viruses, Not Patients, Feel at Home. Washington Post, June 4,1995: A6.\n[12]  Physicians’ Desk Reference (PDR), 1997, p. 1730.\n[13]  Beale, A.J. Vaccines and Antiviral Drugs. In: Topley and Wilson, Principles of Bacteriology, Virology and Immunity, (Publisher, Year?), p. 149.\n[14]  Jegede, V.A. et al. Vaccine Technology. In: Encyclopedia of Chemical Technology, (Publisher, Year?), p. 629.\n[16]  Rappaport, John. Touching All Bases-Exploring Alternative Theories of AIDS. The Reader (Los Angeles Free Weekly), August 7, 1987; Vol. 9, No. 42: 10. [Note: Reference pertains to evidence that calves were recently, or are still, cruelly used for this purpose. Minor details of the process differ from those described in the article.]\n[18]  Kalokerinos and Dettman, op. cit. (Ref. 3), p. 13.\n[20]  Goldberg, B. Origin of AIDS. Lancet, 1992; 339: 1548.\n[21]  Kalokerinos and Dettman, op. cit.\n[22]  Pearson, R.B. The Dream and Lie of Louis Pasteur. Collingwood, Australia: Sumeria Press, 1994, pp. 32-35.\n[24]  Hodgkinson, N. Experts Mount Startling Challenge to AIDS Orthodoxy. Sunday Times (London), April 26,1992; I: 12-13.\n[25]  Carroll, John. The Weird Way to Win a Nobel Prize. San Francisco Chronicle, October 21,1993: E9.\n[26]  Mirvish, S.S., Williamson, J., Badcook, D., Sheng-Chong, C. Mutagenicity of Iso-butyl nitrite vapor in the Ames test and some relevant chemical properties, including the reaction of iso-butyl nitrite with phosphate. Environmental. Molecular Mutagen, 1993; 21: 247-252.\n[27]  Rappaport, John. AIDS Inc. Scandal of the Century., San Mateo, Cal.: Human Energy Press, 1988, p. 38.\n[28]  Fungalbionics Convention: The Fungal/Mycotoxin Etiology of Chronic and Degenerative Disease. Metro Toronto Convention Centre, September 30,1994.\n[29]  Konotey-Ahulu, F.I.D. What is AIDS? Watford England: Tetteh-A’Domeno Co., 1989, p. 109.\n[30]  Rappaport, op. cit. (Ref. 27), p. 73.\n[31]  Williams, A.O. AIDS: An African Perspective. Boca Raton, Fla.: CRC Press, 1992, p. 238.\n[32]  Duesberg, Peter H. Inventing the AIDS t7ras,(**Publisher, YEAR?), p. 293.\n[33]  Konotey-Ahulu, op. cit., pp. 56-57.\n[34]  World Health Organization, The Current Global Situation of the HIV/AIDS Pandemic, 1995.\n[35]  Duesberg, Peter H. AIDS acquired by drug consumption and other noncontagious risk factors. Pharmacology and Therapeutics, 1992; 55: 258.\n[37]  Rappaport, op. cit. (Ref. 27), pp. 71-82.\n[38]  Biggar, R.J. et al. Seroepidemiology of HTLV-III antibodies in a remote population of Eastern Zaire. British Medical Journal, 1985; 290: 808-10 (cited in Coulter, Harris L. AIDS and Syphilis, The Hidden Link. Berkeley, Cal.: North Atlantic Books; and Washington: Wehawken Books, 1987; p. 61).\n[40]  Lemonick, M.D. Return to the Hot Zone. Time International, May 22,1995; 145: 56-57.\n[41]  Duesberg, Peter H. AIDS acquired by drug consumption . . . (Ref. 36), pp. 237-38, 241, 247.\n[42]  Rappaport, op. cit., p. 130.\n[43]  Committee on Government Operations. AIDS Drugs: Where Are They? 73rd Report. U.S. Government Printing Office; October 3, 1988. (Cited in Culbert, Michael L., D.Sc. AIDS: Hope Hoax and Hoopla. Chula Vista, Cal.: The Bradford Foundation, 1898, pp. 10-11).\n[44]  Chiu, D. and Duesberg, P.H. The toxicity of Azidothymidine (AZT) on human and animal cells in culture at concentrations used for antiviral therapy. Genetica 95, 1995: 103-109. Duesberg, Peter H. AIDS acquired by drug consumption . . . (Ref. 36), pp. 201-77.\n[45]  Yarchoan, R, Pluda, J.M., Perno, C.-F., Mitsuya, H., Broder, S. Anti-retroviral therapy of human immunodeficiency virus infection: Current strategies and challenges for the future. Blood, 1991; 78: 859-84. McLeod, G.X., Hammer, S.M. Zidovudine: Five years later. Annals of Internal Medicine, 1992; 117: 487-501.\n[46]  Duesberg, Peter H. Is HIV the cause of AIDS? Lancet, 1995; 346: 1371-72.\n[47]  Coulter, Harris L. AIDS and Syphilis, The Hidden Link. Berkeley, Cal.: North Atlantic Books; and Washington: Wehawken Books, 1987, p. 37.\n[48]  Rappaport, op. cit. (Ref. 27), pp. 152-53.\n[49]  Hearings before the House Committee on Interstate and Foreign Commerce. House of Representatives, 87th Congress, 2nd Session, H.R. 10541, May 1962, pp. 94, 96, 112 (cited in: James, Walene. Immunization: The Reality Behind the Myth, 2nd edition. Westport, CT: Bergin & Garvey (Greenwood Publishing Group, Inc.), 1995, pp. 35-36).\n[50]  Sermos, Gus G. Doctors of Deceit and the AIDS Epidemic-A View From the Inside. McComb, Miss.: GGS Publishing, 1988, p. 3.\nShare on X (Opens in new window) X\nShare on Pinterest (Opens in new window) Pinterest', '[-0.11445818096399307, 0.014998416416347027, -0.05974256992340088, 0.04089440777897835, 0.1201552078127861, 0.03205103799700737, 0.06333499401807785, 0.11813496798276901, 0.0586031936109066, 0.08832637220621109, 0.08442585170269012, -3.1453258998226374e-05, -0.003977081272751093, 0.07321616262197495, -0.10466600954532623, -0.042973436415195465, -0.07210640609264374, 0.05723130702972412, -0.022899968549609184, 0.06590733677148819, -0.0038884312380105257, 0.028244296088814735, 0.00574242090806365, -0.061920154839754105, -0.03371043503284454, 0.03044074960052967, -0.03252718597650528, -0.004626810550689697, 0.01977173611521721, 0.013029847294092178, 0.009738605469465256, 0.030165039002895355, 0.02337663434445858, -0.02048529125750065, -0.011477400548756123, -0.03324136137962341, 0.06230843439698219, -0.003052109619602561, 0.0029221251606941223, 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